D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 81 Citations 36,932 154 World Ranking 11188 National Ranking 5870

Research.com Recognitions

Awards & Achievements

Member of the Association of American Physicians

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Internal medicine

David E. Moller mainly focuses on Internal medicine, Endocrinology, Peroxisome proliferator-activated receptor, Insulin and Receptor. Many of his studies on Internal medicine involve topics that are commonly interrelated, such as Diabetes mellitus. The concepts of his Peroxisome proliferator-activated receptor study are interwoven with issues in Enhancer binding, Transactivation, Peroxisome proliferator-activated receptor delta, Lipid metabolism and Adipogenesis.

His research in Insulin focuses on subjects like Skeletal muscle, which are connected to Glycogen and Gluconeogenesis. In his study, which falls under the umbrella issue of Receptor, PPAR agonist is strongly linked to Nuclear receptor. In Metformin, he works on issues like AMPK, which are connected to Beta oxidation and Carbohydrate metabolism.

His most cited work include:

  • Role of AMP-activated protein kinase in mechanism of metformin action (4181 citations)
  • The Mechanisms of Action of PPARs (1998 citations)
  • New drug targets for type 2 diabetes and the metabolic syndrome (827 citations)

What are the main themes of his work throughout his whole career to date?

His main research concerns Internal medicine, Endocrinology, Insulin, Insulin receptor and Receptor. The Internal medicine study which covers Diabetes mellitus that intersects with Disease. His Endocrinology study frequently intersects with other fields, such as Peroxisome proliferator-activated receptor.

David E. Moller usually deals with Insulin receptor and limits it to topics linked to Insulin-like growth factor 1 receptor and 5-HT5A receptor. His study in Receptor is interdisciplinary in nature, drawing from both Nuclear receptor and Molecular biology. His study focuses on the intersection of Skeletal muscle and fields such as Glucose uptake with connections in the field of Carbohydrate metabolism.

He most often published in these fields:

  • Internal medicine (64.12%)
  • Endocrinology (63.53%)
  • Insulin (36.47%)

What were the highlights of his more recent work (between 2006-2021)?

  • Internal medicine (64.12%)
  • Endocrinology (63.53%)
  • Diabetes mellitus (18.82%)

In recent papers he was focusing on the following fields of study:

David E. Moller focuses on Internal medicine, Endocrinology, Diabetes mellitus, Type 2 diabetes and Biochemistry. His Endocrinology research incorporates themes from Peroxisome proliferator-activated receptor and In vivo. His biological study spans a wide range of topics, including Obesity, FGF21, Gastroenterology, Antagonist and Glucagon receptor.

His work on Amino acid, Endoplasmic reticulum, Peroxisome and Receptor as part of his general Biochemistry study is frequently connected to Phosphatidylcholine, thereby bridging the divide between different branches of science. The various areas that David E. Moller examines in his Receptor study include Adipocyte, Cytokine secretion and Peroxisome proliferator-activated receptor alpha. He has included themes like Nicotinamide adenine dinucleotide, NAD+ kinase and Nicotinamide phosphoribosyltransferase in his Insulin study.

Between 2006 and 2021, his most popular works were:

  • Fibroblast growth factor 21 corrects obesity in mice. (730 citations)
  • The effects of LY2405319, an FGF21 analog, in obese human subjects with type 2 diabetes. (555 citations)
  • Secreted PCSK9 downregulates low density lipoprotein receptor through receptor-mediated endocytosis (179 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Internal medicine

The scientist’s investigation covers issues in FGF21, Obesity, Internal medicine, Endocrinology and Adipose tissue. His FGF21 research incorporates elements of Diabetes mellitus, Insulin, Glycemic, Type 2 diabetes and Leptin. His Obesity study combines topics from a wide range of disciplines, such as Lipogenesis and Fatty liver.

His research related to Glucose transporter, Skeletal muscle, Glucose homeostasis, Homeostatic model assessment and Carbohydrate metabolism might be considered part of Internal medicine. His study involves Insulin resistance, Extensor digitorum longus muscle and Glucose uptake, a branch of Endocrinology. His Adipose tissue study combines topics in areas such as Amino acid and Receptor complex.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Role of AMP-activated protein kinase in mechanism of metformin action

Gaochao Zhou;Robert Myers;Ying Li;Yuli Chen.
Journal of Clinical Investigation (2001)

6151 Citations

The Mechanisms of Action of PPARs

Joel Berger;David E. Moller.
Annual Review of Medicine (2002)

3483 Citations

New drug targets for type 2 diabetes and the metabolic syndrome

David E. Moller.
Nature (2001)

1343 Citations

Fibroblast growth factor 21 corrects obesity in mice.

Tamer Coskun;Holly A. Bina;Michael A. Schneider;James D. Dunbar.
Endocrinology (2008)

1050 Citations

Insulin resistance--mechanisms, syndromes, and implications.

David E. Moller;Jeffrey S. Flier.
The New England Journal of Medicine (1991)

1049 Citations

Metformin increases AMP-activated protein kinase activity in skeletal muscle of subjects with type 2 diabetes.

Nicolas Musi;Michael F. Hirshman;Jonas Nygren;Monika Svanfeldt.
Diabetes (2002)

983 Citations

Potential Role of TNF-α in the Pathogenesis of Insulin Resistance and Type 2 Diabetes

David E Moller.
Trends in Endocrinology and Metabolism (2000)

964 Citations

Metabolic syndrome: a clinical and molecular perspective.

David E Moller;Keith D Kaufman.
Annual Review of Medicine (2005)

846 Citations

The effects of LY2405319, an FGF21 analog, in obese human subjects with type 2 diabetes.

Gregory Gaich;Jenny Y. Chien;Haoda Fu;Leonard C. Glass.
Cell Metabolism (2013)

812 Citations

Induction of Adipocyte Complement-Related Protein of 30 Kilodaltons by PPARγ Agonists: A Potential Mechanism of Insulin Sensitization

Terry P. Combs;John A. Wagner;Joel Berger;Tom Doebber.
Endocrinology (2002)

812 Citations

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