2022 - Research.com Best Scientist Award
2022 - Research.com Medicine in Japan Leader Award
His primary areas of study are Internal medicine, Endocrinology, Insulin resistance, Adiponectin and Insulin. His Internal medicine study combines topics from a wide range of disciplines, such as Diabetes mellitus and AMPK. His study in Diabetes mellitus is interdisciplinary in nature, drawing from both Genotype and Intensive care medicine.
Takashi Kadowaki interconnects Receptor and Peroxisome proliferator-activated receptor in the investigation of issues within Endocrinology. His biological study spans a wide range of topics, including Glucose uptake and Allele. His research in Adiponectin intersects with topics in Hormone, Adipokine and Beta oxidation.
His primary areas of investigation include Internal medicine, Endocrinology, Diabetes mellitus, Type 2 diabetes and Insulin resistance. The various areas that he examines in his Internal medicine study include Gastroenterology and Type 2 Diabetes Mellitus. His Endocrinology research incorporates themes from Receptor and Peroxisome proliferator-activated receptor.
His Diabetes mellitus research is multidisciplinary, relying on both Obesity, Surgery, Incidence and Intensive care medicine. His Type 2 diabetes research includes elements of Hypoglycemia and Genetics. Insulin resistance is closely attributed to Leptin in his research.
Takashi Kadowaki focuses on Internal medicine, Diabetes mellitus, Type 2 diabetes, Endocrinology and Insulin resistance. He works in the field of Internal medicine, namely Insulin. His studies deal with areas such as Obesity and Intensive care medicine as well as Diabetes mellitus.
His Type 2 diabetes study incorporates themes from Observational study, Genome-wide association study, Incidence, Kidney disease and Genetic association. Takashi Kadowaki combines subjects such as Inflammation and Downregulation and upregulation with his study of Endocrinology. As part of one scientific family, Takashi Kadowaki deals mainly with the area of Insulin resistance, narrowing it down to issues related to the Receptor, and often Adiponectin receptor 1.
Internal medicine, Endocrinology, Type 2 diabetes, Diabetes mellitus and Insulin resistance are his primary areas of study. The concepts of his Internal medicine study are interwoven with issues in Gastroenterology, Placebo and Type 2 Diabetes Mellitus. His study in Endocrinology focuses on Adiponectin, White adipose tissue, Adipose tissue, Lipid metabolism and Adipocyte.
His Type 2 diabetes research incorporates elements of Genetics, Genome-wide association study, Surgery, Diabetic nephropathy and Bioinformatics. His Diabetes mellitus research is multidisciplinary, incorporating elements of Medical prescription and Genetic association. His biological study deals with issues like Glucose uptake, which deal with fields such as Glucose clamp technique, Adipose triglyceride lipase, Tofogliflozin and Anagliptin.
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The fat-derived hormone adiponectin reverses insulin resistance associated with both lipoatrophy and obesity
T. Yamauchi;J. Kamon;H. Waki;Y. Terauchi.
Nature Medicine (2001)
Adiponectin stimulates glucose utilization and fatty-acid oxidation by activating AMP-activated protein kinase
T Yamauchi;J Kamon;Y Minokoshi;Y Ito.
Nature Medicine (2002)
Cloning of adiponectin receptors that mediate antidiabetic metabolic effects
Toshimasa Yamauchi;Junji Kamon;Yusuke Ito;Atsushi Tsuchida.
Adiponectin and adiponectin receptors in insulin resistance, diabetes, and the metabolic syndrome
Takashi Kadowaki;Toshimasa Yamauchi;Naoto Kubota;Kazuo Hara.
Journal of Clinical Investigation (2006)
Adiponectin and Adiponectin Receptors
Takashi Kadowaki;Toshimasa Yamauchi.
Endocrine Reviews (2005)
Diabetes in Asia: epidemiology, risk factors, and pathophysiology.
Juliana C. N. Chan;Vasanti Malik;Weiping Jia;Takashi Kadowaki.
Report of the Committee on the Classification and Diagnostic Criteria of Diabetes Mellitus
Yutaka Seino;Kishio Nanjo;Naoko Tajima;Takashi Kadowaki.
Journal of Diabetes Investigation (2010)
CD8 + effector T cells contribute to macrophage recruitment and adipose tissue inflammation in obesity
Satoshi Nishimura;Ichiro Manabe;Mika Nagasaki;Koji Eto.
Nature Medicine (2009)
Disruption of adiponectin causes insulin resistance and neointimal formation.
Naoto Kubota;Yasuo Terauchi;Toshimasa Yamauchi;Tetsuya Kubota.
Journal of Biological Chemistry (2002)
PPARγ Mediates High-Fat Diet–Induced Adipocyte Hypertrophy and Insulin Resistance
Naoto Kubota;Yasuo Terauchi;Hiroshi Miki;Hiroyuki Tamemoto;Hiroyuki Tamemoto.
Molecular Cell (1999)
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