Satoshi Kimura focuses on Internal medicine, Endocrinology, Insulin resistance, Biochemistry and Virology. His work carried out in the field of Internal medicine brings together such families of science as Gastroenterology and Genotype. His work carried out in the field of Endocrinology brings together such families of science as Receptor and Downregulation and upregulation.
His research in Insulin resistance intersects with topics in Peroxisome proliferator-activated receptor, Type 2 diabetes and Leptin. His Cellulosic ethanol, Ammonia, Cellulase and Glycoside hydrolase family 7 study, which is part of a larger body of work in Biochemistry, is frequently linked to Biofuel, bridging the gap between disciplines. As part of one scientific family, Satoshi Kimura deals mainly with the area of Virology, narrowing it down to issues related to the Liver disease, and often Epitope, HBsAg, Immune system and Hbsag negative.
His primary scientific interests are in Internal medicine, Endocrinology, Virology, Immunology and Molecular biology. The concepts of his Internal medicine study are interwoven with issues in Gastroenterology and Diabetes mellitus. His Endocrinology study incorporates themes from Biochemistry and Parathyroid hormone.
His Virology research incorporates themes from Recombinant DNA and Polymerase chain reaction. His studies deal with areas such as Pneumocystis carinii, Acquired immunodeficiency syndrome and Pneumonia as well as Immunology. Insulin resistance and Type 2 diabetes are commonly linked in his work.
Satoshi Kimura mainly focuses on Virology, Acquired immunodeficiency syndrome, Internal medicine, Immunology and Hydrolysis. His Virology research integrates issues from Reverse transcriptase, Antibody, Recombinant DNA and Microbiology. The study incorporates disciplines such as Gastroenterology, Endocrinology and Efavirenz in addition to Internal medicine.
His Endocrinology research includes elements of In situ hybridization and In vivo. Satoshi Kimura interconnects Confidence interval and Antiretroviral therapy in the investigation of issues within Immunology. His Hydrolysis research is multidisciplinary, relying on both Crystallography and Cellulose.
Satoshi Kimura spends much of his time researching Hydrolysis, Trichoderma reesei, Cellulose, Virology and Cellulase. His Hydrolysis study integrates concerns from other disciplines, such as Crystallography and Substrate. Satoshi Kimura has included themes like Glycosidic bond, Fungal protein and Active site in his Substrate study.
His Virology research includes themes of Recombinant DNA, Microbiology, Molecular biology, Sputum and Vancomycin. His study in Molecular biology is interdisciplinary in nature, drawing from both Jurkat cells, T cell, T-cell receptor, NFAT and Flagellin. His Cellulase study introduces a deeper knowledge of Biochemistry.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Disruption of adiponectin causes insulin resistance and neointimal formation.
Naoto Kubota;Yasuo Terauchi;Toshimasa Yamauchi;Tetsuya Kubota.
Journal of Biological Chemistry (2002)
PPARγ Mediates High-Fat Diet–Induced Adipocyte Hypertrophy and Insulin Resistance
Naoto Kubota;Yasuo Terauchi;Hiroshi Miki;Hiroyuki Tamemoto;Hiroyuki Tamemoto.
Molecular Cell (1999)
The core protein of hepatitis C virus induces hepatocellular carcinoma in transgenic mice.
Kyoji Moriya;Hajime Fujie;Yoshizumi Shintani;Hiroshi Yotsuyanagi.
Nature Medicine (1998)
Impaired Multimerization of Human Adiponectin Mutants Associated with Diabetes MOLECULAR STRUCTURE AND MULTIMER FORMATION OF ADIPONECTIN
Hironori Waki;Toshimasa Yamauchi;Junji Kamon;Yusuke Ito.
Journal of Biological Chemistry (2003)
Globular Adiponectin Protected ob/ob Mice from Diabetes and ApoE-deficient Mice from Atherosclerosis
Toshimasa Yamauchi;Junji Kamon;Hironori Waki;Yasushi Imai.
Journal of Biological Chemistry (2003)
Genetic variation in the gene encoding adiponectin is associated with an increased risk of type 2 diabetes in the japanese population
Kazuo Hara;Philippe Boutin;Yasumichi Mori;Kazuyuki Tobe.
Diabetes (2002)
Hepatitis C virus infection and diabetes: direct involvement of the virus in the development of insulin resistance
Yoshizumi Shintani;Hajime Fujie;Hideyuki Miyoshi;Takeya Tsutsumi.
Gastroenterology (2004)
The Mechanisms by Which Both Heterozygous Peroxisome Proliferator-activated Receptor γ (PPARγ) Deficiency and PPARγ Agonist Improve Insulin Resistance
Toshimasa Yamauchi;Junji Kamon;Hironori Waki;Koji Murakami.
Journal of Biological Chemistry (2001)
Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia.
Naoto Kubota;Kazuyuki Tobe;Yasuo Terauchi;Kazuhiro Eto.
Diabetes (2000)
Traffic Jams Reduce Hydrolytic Efficiency of Cellulase on Cellulose Surface
Kiyohiko Igarashi;Takayuki Uchihashi;Anu Koivula;Masahisa Wada;Masahisa Wada.
Science (2011)
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