Thomas W.H. Kay spends much of his time researching Immunology, Internal medicine, Type 1 diabetes, Endocrinology and Insulitis. He interconnects Nod and Proinsulin in the investigation of issues within Immunology. His Type 1 diabetes research is multidisciplinary, incorporating elements of Pancreas transplantation and Islet, Insulin, Glycemic.
His Islet research incorporates elements of Apoptosis and Transplantation. Thomas W.H. Kay has researched Endocrinology in several fields, including Downregulation and upregulation and Transcription factor. His Insulitis study incorporates themes from CD8 and Major histocompatibility complex.
His scientific interests lie mostly in Immunology, Internal medicine, Endocrinology, Islet and NOD mice. His Immunology study integrates concerns from other disciplines, such as Type 1 diabetes and Beta cell. His Internal medicine course of study focuses on Diabetes mellitus and Pancreas and Autoantibody.
His work carried out in the field of Endocrinology brings together such families of science as Proinflammatory cytokine and Signal transduction. The study incorporates disciplines such as Transplantation and Cell biology in addition to Islet. His NOD mice research is multidisciplinary, relying on both T cell and Cytokine.
His primary scientific interests are in Islet, Transplantation, Cell biology, NOD mice and Type 1 diabetes. His research on Islet concerns the broader Internal medicine. His work deals with themes such as Diabetes mellitus, Endocrinology and Gastroenterology, which intersect with Internal medicine.
The various areas that he examines in his NOD mice study include T cell and CD8. His Type 1 diabetes research is multidisciplinary, incorporating perspectives in Proinsulin and Immunology, Immune system, Antigen. Thomas W.H. Kay integrates Immunology with Sting in his study.
His scientific interests lie mostly in Type 1 diabetes, Transplantation, Islet, Diabetes mellitus and T cell. His Type 1 diabetes research includes themes of C-peptide, Immunology, Antigen, Proinsulin and Autoimmunity. The subject of his Islet research is within the realm of Insulin.
His Insulin research is under the purview of Internal medicine. His Internal medicine research includes elements of Endocrinology and Bioenergetics. As part of the same scientific family, Thomas W.H. Kay usually focuses on T cell, concentrating on NOD mice and intersecting with Cancer research, NFKB1 and NF-κB.
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Proapoptotic Bcl-2 relative Bim required for certain apoptotic responses, leukocyte homeostasis, and to preclude autoimmunity.
Philippe Bouillet;Donald Metcalf;David C. S. Huang;David M. Tarlinton.
SOCS1 Is a Critical Inhibitor of Interferon γ Signaling and Prevents the Potentially Fatal Neonatal Actions of this Cytokine
Warren S Alexander;Robyn Starr;Robyn Starr;Jennifer E Fenner;Clare L Scott;Clare L Scott.
Improvement in outcomes of clinical islet transplantation: 1999-2010.
Franca B. Barton;Michael R. Rickels;Rodolfo Alejandro;Bernhard J. Hering.
Diabetes Care (2012)
Demonstration of islet-autoreactive CD8 T cells in insulitic lesions from recent onset and long-term type 1 diabetes patients
Ken T. Coppieters;Francesco Dotta;Natalie Amirian;Peter D. Campbell.
Journal of Experimental Medicine (2012)
Essential role for interferon-gamma and interleukin-6 in autoimmune insulin-dependent diabetes in NOD/Wehi mice.
I. L. Campbell;T. W. H. Kay;L. Oxbrow;L. C. Harrison.
Journal of Clinical Investigation (1991)
Tumor necrosis factor α-induced skeletal muscle insulin resistance involves suppression of AMP-kinase signaling
Gregory r Steinberg;Belinda J Michell;Bryce J W Jw van Denderen;Matthew James Watt.
Cell Metabolism (2006)
Granulocyte Macrophage Colony-Stimulating Factor A New Putative Therapeutic Target in Multiple Sclerosis
Jonathan Luke McQualter;Rima Darwiche;Christine Ewing;Manabu Onuki.
Journal of Experimental Medicine (2001)
Transgenic Expression of Mouse Proinsulin II Prevents Diabetes in Nonobese Diabetic Mice
Michelle B French;Janette Allison;David S Cram;Helen E Thomas.
Hypoxia-inducible factor-1α regulates β cell function in mouse and human islets
Kim Cheng;Kenneth Ho;Rebecca Stokes;Christopher Scott.
Journal of Clinical Investigation (2010)
Responses against islet antigens in NOD mice are prevented by tolerance to proinsulin but not IGRP
Balasubramanian Krishnamurthy;Nadine L. Dudek;Mark D. McKenzie;Anthony W. Purcell.
Journal of Clinical Investigation (2006)
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