Takemi Enomoto spends much of his time researching Molecular biology, Helicase, Bloom syndrome, Methyl methanesulfonate and RecQ helicase. His Molecular biology research is multidisciplinary, incorporating elements of Cell culture, Gene, Transfection, Replication factor C and Genome instability. His work carried out in the field of Genome instability brings together such families of science as Point mutation and DNA repair.
While the research belongs to areas of Bloom syndrome, Takemi Enomoto spends his time largely on the problem of Sister chromatid exchange, intersecting his research to questions surrounding Homologous recombination. His Methyl methanesulfonate research includes elements of Sgs1 and Saccharomyces cerevisiae. His RecQ helicase research is multidisciplinary, incorporating perspectives in Transferase, Circular bacterial chromosome, DNA helicase activity, Genomic library and RNA Helicase A.
His scientific interests lie mostly in Molecular biology, Cell biology, Genetics, DNA replication and Helicase. His studies in Molecular biology integrate themes in fields like Eukaryotic DNA replication, DNA, DNA repair, DNA polymerase and Methyl methanesulfonate. Takemi Enomoto combines subjects such as Chromatin and WRNIP1 with his study of Cell biology.
His study looks at the relationship between DNA replication and topics such as DNA polymerase delta, which overlap with DNA polymerase II. When carried out as part of a general Helicase research project, his work on RNA Helicase A, RecQ helicase and DNA helicase activity is frequently linked to work in Premature aging, therefore connecting diverse disciplines of study. Takemi Enomoto focuses mostly in the field of Sgs1, narrowing it down to topics relating to Homologous recombination and, in certain cases, Bloom syndrome and Genome instability.
Takemi Enomoto mostly deals with Molecular biology, Cell biology, Eukaryotic DNA replication, Origin recognition complex and Control of chromosome duplication. His Molecular biology research incorporates elements of Cell culture, Histone code and Gene, DNA, DNA replication. As part of one scientific family, he deals mainly with the area of DNA replication, narrowing it down to issues related to the RecQ helicase, and often RMI1, Sgs1, Establishment of sister chromatid cohesion and Homologous recombination.
His work deals with themes such as Genetics, WRNIP1, DNA damage, SOD1 and SOD2, which intersect with Cell biology. The concepts of his DNA damage study are interwoven with issues in Sister chromatid exchange and Mutation. His Helicase study incorporates themes from Gene conversion and DNA polymerase.
Takemi Enomoto mainly focuses on Molecular biology, Helicase, RecQ helicase, DNA repair and RNA Helicase A. The study incorporates disciplines such as Replication factor C, DNA replication factor CDT1, Control of chromosome duplication, DNA replication initiation and Origin recognition complex in addition to Molecular biology. His research integrates issues of Gene knockout and DNA replication in his study of Helicase.
His RecQ helicase study combines topics in areas such as Processivity, Polymerase and DNA polymerase.
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Purification and cloning of a nucleotide excision repair complex involving the xeroderma pigmentosum group C protein and a human homologue of yeast RAD23.
C. Masutani;K. Sugasawa;J. Yanagisawa;T. Sonoyama.
The EMBO Journal (1994)
Adenovirus DNA replication in vitro: identification of a host factor that stimulates synthesis of the preterminal protein-dCMP complex
Kyosuke Nagata;Ronald A. Guggenheimer;Takemi Enomoto;Jack H. Lichy.
Proceedings of the National Academy of Sciences of the United States of America (1982)
Ubc9- and Mms21-Mediated Sumoylation Counteracts Recombinogenic Events at Damaged Replication Forks
Dana Branzei;Julie Sollier;Giordano Liberi;Xiaolan Zhao.
Cell (2006)
Molecular cloning of cDNA encoding human DNA helicase Q1 Which has homology to Escherichia coli Rec Q helicase and localization of the gene at chromosome 12p12
Masayuki Seki;Hiroshi Miyazawa;Shusuke Tada;Junn Yanagisawa.
Nucleic Acids Research (1994)
RMI, a new OB-fold complex essential for Bloom syndrome protein to maintain genome stability.
Dongyi Xu;Rong Guo;Alexandra Sobeck;Csanad Z. Bachrati.
Genes & Development (2008)
Structural basis for inhibition of the replication licensing factor Cdt1 by geminin
Changwook Lee;BumSoo Hong;Jung Min Choi;Yugene Kim.
Nature (2004)
Possible association of BLM in decreasing DNA double strand breaks during DNA replication
Wensheng Wang;Masayuki Seki;Yoshiyasu Narita;Eiichiro Sonoda.
The EMBO Journal (2000)
Ubc9 is essential for viability of higher eukaryotic cells.
Tomoko Hayashi;Masayuki Seki;Daisuke Maeda;Wensheng Wang.
Experimental Cell Research (2002)
Adenovirus DNA replication in vitro: purification of the terminal protein in a functional form
Takemi Enomoto;Jack H. Lichy;Joh-E. Ikeda;Jerard Hurwitz.
Proceedings of the National Academy of Sciences of the United States of America (1981)
Functional relationships of FANCC to homologous recombination, translesion synthesis, and BLM.
Seiki Hirano;Kazuhiko Yamamoto;Kazuhiko Yamamoto;Masamichi Ishiai;Mitsuyoshi Yamazoe.
The EMBO Journal (2005)
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