What is he best known for?
The fields of study he is best known for:
Michael J. Lenardo focuses on Cell biology, Apoptosis, Immunology, Fas receptor and Caspase.
Michael J. Lenardo works in the field of Cell biology, focusing on Signal transduction in particular.
His study of Programmed cell death is a part of Apoptosis.
In his study, which falls under the umbrella issue of Programmed cell death, Tumor necrosis factor alpha is strongly linked to CD8.
As part of his studies on Immunology, he often connects relevant subjects like Autoimmune lymphoproliferative syndrome.
The Fas receptor study combines topics in areas such as Fas ligand and FADD.
His most cited work include:
- The TNF and TNF receptor superfamilies: integrating mammalian biology. (3085 citations)
- Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes (1951 citations)
- NF-κB: A pleiotropic mediator of inducible and tissue-specific gene control (1330 citations)
What are the main themes of his work throughout his whole career to date?
Michael J. Lenardo spends much of his time researching Cell biology, Immunology, Apoptosis, T cell and Molecular biology.
Michael J. Lenardo has researched Cell biology in several fields, including Caspase, Programmed cell death, Receptor and T-cell receptor.
As part of one scientific family, he deals mainly with the area of Immunology, narrowing it down to issues related to the Autoimmune lymphoproliferative syndrome, and often Lymphocyte homeostasis, Mutation, Lymphoma, Autoimmunity and Lymphoproliferative disorders.
Apoptosis is a component of his Fas receptor and FADD studies.
His study in T cell is interdisciplinary in nature, drawing from both Cytotoxic T cell, T lymphocyte and Lymphokine.
He has included themes like Enhancer, Transcription factor, Gene, Cellular differentiation and Binding site in his Molecular biology study.
He most often published in these fields:
- Cell biology (40.25%)
- Immunology (33.33%)
- Apoptosis (19.50%)
What were the highlights of his more recent work (between 2013-2021)?
- Immunology (33.33%)
- Immune system (18.55%)
- Cell biology (40.25%)
In recent papers he was focusing on the following fields of study:
Michael J. Lenardo mostly deals with Immunology, Immune system, Cell biology, T cell and Immunodeficiency.
His work in Immunology is not limited to one particular discipline; it also encompasses Disease.
The concepts of his Immune system study are interwoven with issues in Lymphoproliferative disorders, Cytokine and Function.
His Cell biology research integrates issues from Autophagy, PINK1 and Mitophagy.
His work focuses on many connections between T cell and other disciplines, such as Cytotoxic T cell, that overlap with his field of interest in Cancer research.
While the research belongs to areas of T-cell receptor, Michael J. Lenardo spends his time largely on the problem of Signal transduction, intersecting his research to questions surrounding Receptor.
Between 2013 and 2021, his most popular works were:
- 30 Years of NF-κB: A Blossoming of Relevance to Human Pathobiology (670 citations)
- Immune dysregulation in human subjects with heterozygous germline mutations in CTLA4 (485 citations)
- Dominant-activating germline mutations in the gene encoding the PI(3)K catalytic subunit p110δ result in T cell senescence and human immunodeficiency (379 citations)
In his most recent research, the most cited papers focused on:
His primary areas of study are Immunology, Immune system, Cell biology, Immune dysregulation and Immunodeficiency.
Specifically, his work in Immunology is concerned with the study of CD8.
As a part of the same scientific study, Michael J. Lenardo usually deals with the CD8, concentrating on Lymphoma and frequently concerns with Fas receptor and Sepsis.
His Immune system research incorporates themes from Cancer and Second messenger system.
Michael J. Lenardo is interested in Mitochondrion, which is a branch of Cell biology.
His research in Cytotoxic T cell focuses on subjects like Germline mutation, which are connected to PI3K/AKT/mTOR pathway, Molecular biology and Cellular differentiation.
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