What is he best known for?
The fields of study he is best known for:
- Gene
- Enzyme
- Immune system
Pascal Schneider mostly deals with Cell biology, B-cell activating factor, Immunology, B cell and Receptor.
His Cell biology study combines topics from a wide range of disciplines, such as Inhibitor of apoptosis, Apoptosis, Death domain and Caspase 8.
His study in BAFF receptor and B cell homeostasis falls under the purview of B-cell activating factor.
He has researched BAFF receptor in several fields, including B-Cell Maturation Antigen, Marginal zone and Transmembrane activator and CAML interactor.
His work carried out in the field of B cell brings together such families of science as CD23, Lymphocyte and CD40.
In Receptor, he works on issues like Immune system, which are connected to Homology, Structural motif, Structural biology, Protein family and Laminin.
His most cited work include:
- Inhibition of death receptor signals by cellular FLIP (2342 citations)
- Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule. (1385 citations)
- Mice transgenic for BAFF develop lymphocytic disorders along with autoimmune manifestations. (1342 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Cell biology, B-cell activating factor, Receptor, Immunology and Antibody.
His research in Cell biology intersects with topics in Apoptosis, Programmed cell death, Fas receptor and Immune system.
B cell covers Pascal Schneider research in B-cell activating factor.
The various areas that Pascal Schneider examines in his Receptor study include Tumor necrosis factor alpha, Molecular biology, Cancer research and Pharmacology.
His studies deal with areas such as Peptide sequence and Ligand as well as Molecular biology.
His research ties Cytotoxic T cell and Immunology together.
He most often published in these fields:
- Cell biology (33.51%)
- B-cell activating factor (23.43%)
- Receptor (22.62%)
What were the highlights of his more recent work (between 2013-2021)?
- Cell biology (33.51%)
- B-cell activating factor (23.43%)
- Immunology (22.62%)
In recent papers he was focusing on the following fields of study:
The scientist’s investigation covers issues in Cell biology, B-cell activating factor, Immunology, Receptor and Cancer research.
Signal transduction is the focus of his Cell biology research.
B cell and Antibody are all intrinsically tied to his study in B-cell activating factor.
As part of the same scientific family, Pascal Schneider usually focuses on Immunology, concentrating on Systemic lupus erythematosus and intersecting with Autoantibody.
His research integrates issues of Tumor necrosis factor alpha, Cell culture, Antigen, Molecular biology and B-Cell Maturation Antigen in his study of Receptor.
His Cancer research study deals with Immune system intersecting with Cytotoxic T cell and Homeostasis.
Between 2013 and 2021, his most popular works were:
- The BAFF/APRIL system in SLE pathogenesis (233 citations)
- Cutting Edge: IL-1α Is a Crucial Danger Signal Triggering Acute Myocardial Inflammation during Myocardial Infarction (78 citations)
- Targeting of Fn14 Prevents Cancer-Induced Cachexia and Prolongs Survival (71 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Enzyme
- Immune system
Pascal Schneider mainly investigates Cell biology, Immunology, B-cell activating factor, Receptor and Tumor necrosis factor alpha.
Pascal Schneider has included themes like Ectodysplasin A, Acquired immune system, Fas ligand, Apoptosis and Integrin in his Cell biology study.
The concepts of his Immunology study are interwoven with issues in Cancer research and Signal transduction.
His research on B-cell activating factor is centered around B cell and Genetics.
As a member of one scientific family, Pascal Schneider mostly works in the field of Receptor, focusing on Cell culture and, on occasion, Cell, Avidity and Molecular biology.
His BAFF receptor research includes themes of Plasma protein binding, B-Cell Maturation Antigen and Transmembrane activator and CAML interactor.
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