His primary areas of study are Immunology, Molecular biology, T cell, Cytotoxic T cell and Cytokine. His Immunology study combines topics in areas such as Interleukin 12 and Cancer research. His work in Molecular biology addresses issues such as Ifn gamma, which are connected to fields such as Interleukin 18, IFN-gamma-Inducing Factor, T cell clone, Simultaneous stimulation and Monoclonal antibody.
Hiromi Fujiwara does research in T cell, focusing on Antigen-presenting cell specifically. His Cytotoxic T cell research is multidisciplinary, incorporating elements of C-C chemokine receptor type 6, Ovalbumin, Antigen and Cell biology. His research investigates the link between Cytokine and topics such as Pharmacology that cross with problems in Cell, Hapten, Sensitization and Contact sensitivity.
The scientist’s investigation covers issues in Immunology, T cell, Molecular biology, Cytotoxic T cell and Antigen. Hiromi Fujiwara regularly links together related areas like Cancer research in his Immunology studies. His T cell research incorporates elements of Immunotherapy, Cell, B cell and Cell biology.
The concepts of his Molecular biology study are interwoven with issues in Cytokine, CD8, Interleukin 12 and Antibody, Monoclonal antibody. His Interleukin 12 study combines topics from a wide range of disciplines, such as T cell migration and Stimulation. Hiromi Fujiwara works mostly in the field of Antigen, limiting it down to topics relating to Tumor antigen and, in certain cases, Fibrosarcoma.
Hiromi Fujiwara mostly deals with Cell biology, Immunology, Interleukin 12, T cell and Molecular biology. His Cell biology study incorporates themes from Cytotoxic T cell, Antigen-presenting cell, CXCL16 and CD3. His work carried out in the field of Immunology brings together such families of science as Cancer research and CD40.
His studies deal with areas such as T cell migration, Immune system and Stimulation as well as Interleukin 12. As a part of the same scientific family, Hiromi Fujiwara mostly works in the field of T cell, focusing on Receptor and, on occasion, Transforming growth factor. His Molecular biology research is multidisciplinary, incorporating perspectives in XCL2, Mutant, Antigen, C-C chemokine receptor type 6 and CC chemokine receptors.
His primary scientific interests are in Molecular biology, Cytokine, Immunology, Interleukin 12 and Cell biology. His studies deal with areas such as Gene expression, CCL21, CXC chemokine receptors, Chemokine Receptor Antagonist and CXCL13 as well as Molecular biology. His Cytokine research includes elements of Liver injury, Immune system and Cell adhesion molecule.
Immunology and Endocrinology are commonly linked in his work. His research integrates issues of T cell, Stimulation and Parenchyma in his study of Interleukin 12. His Cell biology research focuses on Cytotoxic T cell and how it connects with CXCL16, CCR8 and Priming.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
T cell activation-associated hepatic injury: mediation by tumor necrosis factors and protection by interleukin 6.
Hidekazu Mizuhara;Elaine O'Neill;Nobuo Seki;Toshikazu Ogawa.
Journal of Experimental Medicine (1994)
A mechanism underlying synergy between IL-12 and IFN-gamma-inducing factor in enhanced production of IFN-gamma.
H J Ahn;S Maruo;M Tomura;J Mu.
Journal of Immunology (1997)
Type II collagen-induced murine arthritis. I. Induction and perpetuation of arthritis require synergy between humoral and cell-mediated immunity.
N Seki;Y Sudo;T Yoshioka;S Sugihara.
Journal of Immunology (1988)
Transforming growth factor-beta-induced inhibition of T cell function. Susceptibility difference in T cells of various phenotypes and functions and its relevance to immunosuppression in the tumor-bearing state.
T. Tada;S. Ohzeki;K. Utsumi;H. Takiuchi.
Journal of Immunology (1991)
A Novel Function of Vα14+CD4+NKT Cells: Stimulation of IL-12 Production by Antigen-Presenting Cells in the Innate Immune System
Michio Tomura;Wen-Gong Yu;Hyun-Jong Ahn;Motozo Yamashita.
Journal of Immunology (1999)
Systemic administration of rIL-12 induces complete tumor regression and protective immunity: response is correlated with a striking reversal of suppressed IFN-γ production by anti-tumor T cells
Jian-Ping Zou;Norihiko Yamamoto;Tetsuya Fujii;Hiroshi Takenaka.
International Immunology (1995)
A pivotal involvement of IFN-γ in the pathogenesis of acetaminophen-induced acute liver injury
Yuko Ishida;Toshikazu Kondo;Tohru Ohshima;Hiromi Fujiwara.
The FASEB Journal (2002)
Critical involvement of interferon gamma in the pathogenesis of T-cell activation-associated hepatitis and regulatory mechanisms of interleukin-6 for the manifestations of hepatitis.
H Mizuhara;M Uno;N Seki;M Yamashita.
Hepatology (1996)
Enhanced Induction of Antitumor T-Cell Responses by Cytotoxic T Lymphocyte-associated Molecule-4 Blockade: The Effect Is Manifested Only at the Restricted Tumor-bearing Stages
Yi Fu Yang;Jian Ping Zou;Jie Mu;Rishani Wijesuriya.
Cancer Research (1997)
The role of tumor-specific Lyt-1+2- T cells in eradicating tumor cells in vivo. I. Lyt-1+2- T cells do not necessarily require recruitment of host's cytotoxic T cell precursors for implementation of in vivo immunity.
H Fujiwara;M Fukuzawa;T Yoshioka;H Nakajima.
Journal of Immunology (1984)
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