His primary scientific interests are in Immunology, Molecular biology, Internal medicine, Cell biology and Pathology. His research in Atopic dermatitis, CC chemokine receptors, Immunoglobulin E, Chemokine and Antibody are components of Immunology. His Molecular biology research includes themes of Cytokine, Type I collagen, Receptor, Matrix metalloproteinase and Kinase.
His study in Internal medicine is interdisciplinary in nature, drawing from both Gastroenterology, Endocrinology and Surgery. His Cell biology research is multidisciplinary, incorporating perspectives in Biochemistry and Antigen. Kunihiko Tamaki works in the field of Pathology, focusing on Fibrosis in particular.
His main research concerns Immunology, Pathology, Molecular biology, Internal medicine and Dermatology. His study in Antibody, Atopic dermatitis, Chemokine, Immunoglobulin E and CCL17 falls under the purview of Immunology. His research on Pathology often connects related areas such as Antigen.
His Molecular biology study incorporates themes from Gene expression, Receptor, Signal transduction, Transforming growth factor and Epidermis. His Internal medicine course of study focuses on Endocrinology and Fibroblast. Kunihiko Tamaki works mostly in the field of Connective tissue disease, limiting it down to concerns involving Immunopathology and, occasionally, Autoimmune disease.
Kunihiko Tamaki spends much of his time researching Pathology, Immunology, Dermatology, Internal medicine and Cell biology. As a part of the same scientific study, Kunihiko Tamaki usually deals with the Pathology, concentrating on Epidermis and frequently concerns with Proinflammatory cytokine. His Dermatology study combines topics in areas such as After treatment and Prednisolone.
His work deals with themes such as Gastroenterology, Endocrinology and Immune system, which intersect with Internal medicine. As part of the same scientific family, Kunihiko Tamaki usually focuses on Cell biology, concentrating on CD19 and intersecting with Regulatory B cells. His Regulatory B cells research focuses on Molecular biology and how it relates to Cell culture.
Kunihiko Tamaki mainly focuses on Immunology, Pathology, Internal medicine, Atopic dermatitis and Chemokine. Kunihiko Tamaki interconnects Involucrin, Etretinate and Cell biology in the investigation of issues within Pathology. His Internal medicine study combines topics from a wide range of disciplines, such as Gastroenterology and Endocrinology.
His Atopic dermatitis study integrates concerns from other disciplines, such as Young adult, Allergy, MEDLINE and CCL27. His Chemokine research incorporates themes from Chemotaxis, Cytokine and CD40. His research integrates issues of CD1D, Molecular biology, Adoptive cell transfer, CD19 and CD5 in his study of Regulatory B cells.
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Epidermal Langerhans cells are derived from cells originating in bone marrow.
Stephen I. Katz;Kunihiko Tamaki;David H. Sachs.
Thymus and activation-regulated chemokine in atopic dermatitis: Serum thymus and activation-regulated chemokine level is closely related with disease activity.
Takashi Kakinuma;Koichiro Nakamura;Motoshi Wakugawa;Hiroshi Mitsui.
The Journal of Allergy and Clinical Immunology (2001)
Characterization of SIS3, a novel specific inhibitor of Smad3, and its effect on transforming growth factor-β1-induced extracellular matrix expression
Masatoshi Jinnin;Hironobu Ihn;Kunihiko Tamaki.
Molecular Pharmacology (2006)
Overproduction of Th2-specific chemokines in NC/Nga mice exhibiting atopic dermatitis–like lesions
Christian Vestergaard;Hiroyuki Yoneyama;Masako Murai;Kohichiro Nakamura.
Journal of Clinical Investigation (1999)
Regulatory B cells (B10 cells) have a suppressive role in murine lupus: CD19 and B10 cell deficiency exacerbates systemic autoimmunity.
Rei Watanabe;Nobuko Ishiura;Hiroko Nakashima;Yoshihiro Kuwano.
Journal of Immunology (2010)
Estrogen enhances immunoglobulin production by human PBMCs
Naoko Kanda;Kunihiko Tamaki.
The Journal of Allergy and Clinical Immunology (1999)
Thymus and activation regulated chemokine (TARC)/CCL17 and skin diseases.
Hidehisa Saeki;Kunihiko Tamaki.
Journal of Dermatological Science (2006)
Impaired Smad7-Smurf–mediated negative regulation of TGF-β signaling in scleroderma fibroblasts
Yoshihide Asano;Hironobu Ihn;Kenichi Yamane;Masahide Kubo.
Journal of Clinical Investigation (2004)
Increased Expression of Integrin αvβ3 Contributes to the Establishment of Autocrine TGF-β Signaling in Scleroderma Fibroblasts
Yoshihide Asano;Hironobu Ihn;Kenichi Yamane;Masatoshi Jinnin.
Journal of Immunology (2005)
Ia Antigens in Mouse Skin Are Predominantly Expressed on Langerhans Cells
Kunihiko Tamaki;George Stingl;Marisa Gullino;David H. Sachs.
Journal of Immunology (1979)
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