His scientific interests lie mostly in Immunology, Flagellin, TLR5, Microbiology and Proinflammatory cytokine. His Immunology study frequently links to adjacent areas such as Inflammatory bowel disease. In his research, Interleukin 18, Interferon, Interleukin 22 and Dominant negative is intimately related to Acquired immune system, which falls under the overarching field of Flagellin.
Andrew T. Gewirtz combines subjects such as Molecular biology, Flagellum and Immunity with his study of TLR5. The concepts of his Proinflammatory cytokine study are interwoven with issues in Lymphocyte differentiation, Endocrinology, Gene expression and Intracellular. The Innate immune system study combines topics in areas such as Gut flora and Disease.
His primary areas of investigation include Immunology, Flagellin, TLR5, Inflammation and Microbiology. Immunology is closely attributed to Inflammatory bowel disease in his study. His Flagellin study integrates concerns from other disciplines, such as Acquired immune system, Immunity, Molecular biology, Cell biology and Proinflammatory cytokine.
As part of the same scientific family, Andrew T. Gewirtz usually focuses on Inflammation, concentrating on Endocrinology and intersecting with Inulin. His Microbiology research is multidisciplinary, relying on both Flagellum, Salmonella and Bacteria. His research integrates issues of Matrix metalloproteinase and Intestinal mucosa in his study of Colitis.
The scientist’s investigation covers issues in Gut flora, Immunology, Internal medicine, Inflammation and Microbiology. His work focuses on many connections between Gut flora and other disciplines, such as Microbiome, that overlap with his field of interest in Transplantation. His work deals with themes such as Disease and Flagellin, which intersect with Immunology.
His research in Internal medicine intersects with topics in Gastroenterology and Endocrinology. His research investigates the connection between Microbiology and topics such as Virulence that intersect with issues in Pathogen. He interconnects Acquired immune system, Proinflammatory cytokine and Signal transduction in the investigation of issues within Innate immune system.
Andrew T. Gewirtz mainly focuses on Gut flora, Immunology, Inflammation, Colitis and Endocrinology. His Gut flora research incorporates elements of Receptor, Lipopolysaccharide, Vitamin D and neurology and Microbiology. Andrew T. Gewirtz has included themes like Disease and Flagellin in his Immunology study.
His Inflammation research is multidisciplinary, incorporating perspectives in Regulatory T cell, Interleukin, Metabolic syndrome and Signal transduction. His studies in Colitis integrate themes in fields like Feces, Gene expression, Inflammatory bowel disease, Human microbiome and Proinflammatory cytokine. The study incorporates disciplines such as Internal medicine, Inulin and Programmed cell death in addition to Endocrinology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Metabolic Syndrome and Altered Gut Microbiota in Mice Lacking Toll-Like Receptor 5
Matam Vijay-Kumar;Jesse D. Aitken;Frederic A. Carvalho;Tyler C. Cullender.
Science (2010)
Cutting Edge: Bacterial Flagellin Activates Basolaterally Expressed TLR5 to Induce Epithelial Proinflammatory Gene Expression
Andrew T. Gewirtz;Tony A. Navas;Sean Lyons;Paul J. Godowski.
Journal of Immunology (2001)
Dietary emulsifiers impact the mouse gut microbiota promoting colitis and metabolic syndrome
Benoit Chassaing;Omry Koren;Julia K. Goodrich;Angela C. Poole.
Nature (2015)
Prokaryotic Regulation of Epithelial Responses by Inhibition of IκB-α Ubiquitination
Andrew S. Neish;Andrew T. Gewirtz;Hui Zeng;Andrew N. Young.
Science (2000)
Bacteria penetrate the normally impenetrable inner colon mucus layer in both murine colitis models and patients with ulcerative colitis
Malin E V Johansson;Jenny K Gustafsson;Jessica Holmén-Larsson;Karolina S Jabbar.
Gut (2014)
Cutting Edge: Impaired Toll-Like Receptor Expression and Function in Aging
Mary Renshaw;Julie Rockwell;Carrie Engleman;Andrew Gewirtz.
Journal of Immunology (2002)
Regulation of Legionella phagosome maturation and infection through flagellin and host Ipaf.
Amal Amer;Luigi Franchi;Thirumala Devi Kanneganti;Mathilde Body-Malapel.
Journal of Biological Chemistry (2006)
Dysregulation of Inflammatory Responses by Chronic Circadian Disruption
Oscar Castanon-Cervantes;Mingwei Wu;J. Christopher Ehlen;Ketema Paul.
Journal of Immunology (2010)
Salmonella typhimurium translocates flagellin across intestinal epithelia, inducing a proinflammatory response
Andrew T. Gewirtz;Peter O. Simon;Clare K. Schmitt;Laura J. Taylor.
Journal of Clinical Investigation (2001)
Deletion of TLR5 results in spontaneous colitis in mice.
Matam Vijay-Kumar;Catherine J. Sanders;Rebekah T. Taylor;Amrita Kumar.
Journal of Clinical Investigation (2007)
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