The scientist’s investigation covers issues in Inflammatory bowel disease, Immunology, Colitis, Inflammation and Internal medicine. His work carried out in the field of Immunology brings together such families of science as Intestinal mucosa, Matrix metalloproteinase, Flagellin and Disease. His Colitis study integrates concerns from other disciplines, such as Pharmacology, Pathogenesis and Ulcerative colitis.
Shanthi V. Sitaraman has included themes like Molecular biology, Biochemistry and Cell biology in his Inflammation study. The Internal medicine study combines topics in areas such as Endocrinology and Signal transduction. Shanthi V. Sitaraman works mostly in the field of Endocrinology, limiting it down to topics relating to Proinflammatory cytokine and, in certain cases, TLR5, as a part of the same area of interest.
Shanthi V. Sitaraman mostly deals with Cell biology, Internal medicine, Colitis, Immunology and Molecular biology. His Cell biology research integrates issues from Receptor, Intestinal mucosa and Adenosine. His Internal medicine study incorporates themes from Gastroenterology, Endocrinology and Motility.
His research in Colitis intersects with topics in Cancer research, Inflammation, Proinflammatory cytokine, Dextran sodium sulfate and Pharmacology. His study in Immunology is interdisciplinary in nature, drawing from both Flagellin and Acute colitis, Inflammatory bowel disease. Shanthi V. Sitaraman focuses mostly in the field of Molecular biology, narrowing it down to topics relating to Transfection and, in certain cases, Cytokine.
Shanthi V. Sitaraman mainly focuses on Cancer research, Colitis, Internal medicine, Cell biology and Immunology. His Colitis research also works with subjects such as
His studies in Endocrinology integrate themes in fields like Glial cell line-derived neurotrophic factor and Toll-like receptor. His Cell biology study deals with microRNA intersecting with Molecular biology, Intestinal epithelial cell differentiation, Microarray analysis techniques, Gene expression and Regulation of gene expression. His studies deal with areas such as Metabolic syndrome and Intestinal inflammation as well as Immunology.
His main research concerns Colitis, Inflammatory bowel disease, Proinflammatory cytokine, Immunology and Cancer research. His Colitis research is multidisciplinary, incorporating perspectives in Inflammation, Dextran, Biochemistry and Tumor necrosis factor alpha. His Inflammatory bowel disease research is multidisciplinary, relying on both In vivo, Intestinal mucosa, Pathogenesis and Ulcerative colitis.
The concepts of his Proinflammatory cytokine study are interwoven with issues in Carcinogenesis and Molecular biology. His Molecular biology research incorporates elements of Gene silencing, microRNA, Transfection and Cell biology. His biological study spans a wide range of topics, including Regulation of gene expression and Insulin resistance.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Metabolic Syndrome and Altered Gut Microbiota in Mice Lacking Toll-Like Receptor 5
Matam Vijay-Kumar;Jesse D. Aitken;Frederic A. Carvalho;Tyler C. Cullender.
Orally delivered thioketal nanoparticles loaded with TNF-α–siRNA target inflammation and inhibit gene expression in the intestines
D. Scott Wilson;Guillaume Dalmasso;Lixin Wang;Shanthi V. Sitaraman.
Nature Materials (2010)
Deletion of TLR5 results in spontaneous colitis in mice.
Matam Vijay-Kumar;Catherine J. Sanders;Rebekah T. Taylor;Amrita Kumar.
Journal of Clinical Investigation (2007)
Temporal and Spatial Analysis of Clinical and Molecular Parameters in Dextran Sodium Sulfate Induced Colitis
Yutao Yan;Vasantha Kolachala;Guillaume Dalmasso;Hang Nguyen.
PLOS ONE (2009)
Dextran Sodium Sulfate (DSS) Induces Colitis in Mice by Forming Nano-Lipocomplexes with Medium-Chain-Length Fatty Acids in the Colon
Hamed Laroui;Sarah A. Ingersoll;Sarah A. Ingersoll;Hong Chun Liu;Hong Chun Liu;Mark T. Baker.
PLOS ONE (2012)
Gut microbial products regulate murine gastrointestinal motility via Toll-like receptor 4 signaling.
Mallappa Anitha;Matam Vijay–Kumar;Shanthi V. Sitaraman;Andrew T. Gewirtz.
Leptin as a novel profibrogenic cytokine in hepatic stellate cells: mitogenesis and inhibition of apoptosis mediated by extracellular regulated kinase (Erk) and Akt phosphorylation
Neeraj K. Saxena;Mark A. Titus;Xiaokun Ding;Jeffrey Floyd.
The FASEB Journal (2004)
Targeted deletion of metalloproteinase 9 attenuates experimental colitis in mice: central role of epithelial-derived MMP.
Florencia E Castaneda;Baljit Walia;Matam Vijay-Kumar;Neal R Patel.
GDNF rescues hyperglycemia-induced diabetic enteric neuropathy through activation of the PI3K/Akt pathway
Mallappa Anitha;Chetan Gondha;Roy Sutliff;Alexander Parsadanian.
Journal of Clinical Investigation (2006)
IL-6 induces NF-kappa B activation in the intestinal epithelia.
Lixin Wang;Baljit Walia;John Evans;Andrew T. Gewirtz.
Journal of Immunology (2003)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: