D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 62 Citations 13,012 216 World Ranking 7050 National Ranking 3269

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Cancer

The scientist’s investigation covers issues in Inflammatory bowel disease, Immunology, Colitis, Inflammation and Internal medicine. His work carried out in the field of Immunology brings together such families of science as Intestinal mucosa, Matrix metalloproteinase, Flagellin and Disease. His Colitis study integrates concerns from other disciplines, such as Pharmacology, Pathogenesis and Ulcerative colitis.

Shanthi V. Sitaraman has included themes like Molecular biology, Biochemistry and Cell biology in his Inflammation study. The Internal medicine study combines topics in areas such as Endocrinology and Signal transduction. Shanthi V. Sitaraman works mostly in the field of Endocrinology, limiting it down to topics relating to Proinflammatory cytokine and, in certain cases, TLR5, as a part of the same area of interest.

His most cited work include:

  • Metabolic Syndrome and Altered Gut Microbiota in Mice Lacking Toll-Like Receptor 5 (1499 citations)
  • Orally delivered thioketal nanoparticles loaded with TNF-α–siRNA target inflammation and inhibit gene expression in the intestines (406 citations)
  • Deletion of TLR5 results in spontaneous colitis in mice. (361 citations)

What are the main themes of his work throughout his whole career to date?

Shanthi V. Sitaraman mostly deals with Cell biology, Internal medicine, Colitis, Immunology and Molecular biology. His Cell biology research integrates issues from Receptor, Intestinal mucosa and Adenosine. His Internal medicine study incorporates themes from Gastroenterology, Endocrinology and Motility.

His research in Colitis intersects with topics in Cancer research, Inflammation, Proinflammatory cytokine, Dextran sodium sulfate and Pharmacology. His study in Immunology is interdisciplinary in nature, drawing from both Flagellin and Acute colitis, Inflammatory bowel disease. Shanthi V. Sitaraman focuses mostly in the field of Molecular biology, narrowing it down to topics relating to Transfection and, in certain cases, Cytokine.

He most often published in these fields:

  • Cell biology (30.53%)
  • Internal medicine (21.68%)
  • Colitis (18.14%)

What were the highlights of his more recent work (between 2009-2016)?

  • Cancer research (15.49%)
  • Colitis (18.14%)
  • Internal medicine (21.68%)

In recent papers he was focusing on the following fields of study:

Shanthi V. Sitaraman mainly focuses on Cancer research, Colitis, Internal medicine, Cell biology and Immunology. His Colitis research also works with subjects such as

  • Inflammation, which have a strong connection to Tumor necrosis factor alpha, Immune system and Endogeny,
  • Inflammatory bowel disease and related Ulcerative colitis, Intestinal mucosa, Pathogenesis and In vivo. His Internal medicine study combines topics from a wide range of disciplines, such as Gastroenterology and Endocrinology.

His studies in Endocrinology integrate themes in fields like Glial cell line-derived neurotrophic factor and Toll-like receptor. His Cell biology study deals with microRNA intersecting with Molecular biology, Intestinal epithelial cell differentiation, Microarray analysis techniques, Gene expression and Regulation of gene expression. His studies deal with areas such as Metabolic syndrome and Intestinal inflammation as well as Immunology.

Between 2009 and 2016, his most popular works were:

  • Metabolic Syndrome and Altered Gut Microbiota in Mice Lacking Toll-Like Receptor 5 (1499 citations)
  • Orally delivered thioketal nanoparticles loaded with TNF-α–siRNA target inflammation and inhibit gene expression in the intestines (406 citations)
  • Gut microbial products regulate murine gastrointestinal motility via Toll-like receptor 4 signaling. (204 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Cancer

His main research concerns Colitis, Inflammatory bowel disease, Proinflammatory cytokine, Immunology and Cancer research. His Colitis research is multidisciplinary, incorporating perspectives in Inflammation, Dextran, Biochemistry and Tumor necrosis factor alpha. His Inflammatory bowel disease research is multidisciplinary, relying on both In vivo, Intestinal mucosa, Pathogenesis and Ulcerative colitis.

The concepts of his Proinflammatory cytokine study are interwoven with issues in Carcinogenesis and Molecular biology. His Molecular biology research incorporates elements of Gene silencing, microRNA, Transfection and Cell biology. His biological study spans a wide range of topics, including Regulation of gene expression and Insulin resistance.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Metabolic Syndrome and Altered Gut Microbiota in Mice Lacking Toll-Like Receptor 5

Matam Vijay-Kumar;Jesse D. Aitken;Frederic A. Carvalho;Tyler C. Cullender.
Science (2010)

2162 Citations

Orally delivered thioketal nanoparticles loaded with TNF-α–siRNA target inflammation and inhibit gene expression in the intestines

D. Scott Wilson;Guillaume Dalmasso;Lixin Wang;Shanthi V. Sitaraman.
Nature Materials (2010)

607 Citations

Deletion of TLR5 results in spontaneous colitis in mice.

Matam Vijay-Kumar;Catherine J. Sanders;Rebekah T. Taylor;Amrita Kumar.
Journal of Clinical Investigation (2007)

465 Citations

Temporal and Spatial Analysis of Clinical and Molecular Parameters in Dextran Sodium Sulfate Induced Colitis

Yutao Yan;Vasantha Kolachala;Guillaume Dalmasso;Hang Nguyen.
PLOS ONE (2009)

425 Citations

Dextran Sodium Sulfate (DSS) Induces Colitis in Mice by Forming Nano-Lipocomplexes with Medium-Chain-Length Fatty Acids in the Colon

Hamed Laroui;Sarah A. Ingersoll;Sarah A. Ingersoll;Hong Chun Liu;Hong Chun Liu;Mark T. Baker.
PLOS ONE (2012)

344 Citations

Gut microbial products regulate murine gastrointestinal motility via Toll-like receptor 4 signaling.

Mallappa Anitha;Matam Vijay–Kumar;Shanthi V. Sitaraman;Andrew T. Gewirtz.
Gastroenterology (2012)

307 Citations

Leptin as a novel profibrogenic cytokine in hepatic stellate cells: mitogenesis and inhibition of apoptosis mediated by extracellular regulated kinase (Erk) and Akt phosphorylation

Neeraj K. Saxena;Mark A. Titus;Xiaokun Ding;Jeffrey Floyd.
The FASEB Journal (2004)

289 Citations

Targeted deletion of metalloproteinase 9 attenuates experimental colitis in mice: central role of epithelial-derived MMP.

Florencia E Castaneda;Baljit Walia;Matam Vijay-Kumar;Neal R Patel.
Gastroenterology (2005)

284 Citations

GDNF rescues hyperglycemia-induced diabetic enteric neuropathy through activation of the PI3K/Akt pathway

Mallappa Anitha;Chetan Gondha;Roy Sutliff;Alexander Parsadanian.
Journal of Clinical Investigation (2006)

259 Citations

IL-6 induces NF-kappa B activation in the intestinal epithelia.

Lixin Wang;Baljit Walia;John Evans;Andrew T. Gewirtz.
Journal of Immunology (2003)

243 Citations

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