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Immunology

D-Index
45
Citations
10000
World Ranking
4677
National Ranking
222

Overview

Mustapha Si-Tahar is affiliated with François Rabelais University in France. Their research spans medicine, immunology, and microbiology, with particular focus in subfields such as immunology, pulmonary and respiratory medicine, infectious diseases, epidemiology, and molecular biology.

Their work frequently addresses topics including:

  • Immune Cell Function and Interaction
  • Immune Response and Inflammation
  • Influenza Virus Research Studies
  • COVID-19 Clinical Research Studies
  • SARS-CoV-2 and COVID-19 Research
  • Respiratory Support and Mechanisms
  • Immune cells in cancer

Among the recent publications authored by or involving Mustapha Si-Tahar are:

  • Outcome of SARS-CoV-2 infection is linked to MAIT cell activation and cytotoxicity, 2021, Nature Immunology
  • Phenotypical and functional alteration of unconventional T cells in severe COVID-19 patients, 2020, The Journal of Experimental Medicine
  • High Dimensional Single-Cell Analysis Reveals iNKT Cell Developmental Trajectories and Effector Fate Decision, 2020, Cell Reports
  • Host succinate inhibits influenza virus infection through succinylation and nuclear retention of the viral nucleoprotein, 2022, The EMBO Journal
  • Lung compartmentalization of inflammatory biomarkers in COVID-19-related ARDS, 2021, Critical Care

Frequent co-authors collaborating with Mustapha Si-Tahar include:

  • Antoine Guillon
  • Christophe Paget
  • Adeline Cezard
  • Virginie Vasseur
  • Thomas Baranek

Publications have appeared repeatedly in venues such as:

  • Revue des Maladies Respiratoires
  • bioRxiv (Cold Spring Harbor Laboratory)
  • Journal of Cystic Fibrosis
  • The EMBO Journal
  • 10.01 - Respiratory infections and bronchiectasis

Best Publications

  • Involvement of Toll-like Receptor 3 in the Immune Response of Lung Epithelial Cells to Double-stranded RNA and Influenza A Virus *

    Loïc Guillot;Ronan Le Goffic;Sarah Bloch;Nicolas Escriou

  • Detrimental contribution of the Toll-like receptor (TLR)3 to influenza A virus-induced acute pneumonia.

    Ronan Le Goffic;Ronan Le Goffic;Viviane Balloy;Viviane Balloy;Micheline Lagranderie;Lena Alexopoulou;Lena Alexopoulou

  • In vivo equilibrium of proinflammatory IL-17+ and regulatory IL-10+ Foxp3+ RORγt+ T cells

    Matthias Lochner;Lucie Peduto;Marie Cherrier;Shinichiro Sawa

  • Cutting Edge: Influenza A virus activates TLR3-dependent inflammatory and RIG-I-dependent antiviral responses in human lung epithelial cells.

    Ronan Le Goffic;Ronan Le Goffic;Julien Pothlichet;Julien Pothlichet;Damien Vitour;Takashi Fujita

  • Response of Human Pulmonary Epithelial Cells to Lipopolysaccharide Involves Toll-like Receptor 4 (TLR4)-dependent Signaling Pathways EVIDENCE FOR AN INTRACELLULAR COMPARTMENTALIZATION OF TLR4

    Loïc Guillot;Samir Medjane;Karine Le-Barillec;Viviane Balloy

  • Cutting Edge: The Immunostimulatory Activity of the Lung Surfactant Protein-A Involves Toll-Like Receptor 4

    Loïc Guillot;Viviane Balloy;Francis X. McCormack;Douglas T. Golenbock

  • Microbiota-induced tertiary lymphoid tissues aggravate inflammatory disease in the absence of RORγt and LTi cells

    Matthias Lochner;Caspar Ohnmacht;Caspar Ohnmacht;Laura Presley;Laura Presley;Pierre Bruhns;Pierre Bruhns

  • Innate sensing of HIV-infected cells.

    Alice Lepelley;Stephanie Louis;Marion Sourisseau;Helen K. W. Law

  • Neutrophil-epithelial crosstalk at the intestinal lumenal surface mediated by reciprocal secretion of adenosine and IL-6

    Shanthi V. Sitaraman;Didier Merlin;Lixin Wang;Michelle Wong

  • Colonic epithelial hPepT1 expression occurs in inflammatory bowel disease: Transport of bacterial peptides influences expression of MHC class 1 molecules

    Didier Merlin;Mustapha Si-Tahar;Shanthi V. Sitaraman;Karen Eastburn

  • Outcome of SARS-CoV-2 infection is linked to MAIT cell activation and cytotoxicity.

    Héloïse Flament;Héloïse Flament;Matthieu Rouland;Lucie Beaudoin;Amine Toubal

  • Cutting Edge: Innate Immune Response Triggered by Influenza A Virus Is Negatively Regulated by SOCS1 and SOCS3 through a RIG-I/IFNAR1-Dependent Pathway

    Julien Pothlichet;Michel Chignard;Mustapha Si-Tahar

  • Interleukin-22 Is Produced by Invariant Natural Killer T Lymphocytes during Influenza A Virus Infection: POTENTIAL ROLE IN PROTECTION AGAINST LUNG EPITHELIAL DAMAGES*

    Christophe Paget;Stoyan Ivanov;Josette Fontaine;Joelle Renneson

  • Phenotypical and functional alteration of unconventional T cells in severe COVID-19 patients.

    Youenn Jouan;Antoine Guillon;Antoine Guillon;Loïc Gonzalez;Loïc Gonzalez;Yonatan Perez;Yonatan Perez

  • Interleukin-22 Reduces Lung Inflammation during Influenza A Virus Infection and Protects against Secondary Bacterial Infection

    Stoyan Ivanov;Joelle Renneson;Josette Fontaine;Adeline Barthelemy

  • Specific inhibition of thrombin-induced cell activation by the neutrophil proteinases elastase, cathepsin G, and proteinase 3: evidence for distinct cleavage sites within the aminoterminal domain of the thrombin receptor.

    Patricia Renesto;Mustapha Si-Tahar;Marc Moniatte;Viviane Balloy

  • Proteolysis of monocyte CD14 by human leukocyte elastase inhibits lipopolysaccharide-mediated cell activation

    Karine Le-Barillec;Mustapha Si-Tahar;Viviane Balloy;Michel Chignard

  • Control of Pseudomonas aeruginosa in the lung requires the recognition of either lipopolysaccharide or flagellin.

    Reuben Ramphal;Viviane Balloy;Viviane Balloy;Jeevan Jyot;Amrisha Verma

  • Involvement of toll-like receptor 2 in experimental invasive pulmonary aspergillosis.

    Viviane Balloy;Mustapha Si-Tahar;Osamu Takeuchi;Bruno Philippe

  • A loss‐of‐function variant of the antiviral molecule MAVS is associated with a subset of systemic lupus patients

    Julien Pothlichet;Julien Pothlichet;Julien Pothlichet;Timothy B. Niewold;Damien Vitour;Damien Vitour;Damien Vitour;Brigitte Solhonne;Brigitte Solhonne

  • FHL2 Regulates Natural Killer Cell Development and Activation during Streptococcus pneumoniae Infection

    Thomas Baranek;Thomas Baranek;Eric Morello;Eric Morello;Alexandre Valayer;Alexandre Valayer;Rose-France Aimar;Rose-France Aimar

Frequent Co-Authors

Michel Chignard
Michel Chignard Institut Pasteur
Viviane Balloy
Viviane Balloy Sorbonne University
Reuben Ramphal
Reuben Ramphal University of Florida
James L. Madara
James L. Madara American Medical Association
François Trottein
François Trottein Institut Pasteur
Shanthi V. Sitaraman
Shanthi V. Sitaraman Emory University
Philippe Gosset
Philippe Gosset Institut Pasteur
Didier Merlin
Didier Merlin Georgia State University
Gérard Eberl
Gérard Eberl Université Paris Cité
Jean-Michel Sallenave
Jean-Michel Sallenave Université Paris Cité

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