Immunology, Influenza A virus, Inflammation, TLR3 and Proinflammatory cytokine are his primary areas of study. His study in the field of Interleukin 21 is also linked to topics like FHL2. His study in Influenza A virus is interdisciplinary in nature, drawing from both Innate immune system, Interleukin 22 and Cytokine.
Mustapha Si-Tahar interconnects CD8, Innate lymphoid cell and Pathogenesis in the investigation of issues within Inflammation. His TLR3 research is multidisciplinary, relying on both RIG-I, T cell and Mitochondrial antiviral-signaling protein. His work carried out in the field of Proinflammatory cytokine brings together such families of science as RAR-related orphan receptor gamma and Endocrinology.
Mustapha Si-Tahar mainly focuses on Immunology, Inflammation, Microbiology, Immune system and Signal transduction. His research links Lung with Immunology. Mustapha Si-Tahar focuses mostly in the field of Inflammation, narrowing it down to topics relating to Cystic fibrosis and, in certain cases, Pathology.
Mustapha Si-Tahar studied Immune system and Cell that intersect with Effector. The Signal transduction study which covers Toll-like receptor that intersects with Aspergillus fumigatus. His Influenza A virus research incorporates elements of Natural killer T cell, CD8, TLR3 and Viral replication.
His scientific interests lie mostly in Immunology, Immune system, Lung, Inflammation and Pseudomonas aeruginosa. Mustapha Si-Tahar works on Immunology which deals in particular with Interleukin. His work deals with themes such as Receptor and Respiratory infection, which intersect with Interleukin.
His Immune system study incorporates themes from Regulator, Cell biology, Effector and Cytokine. The study incorporates disciplines such as Cell activation, Cytotoxic T cell, Proinflammatory cytokine and Monocyte in addition to Cytokine. Mustapha Si-Tahar has researched Pseudomonas aeruginosa in several fields, including Bronchoalveolar lavage, Cystic fibrosis and Microbiology.
His main research concerns Immunology, Immune system, Lung, Inflammation and Pneumonia. His Immunology study combines topics from a wide range of disciplines, such as ARDS and Airway. His ARDS research is multidisciplinary, incorporating elements of T cell, Pathophysiology and Systemic inflammation.
His Airway research includes themes of Pseudomonas aeruginosa, Pharmacokinetics, Antibiotic resistance and Toxicity. Mustapha Si-Tahar combines subjects such as Cell activation, Monocyte, Proinflammatory cytokine, Cytotoxic T cell and Macrophage with his study of Cytokine. His Antibody study combines topics in areas such as Homeostasis, RAR-related orphan receptor gamma, Immunity, Neutrophilia and Interleukin.
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Involvement of Toll-like Receptor 3 in the Immune Response of Lung Epithelial Cells to Double-stranded RNA and Influenza A Virus *
Loïc Guillot;Ronan Le Goffic;Sarah Bloch;Nicolas Escriou.
Journal of Biological Chemistry (2005)
Detrimental contribution of the Toll-like receptor (TLR)3 to influenza A virus-induced acute pneumonia.
Ronan Le Goffic;Ronan Le Goffic;Viviane Balloy;Viviane Balloy;Micheline Lagranderie;Lena Alexopoulou;Lena Alexopoulou.
PLOS Pathogens (2006)
In vivo equilibrium of proinflammatory IL-17+ and regulatory IL-10+ Foxp3+ RORγt+ T cells
Matthias Lochner;Lucie Peduto;Marie Cherrier;Shinichiro Sawa.
Journal of Experimental Medicine (2008)
Cutting Edge: Influenza A virus activates TLR3-dependent inflammatory and RIG-I-dependent antiviral responses in human lung epithelial cells.
Ronan Le Goffic;Ronan Le Goffic;Julien Pothlichet;Julien Pothlichet;Damien Vitour;Takashi Fujita.
Journal of Immunology (2007)
Response of Human Pulmonary Epithelial Cells to Lipopolysaccharide Involves Toll-like Receptor 4 (TLR4)-dependent Signaling Pathways EVIDENCE FOR AN INTRACELLULAR COMPARTMENTALIZATION OF TLR4
Loïc Guillot;Samir Medjane;Karine Le-Barillec;Viviane Balloy.
Journal of Biological Chemistry (2004)
Cutting Edge: The Immunostimulatory Activity of the Lung Surfactant Protein-A Involves Toll-Like Receptor 4
Loïc Guillot;Viviane Balloy;Francis X. McCormack;Douglas T. Golenbock.
Journal of Immunology (2002)
Microbiota-induced tertiary lymphoid tissues aggravate inflammatory disease in the absence of RORγt and LTi cells
Matthias Lochner;Caspar Ohnmacht;Caspar Ohnmacht;Laura Presley;Laura Presley;Pierre Bruhns;Pierre Bruhns.
Journal of Experimental Medicine (2011)
Innate sensing of HIV-infected cells.
Alice Lepelley;Stephanie Louis;Marion Sourisseau;Helen K. W. Law.
PLOS Pathogens (2011)
Neutrophil-epithelial crosstalk at the intestinal lumenal surface mediated by reciprocal secretion of adenosine and IL-6
Shanthi V. Sitaraman;Didier Merlin;Lixin Wang;Michelle Wong.
Journal of Clinical Investigation (2001)
Colonic epithelial hPepT1 expression occurs in inflammatory bowel disease: Transport of bacterial peptides influences expression of MHC class 1 molecules
Didier Merlin;Mustapha Si-Tahar;Shanthi V. Sitaraman;Karen Eastburn.
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