The scientist’s investigation covers issues in Internal medicine, Fibronectin, Endocrinology, Idiopathic pulmonary fibrosis and Lung. His studies in Fibronectin integrate themes in fields like Molecular biology, Signal transduction, Cell growth and Nicotine. His work on Transforming growth factor is typically connected to In utero as part of general Endocrinology study, connecting several disciplines of science.
His Idiopathic pulmonary fibrosis research is multidisciplinary, relying on both Fibrosis, Pulmonary fibrosis, Exacerbation and Intensive care medicine. His Pulmonary fibrosis study is focused on Pathology in general. His work carried out in the field of Lung brings together such families of science as Disease progression and Incidence.
Jesse Roman mostly deals with Lung, Fibronectin, Pathology, Immunology and Cell biology. His Lung research is under the purview of Internal medicine. Jesse Roman focuses mostly in the field of Fibronectin, narrowing it down to matters related to Cancer research and, in some cases, Kinase, Cell growth, MAPK/ERK pathway, Lung cancer and Signal transduction.
His study in Pathology is interdisciplinary in nature, drawing from both Respiratory disease, Idiopathic pulmonary fibrosis and Transplantation. His Cell biology research includes themes of Receptor, Morphogenesis and Downregulation and upregulation. The Lung injury study which covers Endocrinology that intersects with Nicotine and Matrix metalloproteinase.
Jesse Roman mainly focuses on Lung, Idiopathic pulmonary fibrosis, Pulmonary fibrosis, Internal medicine and Immunology. His Lung research is multidisciplinary, incorporating perspectives in Inflammation, Pulmonary hypertension and Fibrosis, Pathology. His biological study spans a wide range of topics, including Interstitial lung disease, Integrin and Intensive care medicine.
The various areas that Jesse Roman examines in his Internal medicine study include Gastroenterology and Endocrinology. Jesse Roman interconnects Lung cancer, Signal transduction and Scleroderma in the investigation of issues within Immunology. His Signal transduction research also works with subjects such as
Pulmonary fibrosis, Lung, Idiopathic pulmonary fibrosis, Immunology and Pathology are his primary areas of study. His Pulmonary fibrosis study combines topics from a wide range of disciplines, such as Patient advocacy, Natural history and Gerontology. His Lung research is multidisciplinary, incorporating elements of Psychological intervention and Pulmonary hypertension.
His studies deal with areas such as Hermansky–Pudlak syndrome, Proteome and Intensive care medicine as well as Idiopathic pulmonary fibrosis. The Immunology study combines topics in areas such as Lung cancer, Signal transduction, Bioinformatics and Proteomics. Jesse Roman studied Pathology and Inflammation that intersect with Molecular biology, Oxidative stress and COPD.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Acute Exacerbations of Idiopathic Pulmonary Fibrosis
Harold R. Collard;Bethany B. Moore;Kevin R. Flaherty;Kevin K. Brown.
American Journal of Respiratory and Critical Care Medicine (2007)
Bone marrow-derived mesenchymal stem cells in repair of the injured lung.
Mauricio Rojas;Jianguo Xu;Charles R. Woods;Ana L. Mora.
American Journal of Respiratory Cell and Molecular Biology (2005)
Peroxisome Proliferator-Activated Receptor γ Ligands Increase Release of Nitric Oxide From Endothelial Cells
David S. Calnek;Louis Mazzella;Susanne Roser;Jesse Roman.
Arteriosclerosis, Thrombosis, and Vascular Biology (2003)
Targeted deletion of metalloproteinase 9 attenuates experimental colitis in mice: central role of epithelial-derived MMP.
Florencia E Castaneda;Baljit Walia;Matam Vijay-Kumar;Neal R Patel.
Assessing the carcinogenic potential of low-dose exposures to chemical mixtures in the environment: The challenge ahead
William H Goodson;Leroy Lowe;David O Carpenter;Michael Gilbertson.
Fibronectin Stimulates Non–Small Cell Lung Carcinoma Cell Growth through Activation of Akt/Mammalian Target of Rapamycin/S6 Kinase and Inactivation of LKB1/AMP-Activated Protein Kinase Signal Pathways
ShouWei Han;Fadlo R. Khuri;Jesse Roman.
Cancer Research (2006)
Activation of Alveolar Macrophages via the Alternative Pathway in Herpesvirus-Induced Lung Fibrosis
Ana L. Mora;Edilson Torres-González;Mauricio Rojas;Claudia Corredor.
American Journal of Respiratory Cell and Molecular Biology (2006)
Declining Bone Mass in Men With Chronic Pulmonary Disease: Contribution of Glucocorticoid Treatment, Body Mass Index, and Gonadal Function
Farrukh Iqbal;Jeffrey Michaelson;Leonard Thaler;Janet Rubin.
Future directions in idiopathic pulmonary fibrosis research. An NHLBI workshop report.
Timothy S. Blackwell;Andrew M. Tager;Zea Borok;Bethany B. Moore.
American Journal of Respiratory and Critical Care Medicine (2014)
Vitamin D inhibition of pro-fibrotic effects of transforming growth factor β1 in lung fibroblasts and epithelial cells
Allan M. Ramirez;Cherry Wongtrakool;Teresa Welch;Andreas Steinmeyer.
The Journal of Steroid Biochemistry and Molecular Biology (2010)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: