Richard J. Gregory spends much of his time researching Cystic fibrosis transmembrane conductance regulator, Chloride channel, Cystic fibrosis, Cell biology and Molecular biology. Richard J. Gregory merges many fields, such as Cystic fibrosis transmembrane conductance regulator and Membrane protein, in his writings. As a part of the same scientific family, he mostly works in the field of Chloride channel, focusing on Mutation and, on occasion, Apical membrane and Transfection.
His Cystic fibrosis research integrates issues from Endocrinology and Mutant. His Cell biology research incorporates elements of Angiogenesis and Immunology. Richard J. Gregory has included themes like Genetic enhancement and Heterologous expression in his Molecular biology study.
Molecular biology, Cystic fibrosis transmembrane conductance regulator, Genetic enhancement, Cell biology and Vector are his primary areas of study. The concepts of his Molecular biology study are interwoven with issues in Transgene, Recombinant DNA, Expression vector, Viral vector and Gene. The Cystic fibrosis transmembrane conductance regulator study combines topics in areas such as Chloride channel, Peptide sequence and Mutant.
His Genetic enhancement research is multidisciplinary, relying on both Tropism, Cancer research, Vectors in gene therapy and Virology. His Cell biology study integrates concerns from other disciplines, such as Binding domain, Transcription factor, Transactivation, Transfection and Immunology. His Cystic fibrosis research is multidisciplinary, relying on both Apical membrane and Endocrinology.
The scientist’s investigation covers issues in Molecular biology, Transgene, Antibody, Pharmacology and Cell biology. His Molecular biology research is multidisciplinary, incorporating elements of Gene expression, Gene, Vector, Cytochrome c and Viral vector. His work in Vector addresses subjects such as Insert, which are connected to disciplines such as Recombinant DNA.
His work carried out in the field of Transgene brings together such families of science as Reactive oxygen species, Cardiotoxicity, Genetic enhancement and Immune system. He combines subjects such as Caspase 3, Caspase-9, Caspase 8, Fas ligand and Viability assay with his study of Cell biology. In his study, Immunology is strongly linked to Regulation of gene expression, which falls under the umbrella field of Immune tolerance.
Richard J. Gregory mostly deals with Genetic enhancement, Molecular biology, Transgene, Angiogenesis and Immunology. His study in Genetic enhancement is interdisciplinary in nature, drawing from both Vector, Antibody and Immune system, Immune tolerance. His Molecular biology study combines topics from a wide range of disciplines, such as Tropism, Virus, T cell, Viral vector and Gene delivery.
His Angiopoietin receptor study in the realm of Angiogenesis interacts with subjects such as Vascular endothelial growth factor C and Clear cell. His studies deal with areas such as Ischemic preconditioning, Adenoviridae, Hemangioblastoma and Adenocarcinoma as well as Immunology. His work deals with themes such as Endocrinology and Internal medicine, which intersect with Cell biology.
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Defective intracellular transport and processing of CFTR is the molecular basis of most cystic fibrosis
Seng H. Cheng;Richard J. Gregory;John Marshall;Sucharita Paul.
Demonstration That CFTR Is a Chloride Channel by Alteration of Its Anion Selectivity
Matthew P. Anderson;Richard J. Gregory;Simon Thompson;David W. Souza.
Adenovirus-mediated gene transfer transiently corrects the chloride transport defect in nasal epithelia of patients with cystic fibrosis
Joseph Zabner;Larry A. Couture;Richard J. Gregory;Scott M. Graham.
Expression of cystic fibrosis transmembrane conductance regulator corrects defective chloride channel regulation in cystic fibrosis airway epithelial cells.
Devra P. Rich;Matthew P. Anderson;Richard J. Gregory;Seng H. Cheng.
Generation of cAMP-activated chloride currents by expression of CFTR
Matthew P. Anderson;Devra P. Rich;Richard J. Gregory;Alan E. Smith.
Phosphorylation of the R domain by cAMP-dependent protein kinase regulates the CFTR chloride channel.
S.H. Cheng;D.P. Rich;J. Marshall;R.J. Gregory.
Gene therapy for cystic fibrosis
Richard J. Gregory;Donna Armentano;Larry A. Couture;Alan E. Smith.
Nucleoside triphosphates are required to open the CFTR chloride channel
Matthew P. Anderson;Herbert A. Berger;Devra P. Rich;Richard J. Gregory.
Mutations in CFTR associated with mild-disease-form Cl- channels with altered pore properties.
David N. Sheppard;Devra P. Rich;Lynda S. Ostedgaard;Richard J. Gregory;Richard J. Gregory.
Recombinant adenoviral vector and methods of use
Richard J. Gregory;Ken N. Wills;Daniel C. Maneval.
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