Frédéric Becq mainly investigates Cystic fibrosis transmembrane conductance regulator, Chloride channel, Biochemistry, Cell biology and Cystic fibrosis. His Cystic fibrosis transmembrane conductance regulator study improves the overall literature in Internal medicine. His research in Chloride channel intersects with topics in Transport protein, Wild type, Chloride channel activity, IBMX and Theophylline.
His work in the fields of Biochemistry, such as Secretion, ATP-binding cassette transporter and Chinese hamster ovary cell, intersects with other areas such as Spermatocyte and Sulfate permease. He has included themes like Vascular smooth muscle and Transfection in his Cell biology study. Frédéric Becq interconnects Endoplasmic reticulum, Stereochemistry, Miglustat and Pathology in the investigation of issues within Cystic fibrosis.
His primary scientific interests are in Cystic fibrosis transmembrane conductance regulator, Cystic fibrosis, Cell biology, Chloride channel and Biochemistry. Frédéric Becq combines subjects such as Apical membrane, Molecular biology, Endocrinology and Forskolin with his study of Cystic fibrosis transmembrane conductance regulator. Frédéric Becq focuses mostly in the field of Cystic fibrosis, narrowing it down to topics relating to Mutation and, in certain cases, Phenotype.
His study in Cell biology is interdisciplinary in nature, drawing from both Secretion, Cell culture and Ion transporter. Frédéric Becq focuses mostly in the field of Chloride channel, narrowing it down to matters related to IBMX and, in some cases, Xanthine. His research links Biophysics with Biochemistry.
His primary areas of investigation include Cystic fibrosis, Cell biology, Cystic fibrosis transmembrane conductance regulator, Chloride channel and Mutation. His biological study spans a wide range of topics, including Inflammation, Cancer research, Mutant and Potentiator. His Cell biology study combines topics in areas such as Secretion, Cell and Sertoli cell.
His Cystic fibrosis transmembrane conductance regulator research is multidisciplinary, incorporating elements of Function, Transmembrane protein, Molecular biology, Endoplasmic reticulum and Forskolin. While the research belongs to areas of Chloride channel, Frédéric Becq spends his time largely on the problem of TRPC6, intersecting his research to questions surrounding TRPM7, TRPV4, Tyrosine kinase and Adenylyl cyclase. His studies deal with areas such as Phenotype, Epididymis, Internal medicine and Endocrinology as well as Mutation.
Frédéric Becq spends much of his time researching Cystic fibrosis transmembrane conductance regulator, Cystic fibrosis, Cell biology, Stereochemistry and Function. His studies in Cystic fibrosis transmembrane conductance regulator integrate themes in fields like Transient receptor potential channel, TRPV6, Molecular biology, Endoplasmic reticulum and Western blot. Endoplasmic reticulum is a primary field of his research addressed under Biochemistry.
His biological study spans a wide range of topics, including Mutation, Therapeutic approach, Mutant and Genetic counseling. Frédéric Becq studies Cell biology, namely Mitochondrion. His Function study which covers Patch clamp that intersects with Chloride channel.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Interleukin-1β Secretion Is Impaired by Inhibitors of the Atp Binding Cassette Transporter, ABC1
Yannick Hamon;Marie Françoise Luciani;Frederic Becq;Bernard Verrier.
Rescue of functional delF508-CFTR channels in cystic fibrosis epithelial cells by the α-glucosidase inhibitor miglustat
Caroline Norez;Sabrina Noel;Martina Wilke;Marcel Bijvelds.
FEBS Letters (2006)
9-Phenanthrol inhibits human TRPM4 but not TRPM5 cationic channels
Teddy Grand;Marie Demion;Marie Demion;Caroline Norez;Yvette Mettey.
British Journal of Pharmacology (2008)
ABC1, an ATP Binding Cassette Transporter Required for Phagocytosis of Apoptotic Cells, Generates a Regulated Anion Flux after Expression in Xenopus laevis Oocytes
Frédéric Becq;Yannick Hamon;Adriana Bajetto;Maurice Gola.
Journal of Biological Chemistry (1997)
Correction of delF508-CFTR activity with benzo(c)quinolizinium compounds through facilitation of its processing in cystic fibrosis airway cells
Robert L. Dormer;Renaud Dérand;Ceinwen M. McNeilly;Yvette Mettey.
Journal of Cell Science (2001)
Development of Substituted Benzo[c]quinolizinium Compounds as Novel Activators of the Cystic Fibrosis Chloride Channel
Frédéric Becq;Frédéric Becq;Yvette Mettey;Mike A. Gray;Luis J.V. Galietta.
Journal of Biological Chemistry (1999)
Sildenafil (Viagra) corrects DeltaF508-CFTR location in nasal epithelial cells from patients with cystic fibrosis.
Robert Leslie Dormer;Ceinwen Menai Harris;Zoe Elizabeth Clark;Malcolm Martin Claude Pereira.
Tat1, a Novel Sulfate Transporter Specifically Expressed in Human Male Germ Cells and Potentially Linked to RhoGTPase Signaling *
Aminata Touré;Laurence Morin;Charles Pineau;Frédéric Becq.
Journal of Biological Chemistry (2001)
Disruption of CFTR chloride channel alters mechanical properties and cAMP-dependent Cl− transport of mouse aortic smooth muscle cells
Renaud Robert;Caroline Norez;Frédéric Becq.
The Journal of Physiology (2005)
Possible regulation of CFTR‐chloride channels by membrane‐bound phosphatases in pancreatic duct cells
F. Becq;M. Fanjul;M. Merten;C. Figarella.
FEBS Letters (1993)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: