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Biology and Biochemistry

D-Index
85
Citations
27736
World Ranking
3131
National Ranking
239

Overview

Marcus A. Mall is affiliated with Charité - University Medicine Berlin in Germany. Their research primarily spans the field of Medicine with a strong focus on Pulmonary and Respiratory Medicine, Infectious Diseases, Immunology, Epidemiology, and Molecular Biology.

Their work covers several major research topics, including:

  • Cystic Fibrosis Research Advances
  • Neonatal Respiratory Health Research
  • Tracheal and airway disorders
  • Pediatric health and respiratory diseases
  • Respiratory viral infections research
  • SARS-CoV-2 and COVID-19 Research
  • COVID-19 Clinical Research Studies

Marcus A. Mall has contributed extensively to numerous scientific papers, with some recent significant publications including:

  • "SARS-CoV-2-reactive T cells in healthy donors and patients with COVID-19" (2020), published in Nature
  • "Pre-activated antiviral innate immunity in the upper airways controls early SARS-CoV-2 infection in children" (2021), published in Nature Biotechnology
  • "Cross-reactive CD4 + T cells enhance SARS-CoV-2 immune responses upon infection and vaccination" (2021), published in Science
  • "Untimely TGFβ responses in COVID-19 limit antiviral functions of NK cells" (2021), published in Nature
  • "A Phase 3 Open-Label Study of Elexacaftor/Tezacaftor/Ivacaftor in Children 6 through 11 Years of Age with Cystic Fibrosis and at Least One F508del Allele" (2021), published in American Journal of Respiratory and Critical Care Medicine

Their frequent collaborators include:

  • Mirjam Stahl
  • Simon Y. Graeber
  • Olaf Sommerburg
  • Mark O. Wielpütz
  • Julia Duerr

Publications by Marcus A. Mall appear predominantly in the following journals and venues:

  • Journal of Cystic Fibrosis
  • bioRxiv (Cold Spring Harbor Laboratory)
  • European Respiratory Journal
  • American Journal of Respiratory and Critical Care Medicine
  • Klinische Pädiatrie

Best Publications

  • Elexacaftor-Tezacaftor-Ivacaftor for Cystic Fibrosis with a Single Phe508del Allele

    Peter G. Middleton;Marcus A. Mall;Pavel Dřevínek;Larry C. Lands

  • SARS-CoV-2-reactive T cells in healthy donors and patients with COVID-19.

    Julian Braun;Lucie Loyal;Marco Frentsch;Daniel Wendisch

  • Efficacy and safety of the elexacaftor plus tezacaftor plus ivacaftor combination regimen in people with cystic fibrosis homozygous for the F508del mutation : a double-blind, randomised, phase 3 trial

    Harry G M Heijerman;Edward F McKone;Damian G Downey;Eva Van Braeckel

  • Increased airway epithelial Na+ absorption produces cystic fibrosis-like lung disease in mice.

    Marcus Mall;Barbara R Grubb;Jack R Harkema;Wanda K O'Neal

  • Electrolyte Transport in the Mammalian Colon: Mechanisms and Implications for Disease

    Karl Kunzelmann;Marcus Mall

  • VX-445-Tezacaftor-Ivacaftor in Patients with Cystic Fibrosis and One or Two Phe508del Alleles.

    Dominic Keating;Gautham Marigowda;Lucy Burr;Cori Daines

  • The future of cystic fibrosis care: a global perspective

    Scott C Bell;Marcus A Mall;Hector Gutierrez;Milan Macek

  • Pre-activated antiviral innate immunity in the upper airways controls early SARS-CoV-2 infection in children.

    Jennifer Loske;Jobst Röhmel;Soeren Lukassen;Sebastian Stricker

  • VX-659–Tezacaftor–Ivacaftor in Patients with Cystic Fibrosis and One or Two Phe508del Alleles

    Jane C. Davies;Samuel M. Moskowitz;Cynthia Brown;Alexander Horsley

  • Characterization of Wild-Type and ΔF508 Cystic Fibrosis Transmembrane Regulator in Human Respiratory Epithelia

    Silvia M. Kreda;Marcus Mall;April Mengos;Lori Rochelle

  • Cross-reactive CD4 + T cells enhance SARS-CoV-2 immune responses upon infection and vaccination.

    Lucie Loyal;Lucie Loyal;Julian Braun;Julian Braun;Larissa Henze;Larissa Henze;Beate Kruse;Beate Kruse

  • Innate immunity in cystic fibrosis lung disease

    D. Hartl;A. Gaggar;E. Bruscia;A. Hector

  • CFTR: cystic fibrosis and beyond

    Marcus A. Mall;Dominik Hartl

  • Magnetic resonance imaging detects changes in structure and perfusion, and response to therapy in early cystic fibrosis lung disease.

    Mark O. Wielputz;Michael Puderbach;Annette Kopp-Schneider;Mirjam Stahl

  • Role of Cilia, Mucus, and Airway Surface Liquid in Mucociliary Dysfunction: Lessons from Mouse Models

    Marcus A. Mall

  • CXCR2 mediates NADPH oxidase–independent neutrophil extracellular trap formation in cystic fibrosis airway inflammation

    Veronica Marcos;Zhe Zhou;Ali Önder Yildirim;Alexander Bohla

  • The amiloride-inhibitable Na+ conductance is reduced by the cystic fibrosis transmembrane conductance regulator in normal but not in cystic fibrosis airways.

    M Mall;M Bleich;R Greger;R Schreiber

  • Development of Chronic Bronchitis and Emphysema in β-Epithelial Na+ Channel–Overexpressing Mice

    Marcus A. Mall;Jack R. Harkema;Joanna B. Trojanek;Diana Treis

  • Morphologic and functional scoring of cystic fibrosis lung disease using MRI

    Monika Eichinger;Daiva Elzbieta Optazaite;Annette Kopp-Schneider;Christian Hintze

  • Comparison of Lung Clearance Index and Magnetic Resonance Imaging for Assessment of Lung Disease in Children with Cystic Fibrosis

    Mirjam Stahl;Mark O Wielpütz;Simon Y Graeber;Cornelia Joachim

Frequent Co-Authors

Carsten Schultz
Carsten Schultz Oregon Health & Science University
Karl Kunzelmann
Karl Kunzelmann University of Regensburg
Richard C. Boucher
Richard C. Boucher University of North Carolina at Chapel Hill
Martina U. Muckenthaler
Martina U. Muckenthaler Heidelberg University
Hans-Ulrich Kauczor
Hans-Ulrich Kauczor Heidelberg University
Dominik Hartl
Dominik Hartl Novartis (Switzerland)
Catherine M. Greene
Catherine M. Greene Royal College of Surgeons in Ireland
Burkhard Tümmler
Burkhard Tümmler Hannover Medical School
Steven M. Rowe
Steven M. Rowe University of Alabama at Birmingham
Alexander H. Dalpke
Alexander H. Dalpke Heidelberg University

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