2023 - Research.com Biology and Biochemistry in Germany Leader Award
2000 - Member of Academia Europaea
His primary areas of study are Cell biology, Molecular biology, Internal medicine, Endocrinology and Immunology. Josef Pfeilschifter interconnects Regulation of gene expression, Biochemistry and Nitric oxide synthase, Nitric oxide in the investigation of issues within Cell biology. His research integrates issues of Mesangial cell and Keratinocyte in his study of Nitric oxide.
His Molecular biology research incorporates elements of Cell culture, Protein kinase A, Small interfering RNA, Promoter and Superoxide. He specializes in Internal medicine, namely Kidney. The study incorporates disciplines such as Granulation tissue and Receptor tyrosine kinase in addition to Endocrinology.
His scientific interests lie mostly in Cell biology, Molecular biology, Internal medicine, Endocrinology and Immunology. His Cell biology study combines topics from a wide range of disciplines, such as Sphingosine, Apoptosis, Biochemistry and Nitric oxide. His Nitric oxide research is multidisciplinary, incorporating elements of Superoxide, Keratinocyte and Glomerular Mesangial Cell.
Josef Pfeilschifter has included themes like Cell culture, Nitric oxide synthase, Gene expression, Protein kinase A and Protein kinase C in his Molecular biology study. His research in Endocrinology intersects with topics in Receptor and Downregulation and upregulation. Immunology and Cancer research are commonly linked in his work.
His primary scientific interests are in Cancer research, Cell biology, Sphingosine, Sphingolipid and Internal medicine. His studies in Cancer research integrate themes in fields like Apoptosis, Gene knockdown, Downregulation and upregulation, Immunology and Colorectal cancer. His Cell biology study incorporates themes from Inflammation, Endothelial stem cell and RNA-binding protein.
His Inflammation research integrates issues from Tumor necrosis factor alpha, Interferon, Interleukin 18 and Interleukin. His biological study deals with issues like Nitric oxide, which deal with fields such as Cytokine. The study incorporates disciplines such as Gastroenterology and Endocrinology in addition to Internal medicine.
Josef Pfeilschifter mainly focuses on Cell biology, Sphingolipid, Cancer research, Downregulation and upregulation and Inflammation. The Cell biology study combines topics in areas such as Sphingosine and Sphingosine kinase 1. As part of the same scientific family, Josef Pfeilschifter usually focuses on Sphingolipid, concentrating on Ceramide and intersecting with Internal medicine, Sphingosine-1-phosphate, Insulin, Alpha-fetoprotein and Gastroenterology.
His study explores the link between Cancer research and topics such as Gene knockdown that cross with problems in Gene silencing, Molecular biology and DNA repair. His Inflammation study improves the overall literature in Immunology. His Biglycan research focuses on subjects like Kinase, which are linked to Nitric oxide.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Cyclooxygenase-independent actions of cyclooxygenase inhibitors
Irmgard Tegeder;Josef Pfeilschifter;Gerd Geisslinger.
The FASEB Journal (2001)
Large and sustained induction of chemokines during impaired wound healing in the genetically diabetic mouse: prolonged persistence of neutrophils and macrophages during the late phase of repair.
Christian Wetzler;Heiko Kämpfer;Birgit Stallmeyer;Josef Pfeilschifter.
Journal of Investigative Dermatology (2000)
Physiology and pathophysiology of sphingolipid metabolism and signaling.
Andrea Huwiler;Thomas Kolter;Josef Pfeilschifter;Konrad Sandhoff.
Biochimica et Biophysica Acta (2000)
Biglycan: A danger signal that activates the NLRP3 inflammasome via toll-like and P2X receptors
Andrea Babelova;Kristin Moreth;Kristin Moreth;Wasiliki Tsalastra-Greul;Jinyang Zeng-Brouwers;Jinyang Zeng-Brouwers.
Journal of Biological Chemistry (2009)
A transgenic mouse model of inducible macrophage depletion: effects of diphtheria toxin-driven lysozyme M-specific cell lineage ablation on wound inflammatory, angiogenic, and contractive processes.
Itamar Goren;Nadine Allmann;Nir Yogev;Christoph Schürmann.
American Journal of Pathology (2009)
IL-1 family nomenclature
Charles Dinarello;William Arend;John Sims;Dirk Smith.
Nature Immunology (2010)
Nitric oxide triggers enhanced induction of vascular endothelial growth factor expression in cultured keratinocytes (HaCaT) and during cutaneous wound repair
Stefan Frank;Birgit Stallmeyer;Heiko Kämpfer;Nicole Kolb.
The FASEB Journal (1999)
The Function of Nitric Oxide in Wound Repair: Inhibition of Inducible Nitric Oxide-Synthase Severely Impairs Wound Reepithelialization
Birgit Stallmeyer;Heiko Kämpfer;Nicole Kolb;Josef Pfeilschifter.
Journal of Investigative Dermatology (1999)
Amplification of IL-1β-Induced Matrix Metalloproteinase-9 Expression by Superoxide in Rat Glomerular Mesangial Cells Is Mediated by Increased Activities of NF-κB and Activating Protein-1 and Involves Activation of the Mitogen-Activated Protein Kinase Pathways
Wolfgang Eberhardt;Andrea Huwiler;Karl-Friedrich Beck;Sebastian Walpen.
Journal of Immunology (2000)
Anti-inflammatory properties of pro-inflammatory interferon-gamma.
Heiko Mühl;Josef Pfeilschifter.
International Immunopharmacology (2003)
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