2000 - Member of the National Academy of Sciences
1999 - Fellow of the American Association for the Advancement of Science (AAAS)
1998 - Fellow of the American Academy of Arts and Sciences
Cystic fibrosis transmembrane conductance regulator, Cell biology, Cystic fibrosis, Internal medicine and Endocrinology are his primary areas of study. His Cystic fibrosis transmembrane conductance regulator research includes themes of Chloride channel, Mutant, Molecular biology and Phosphorylation. His Cell biology study incorporates themes from Epithelial polarity, Amiloride, Capsid, Ion channel and Adenoviridae.
The Cystic fibrosis study combines topics in areas such as Mutation, Allele and Immunology, Pathogenesis. His Immunology study integrates concerns from other disciplines, such as Lung, Lung disease, In vivo and Bacteria. His Endocrinology research is multidisciplinary, relying on both Respiratory epithelium, Transfection and Transepithelial potential difference.
His main research concerns Cystic fibrosis transmembrane conductance regulator, Cystic fibrosis, Cell biology, Biochemistry and Internal medicine. Michael J. Welsh has included themes like Mutation, Transmembrane protein, Chloride channel, Molecular biology and Phosphorylation in his Cystic fibrosis transmembrane conductance regulator study. His Cystic fibrosis study also includes
His work carried out in the field of Cell biology brings together such families of science as Apical membrane, Epithelium, Secretion and Mutant. His Biochemistry research integrates issues from Biophysics and Gating. Michael J. Welsh works mostly in the field of Internal medicine, limiting it down to concerns involving Endocrinology and, occasionally, Respiratory epithelium.
Michael J. Welsh mostly deals with Cystic fibrosis, Cystic fibrosis transmembrane conductance regulator, Airway, Cell biology and Internal medicine. His Cystic fibrosis research includes elements of Lung and Immunology, Pathogenesis, Pathology. Michael J. Welsh combines subjects such as Mutation, Secretion, Bicarbonate and Ion transporter with his study of Cystic fibrosis transmembrane conductance regulator.
His study looks at the relationship between Airway and fields such as Respiratory system, as well as how they intersect with chemical problems. His Cell biology research is multidisciplinary, incorporating elements of Gene expression, Human airway, Ion channel and Transmembrane protein. His research in Internal medicine intersects with topics in Gastroenterology and Diabetes mellitus, Endocrinology.
His scientific interests lie mostly in Cystic fibrosis, Cystic fibrosis transmembrane conductance regulator, Lung, Immunology and Pathology. His Cystic fibrosis study results in a more complete grasp of Internal medicine. His studies in Cystic fibrosis transmembrane conductance regulator integrate themes in fields like Cell biology, Airway, Microbiology and Genetic enhancement.
His research integrates issues of Fibrosis, Respiratory epithelium, Ion channel and Transmembrane protein in his study of Cell biology. The study incorporates disciplines such as Mutation, Olfactory receptor and In vivo in addition to Lung. Michael J. Welsh has researched Immunology in several fields, including Neuropeptide, Calcitonin gene-related peptide, Cell culture and Calcitonin.
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Molecular mechanisms of CFTR chloride channel dysfunction in cystic fibrosis
Michael J. Welsh;Alan E. Smith.
Quorum-sensing signals indicate that cystic fibrosis lungs are infected with bacterial biofilms
Pradeep K. Singh;Amy L. Schaefer;Matthew R. Parsek;Thomas O. Moninger.
Cellular and Molecular Barriers to Gene Transfer by a Cationic Lipid
Joseph Zabner;Al J. Fasbender;Tom Moninger;Kristi A. Poellinger.
Journal of Biological Chemistry (1995)
Cystic Fibrosis Airway Epithelia Fail to Kill Bacteria Because of Abnormal Airway Surface Fluid
Jeffrey J Smith;Sue M Travis;E.Peter Greenberg;Michael J Welsh;Michael J Welsh.
Processing of mutant cystic fibrosis transmembrane conductance regulator is temperature-sensitive.
Gerene M. Denning;Matthew P. Anderson;Jane F. Amara;John Marshall.
Structure and Function of the CFTR Chloride Channel
David N. Sheppard;Michael J. Welsh.
Physiological Reviews (1999)
Demonstration that CFTR is a Chloride Channel by Alteration of its Anion Selectivity
Matthew P. Anderson;Richard J. Gregory;Simon Thompson;David W. Souza.
A component of innate immunity prevents bacterial biofilm development
Pradeep K. Singh;Matthew R. Parsek;E. Peter Greenberg;E. Peter Greenberg;Michael J. Welsh;Michael J. Welsh;Michael J. Welsh.
Neuroprotection in ischemia: blocking calcium-permeable acid-sensing ion channels.
Zhi-Gang Xiong;Xiao-Man Zhu;Xiang-Ping Chu;Manabu Minami.
Adenovirus-mediated gene transfer transiently corrects the chloride transport defect in nasal epithelia of patients with cystic fibrosis
Joseph Zabner;Larry A. Couture;Richard J. Gregory;Scott M. Graham.
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