D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 130 Citations 66,867 374 World Ranking 173 National Ranking 127

Research.com Recognitions

Awards & Achievements

2000 - Member of the National Academy of Sciences

1999 - Fellow of the American Association for the Advancement of Science (AAAS)

1998 - Fellow of the American Academy of Arts and Sciences

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Internal medicine

Cystic fibrosis transmembrane conductance regulator, Cell biology, Cystic fibrosis, Internal medicine and Endocrinology are his primary areas of study. His Cystic fibrosis transmembrane conductance regulator research includes themes of Chloride channel, Mutant, Molecular biology and Phosphorylation. His Cell biology study incorporates themes from Epithelial polarity, Amiloride, Capsid, Ion channel and Adenoviridae.

The Cystic fibrosis study combines topics in areas such as Mutation, Allele and Immunology, Pathogenesis. His Immunology study integrates concerns from other disciplines, such as Lung, Lung disease, In vivo and Bacteria. His Endocrinology research is multidisciplinary, relying on both Respiratory epithelium, Transfection and Transepithelial potential difference.

His most cited work include:

  • Cellular and Molecular Barriers to Gene Transfer by a Cationic Lipid (1265 citations)
  • Quorum-sensing signals indicate that cystic fibrosis lungs are infected with bacterial biofilms (1261 citations)
  • Molecular mechanisms of CFTR chloride channel dysfunction in cystic fibrosis (1193 citations)

What are the main themes of his work throughout his whole career to date?

His main research concerns Cystic fibrosis transmembrane conductance regulator, Cystic fibrosis, Cell biology, Biochemistry and Internal medicine. Michael J. Welsh has included themes like Mutation, Transmembrane protein, Chloride channel, Molecular biology and Phosphorylation in his Cystic fibrosis transmembrane conductance regulator study. His Cystic fibrosis study also includes

  • Pathology and related Pancreatic disease,
  • Immunology which is related to area like Bacteria.

His work carried out in the field of Cell biology brings together such families of science as Apical membrane, Epithelium, Secretion and Mutant. His Biochemistry research integrates issues from Biophysics and Gating. Michael J. Welsh works mostly in the field of Internal medicine, limiting it down to concerns involving Endocrinology and, occasionally, Respiratory epithelium.

He most often published in these fields:

  • Cystic fibrosis transmembrane conductance regulator (32.44%)
  • Cystic fibrosis (30.60%)
  • Cell biology (32.65%)

What were the highlights of his more recent work (between 2013-2021)?

  • Cystic fibrosis (30.60%)
  • Cystic fibrosis transmembrane conductance regulator (32.44%)
  • Airway (9.45%)

In recent papers he was focusing on the following fields of study:

Michael J. Welsh mostly deals with Cystic fibrosis, Cystic fibrosis transmembrane conductance regulator, Airway, Cell biology and Internal medicine. His Cystic fibrosis research includes elements of Lung and Immunology, Pathogenesis, Pathology. Michael J. Welsh combines subjects such as Mutation, Secretion, Bicarbonate and Ion transporter with his study of Cystic fibrosis transmembrane conductance regulator.

His study looks at the relationship between Airway and fields such as Respiratory system, as well as how they intersect with chemical problems. His Cell biology research is multidisciplinary, incorporating elements of Gene expression, Human airway, Ion channel and Transmembrane protein. His research in Internal medicine intersects with topics in Gastroenterology and Diabetes mellitus, Endocrinology.

Between 2013 and 2021, his most popular works were:

  • Origins of Cystic Fibrosis Lung Disease (369 citations)
  • Impaired mucus detachment disrupts mucociliary transport in a piglet model of cystic fibrosis (239 citations)
  • Airway acidification initiates host defense abnormalities in cystic fibrosis mice (178 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Internal medicine

His scientific interests lie mostly in Cystic fibrosis, Cystic fibrosis transmembrane conductance regulator, Lung, Immunology and Pathology. His Cystic fibrosis study results in a more complete grasp of Internal medicine. His studies in Cystic fibrosis transmembrane conductance regulator integrate themes in fields like Cell biology, Airway, Microbiology and Genetic enhancement.

His research integrates issues of Fibrosis, Respiratory epithelium, Ion channel and Transmembrane protein in his study of Cell biology. The study incorporates disciplines such as Mutation, Olfactory receptor and In vivo in addition to Lung. Michael J. Welsh has researched Immunology in several fields, including Neuropeptide, Calcitonin gene-related peptide, Cell culture and Calcitonin.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Molecular mechanisms of CFTR chloride channel dysfunction in cystic fibrosis

Michael J. Welsh;Alan E. Smith.
Cell (1993)

1892 Citations

Quorum-sensing signals indicate that cystic fibrosis lungs are infected with bacterial biofilms

Pradeep K. Singh;Amy L. Schaefer;Matthew R. Parsek;Thomas O. Moninger.
Nature (2000)

1795 Citations

Cellular and Molecular Barriers to Gene Transfer by a Cationic Lipid

Joseph Zabner;Al J. Fasbender;Tom Moninger;Kristi A. Poellinger.
Journal of Biological Chemistry (1995)

1745 Citations

Cystic Fibrosis Airway Epithelia Fail to Kill Bacteria Because of Abnormal Airway Surface Fluid

Jeffrey J Smith;Sue M Travis;E.Peter Greenberg;Michael J Welsh;Michael J Welsh.
Cell (1996)

1462 Citations

Processing of mutant cystic fibrosis transmembrane conductance regulator is temperature-sensitive.

Gerene M. Denning;Matthew P. Anderson;Jane F. Amara;John Marshall.
Nature (1992)

1409 Citations

Structure and Function of the CFTR Chloride Channel

David N. Sheppard;Michael J. Welsh.
Physiological Reviews (1999)

1273 Citations

Demonstration that CFTR is a Chloride Channel by Alteration of its Anion Selectivity

Matthew P. Anderson;Richard J. Gregory;Simon Thompson;David W. Souza.
Science (1991)

1269 Citations

A component of innate immunity prevents bacterial biofilm development

Pradeep K. Singh;Matthew R. Parsek;E. Peter Greenberg;E. Peter Greenberg;Michael J. Welsh;Michael J. Welsh;Michael J. Welsh.
Nature (2002)

1216 Citations

Neuroprotection in ischemia: blocking calcium-permeable acid-sensing ion channels.

Zhi-Gang Xiong;Xiao-Man Zhu;Xiang-Ping Chu;Manabu Minami.
Cell (2004)

1057 Citations

Adenovirus-mediated gene transfer transiently corrects the chloride transport defect in nasal epithelia of patients with cystic fibrosis

Joseph Zabner;Larry A. Couture;Richard J. Gregory;Scott M. Graham.
Cell (1993)

788 Citations

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