Alan E. Smith spends much of his time researching Cystic fibrosis transmembrane conductance regulator, Cystic fibrosis, Molecular biology, Chloride channel and Internal medicine. His research integrates issues of Mutation, Mutant, Membrane protein and Cell biology in his study of Cystic fibrosis transmembrane conductance regulator. Alan E. Smith works mostly in the field of Cell biology, limiting it down to topics relating to ΔF508 and, in certain cases, Mutant protein and Intracellular.
The concepts of his Cystic fibrosis study are interwoven with issues in Immunology, Transfection, Pathology, Complementary DNA and Lung. His Molecular biology research incorporates elements of Adenoviridae, Genetic transfer, Genetic enhancement, Capsid and Peptide sequence. His work is dedicated to discovering how Internal medicine, Endocrinology are connected with Transepithelial potential difference and other disciplines.
Molecular biology, Cystic fibrosis transmembrane conductance regulator, Cystic fibrosis, Biochemistry and Cell biology are his primary areas of study. His Molecular biology research incorporates themes from Mutation, Mutant, DNA, Antigen and Virus. His Cystic fibrosis transmembrane conductance regulator study combines topics from a wide range of disciplines, such as Chloride channel and Membrane protein.
His studies in Chloride channel integrate themes in fields like Apical membrane and Chinese hamster ovary cell. His Cystic fibrosis research includes themes of Immunology, Pathology, Complementary DNA, Genetic enhancement and Lung. He does research in Cell biology, focusing on Intracellular specifically.
His primary scientific interests are in Cystic fibrosis, Cystic fibrosis transmembrane conductance regulator, Flora, Moss and Viral vector. His research in Cystic fibrosis intersects with topics in Lung, Gene and Pathology. His Cystic fibrosis transmembrane conductance regulator research integrates issues from Molecular biology, Transfection and Binding site.
Alan E. Smith has included themes like Cell culture, Mutant, Complementary DNA, Chloride channel and In vivo in his Molecular biology study. His study in Viral vector is interdisciplinary in nature, drawing from both Lung disease, Genetic enhancement, Transgene and Cell biology. While the research belongs to areas of Internal medicine, Alan E. Smith spends his time largely on the problem of Endocrinology, intersecting his research to questions surrounding Regulator, Chloride channel activity and Biological activity.
His primary areas of investigation include Genetic enhancement, Molecular biology, Cystic fibrosis, Vector and Cystic fibrosis transmembrane conductance regulator. His Genetic enhancement research is multidisciplinary, incorporating elements of Immunology and Virology. His Molecular biology study integrates concerns from other disciplines, such as Transgene and In vivo.
Alan E. Smith has researched Cystic fibrosis in several fields, including Gene, Lung and Pathology. His Vector study deals with Gene transfer intersecting with Open reading frame, Chromatography, Disease and Lysosomal storage disorders. His Cystic fibrosis transmembrane conductance regulator study is associated with Internal medicine.
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A short amino acid sequence able to specify nuclear location
Daniel Kalderon;Bruce L. Roberts;William D. Richardson;Alan E. Smith.
Defective intracellular transport and processing of CFTR is the molecular basis of most cystic fibrosis
Seng H. Cheng;Richard J. Gregory;John Marshall;Sucharita Paul.
Molecular mechanisms of CFTR chloride channel dysfunction in cystic fibrosis
Michael J. Welsh;Alan E. Smith.
Sequence requirements for nuclear location of simian virus 40 large-T antigen.
Daniel Kalderon;William D. Richardson;Alexander F. Markham;Alan E. Smith.
Processing of mutant cystic fibrosis transmembrane conductance regulator is temperature-sensitive.
Gerene M. Denning;Matthew P. Anderson;Jane F. Amara;John Marshall.
Demonstration That CFTR Is a Chloride Channel by Alteration of Its Anion Selectivity
Matthew P. Anderson;Richard J. Gregory;Simon Thompson;David W. Souza.
Adenovirus-mediated gene transfer transiently corrects the chloride transport defect in nasal epithelia of patients with cystic fibrosis
Joseph Zabner;Larry A. Couture;Richard J. Gregory;Scott M. Graham.
Expression of cystic fibrosis transmembrane conductance regulator corrects defective chloride channel regulation in cystic fibrosis airway epithelial cells.
Devra P. Rich;Matthew P. Anderson;Richard J. Gregory;Seng H. Cheng.
Generation of cAMP-activated chloride currents by expression of CFTR
Matthew P. Anderson;Devra P. Rich;Richard J. Gregory;Alan E. Smith.
Phosphorylation of the R domain by cAMP-dependent protein kinase regulates the CFTR chloride channel.
S.H. Cheng;D.P. Rich;J. Marshall;R.J. Gregory.
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