David A. Stoltz spends much of his time researching Cystic fibrosis, Cystic fibrosis transmembrane conductance regulator, Immunology, Lung and Respiratory system. His Cystic fibrosis study incorporates themes from Inflammation, Endocrinology, Pancreatic disease and Pathogenesis. His Cystic fibrosis transmembrane conductance regulator research incorporates elements of Mutation, Molecular biology and Pathology.
His Pathology study combines topics in areas such as Exocrine gland and Mucociliary clearance. His Immunology research is multidisciplinary, incorporating perspectives in Andrology, Airway and In vivo. His study in Respiratory system is interdisciplinary in nature, drawing from both Secretion, Mucin and Polysaccharide.
His primary scientific interests are in Cystic fibrosis, Cystic fibrosis transmembrane conductance regulator, Pathology, Lung and Airway. His studies deal with areas such as Inflammation, Immunology, Pathogenesis and Molecular biology as well as Cystic fibrosis. His Immunology research is multidisciplinary, relying on both Microbiology and Bacteria.
Cystic fibrosis transmembrane conductance regulator is often connected to Mutation in his work. His Lung study also includes
His primary areas of investigation include Cystic fibrosis, Airway, Lung, Submucosal glands and Respiratory system. His Cystic fibrosis study is concerned with Internal medicine in general. In his research, Cell type is intimately related to Cell biology, which falls under the overarching field of Airway.
His biological study spans a wide range of topics, including Contraction, Muscle contraction, Anatomy, Microscopy and Cystic fibrosis transmembrane conductance regulator. David A. Stoltz interconnects Cell, In vitro and Gene expression in the investigation of issues within Cystic fibrosis transmembrane conductance regulator. His Submucosal glands research includes themes of Ex vivo, In vivo and Host.
David A. Stoltz mainly focuses on Cystic fibrosis, Respiratory system, Submucosal glands, In vivo and Ex vivo. He conducts interdisciplinary study in the fields of Cystic fibrosis and Persistence through his research. His research integrates issues of Secretion, Gland secretion, Biophysics, Methacholine and Lung in his study of Respiratory system.
Submucosal glands is a subfield of Airway that he studies.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Disruption of the CFTR gene produces a model of cystic fibrosis in newborn pigs.
Christopher S. Rogers;David A. Stoltz;David K. Meyerholz;Lynda S. Ostedgaard.
Science (2008)
Reduced airway surface pH impairs bacterial killing in the porcine cystic fibrosis lung
Alejandro A. Pezzulo;Xiao Xiao Tang;Mark J. Hoegger;Mahmoud H. Abou Alaiwa.
Nature (2012)
Origins of Cystic Fibrosis Lung Disease
David A. Stoltz;David K. Meyerholz;Michael J. Welsh.
The New England Journal of Medicine (2015)
Cystic Fibrosis Pigs Develop Lung Disease and Exhibit Defective Bacterial Eradication at Birth
David A. Stoltz;David K. Meyerholz;Alejandro A. Pezzulo;Shyam Ramachandran.
Science Translational Medicine (2010)
Production of CFTR-null and CFTR-ΔF508 heterozygous pigs by adeno-associated virus–mediated gene targeting and somatic cell nuclear transfer
Christopher S. Rogers;Yanhong Hao;Tatiana Rokhlina;Melissa Samuel.
Journal of Clinical Investigation (2008)
IL-17 Stimulates Granulopoiesis in Mice: Use of an Alternate, Novel Gene Therapy-Derived Method for In Vivo Evaluation of Cytokines
P. Schwarzenberger;V. La Russa;A. Miller;Peng Ye.
Journal of Immunology (1998)
Impaired mucus detachment disrupts mucociliary transport in a piglet model of cystic fibrosis
Mark J. Hoegger;Anthony J. Fischer;James D. McMenimen;Lynda S. Ostedgaard.
Science (2014)
The porcine lung as a potential model for cystic fibrosis.
Christopher S. Rogers;William M. Abraham;Kim A. Brogden;John F. Engelhardt.
American Journal of Physiology-lung Cellular and Molecular Physiology (2008)
Loss of anion transport without increased sodium absorption characterizes newborn porcine cystic fibrosis airway epithelia.
Jeng-Haur Chen;David A. Stoltz;Philip H. Karp;Sarah E. Ernst.
Cell (2010)
Airway acidification initiates host defense abnormalities in cystic fibrosis mice
Viral S. Shah;David K. Meyerholz;Xiao Xiao Tang;Leah Reznikov.
Science (2016)
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