David K. Meyerholz mainly focuses on Immunology, Virus, Lung, Cystic fibrosis and Virology. The study incorporates disciplines such as Disease and Respiratory system in addition to Immunology. The various areas that he examines in his Virus study include Middle East respiratory syndrome coronavirus, Interferon, Middle East respiratory syndrome, Proinflammatory cytokine and Coronavirus.
The concepts of his Lung study are interwoven with issues in Inflammation, Acquired immune system and FOXP3. His work carried out in the field of Cystic fibrosis brings together such families of science as Pancreatic disease and Pathogenesis, Pathology. His Virology study combines topics from a wide range of disciplines, such as Epitope, Antigen, Immunity, Hepatitis A Virus Cellular Receptor 1 and Genetically modified mouse.
Immunology, Pathology, Lung, Cystic fibrosis and Internal medicine are his primary areas of study. His Pathology research includes themes of Cancer research and Anatomy. His Lung study frequently links to other fields, such as Respiratory system.
His work is dedicated to discovering how Cystic fibrosis, Airway are connected with Cell biology and other disciplines. In Internal medicine, David K. Meyerholz works on issues like Endocrinology, which are connected to Proinflammatory cytokine. His Virus research incorporates themes from Respiratory disease and Coronavirus.
David K. Meyerholz mostly deals with Immunology, Lung, Disease, Coronavirus and Pathology. His study in Immunology is interdisciplinary in nature, drawing from both Receptor and Remyelination. His biological study spans a wide range of topics, including Cell, Cell type, Airway, Submucosal glands and Cell biology.
His Disease research integrates issues from Immunohistochemistry, Hospitalized patients, Pneumonia, Common cold and Intensive care unit. David K. Meyerholz has included themes like Middle East respiratory syndrome coronavirus, Virus, Virology, Colony stimulating factor 1 receptor and Myelin in his Coronavirus study. His research integrates issues of Mouse Lung, Gallbladder and Organoid in his study of Pathology.
His scientific interests lie mostly in Immunology, Disease, Lung, Pathogenesis and Coronavirus. His work is connected to Cytokine and Tumor necrosis factor alpha, as a part of Immunology. His studies deal with areas such as Parenchyma and Pneumonia as well as Disease.
His Lung research is multidisciplinary, incorporating perspectives in Immunohistochemistry, Respiratory tract, Receptor, Receptor expression and Messenger RNA. His Pathogenesis study integrates concerns from other disciplines, such as Pathological, Vasculitis, Asymptomatic, Anosmia and Programmed cell death. His Coronavirus research is multidisciplinary, incorporating elements of Middle East respiratory syndrome coronavirus, Virology, Chemokine, CXCL10 and Macrophage.
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Dysregulated Type I Interferon and Inflammatory Monocyte-Macrophage Responses Cause Lethal Pneumonia in SARS-CoV-Infected Mice
Rudragouda Channappanavar;Anthony R. Fehr;Rahul Vijay;Matthias Mack.
Cell Host & Microbe (2016)
Severe acute respiratory syndrome coronavirus infection causes neuronal death in the absence of encephalitis in mice transgenic for human ACE2.
Jason Netland;David K. Meyerholz;Steven Moore;Martin Cassell.
Journal of Virology (2008)
Disruption of the CFTR gene produces a model of cystic fibrosis in newborn pigs.
Christopher S. Rogers;David A. Stoltz;David K. Meyerholz;Lynda S. Ostedgaard.
Systemic administration of optimized aptamer-siRNA chimeras promotes regression of PSMA-expressing tumors
Justin P Dassie;Xiu-ying Liu;Gregory S Thomas;Ryan M Whitaker.
Nature Biotechnology (2009)
Sex-Based Differences in Susceptibility to Severe Acute Respiratory Syndrome Coronavirus Infection.
Rudragouda Channappanavar;Craig Fett;Matthias Mack;Patrick P. Ten Eyck.
Journal of Immunology (2017)
Origins of Cystic Fibrosis Lung Disease
David A. Stoltz;David K. Meyerholz;Michael J. Welsh.
The New England Journal of Medicine (2015)
Cystic Fibrosis Pigs Develop Lung Disease and Exhibit Defective Bacterial Eradication at Birth
David A. Stoltz;David K. Meyerholz;Alejandro A. Pezzulo;Shyam Ramachandran.
Science Translational Medicine (2010)
Lethal Infection of K18-hACE2 Mice Infected with Severe Acute Respiratory Syndrome Coronavirus
Paul B. McCray;Lecia Pewe;Christine Wohlford-Lenane;Melissa Hickey.
Journal of Virology (2007)
T-cell immunoglobulin and mucin domain 1 (TIM-1) is a receptor for Zaire Ebolavirus and Lake Victoria Marburgvirus
Andrew S Kondratowicz;Nicholas J Lennemann;Patrick L Sinn;Robert A Davey.
Proceedings of the National Academy of Sciences of the United States of America (2011)
Disease phenotype of a ferret CFTR-knockout model of cystic fibrosis
Xingshen Sun;Hongshu Sui;John T. Fisher;Ziying Yan.
Journal of Clinical Investigation (2010)
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