The scientist’s investigation covers issues in Neuroscience, Cerebellum, Purkinje cell, Cell biology and Neurodegeneration. His Neuroscience research includes themes of Dysbindin and Anatomy. His Anatomy study incorporates themes from Sonic hedgehog and Sensory system.
His study on Mossy fiber is often connected to Relation as part of broader study in Cerebellum. His Purkinje cell research is multidisciplinary, incorporating elements of Homeobox, Molecular biology, Cerebellar cortex and Calbindin. His Cell biology study integrates concerns from other disciplines, such as Cerebral cortex, Genetics, Ataxia and Mutant protein.
Roy V. Sillitoe spends much of his time researching Neuroscience, Cerebellum, Purkinje cell, Cell biology and Cerebellar cortex. His Neuroscience and Cognition, Dystonia, Inhibitory postsynaptic potential, Excitatory postsynaptic potential and Cytoarchitecture investigations all form part of his Neuroscience research activities. His Mossy fiber study in the realm of Cerebellum interacts with subjects such as Cell type.
He combines subjects such as Premovement neuronal activity and Pathology with his study of Purkinje cell. The concepts of his Cell biology study are interwoven with issues in Genetics, Biological neural network, Mutant protein and Histone. His research in the fields of Fourth ventricle overlaps with other disciplines such as Central Zone.
His primary areas of study are Neuroscience, Cerebellum, Purkinje cell, Dystonia and Ataxia. Roy V. Sillitoe performs multidisciplinary study in the fields of Neuroscience and Chemistry via his papers. Roy V. Sillitoe interconnects Electrophysiology, Cognition, Motor learning, Eupnea and Cerebellar ataxia in the investigation of issues within Cerebellum.
His Purkinje cell research includes elements of Granule cell, Premovement neuronal activity, Cerebellar cortex, In vivo and Cerebellar disorder. His work in the fields of Dystonic disorder overlaps with other areas such as Schizophrenia, Context, Dyslexia and Psychological intervention. His Ataxia study combines topics from a wide range of disciplines, such as Neuromodulation, Developmental biology and Morphogenesis.
Roy V. Sillitoe mainly focuses on Cerebellum, Neuroscience, Purkinje cell, Dystonia and Ataxia. In his papers, Roy V. Sillitoe integrates diverse fields, such as Cerebellum and Action tremor. His study in Cognition, Neurology, Cerebellar Purkinje cell, Optogenetics and Neural substrate is carried out as part of his studies in Neuroscience.
His study in the field of Aldolase C also crosses realms of Central Zone. In the field of Dystonia, his study on Dystonic disorder overlaps with subjects such as Extramural, Psychological intervention and Key features. His biological study spans a wide range of topics, including Hippocampal formation, Prefrontal cortex and Local field potential.
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Morphology, molecular codes, and circuitry produce the three-dimensional complexity of the cerebellum.
Roy V Sillitoe;Alexandra L Joyner.
Annual Review of Cell and Developmental Biology (2007)
Consensus Paper: Cerebellar Development.
Ketty Leto;Marife Arancillo;Esther B. E. Becker;Annalisa Buffo.
The Cerebellum (2016)
Redefining the cerebellar cortex as an assembly of non-uniform Purkinje cell microcircuits
Nadia L. Cerminara;Eric J. Lang;Roy V. Sillitoe;Richard Apps.
Nature Reviews Neuroscience (2015)
Patterned Purkinje cell degeneration in mouse models of Niemann-Pick type C disease.
Justyna R Sarna;Matt Larouche;Hassan Marzban;Roy V Sillitoe.
The Journal of Comparative Neurology (2003)
Whole-mount immunohistochemistry: a high-throughput screen for patterning defects in the mouse cerebellum.
Roy V. Sillitoe;Richard Hawkes.
Journal of Histochemistry and Cytochemistry (2002)
New roles for the cerebellum in health and disease
Stacey L Reeber;Tom S. Otis;Roy Vincent Sillitoe.
Frontiers in Systems Neuroscience (2013)
Engrailed Homeobox Genes Regulate Establishment of the Cerebellar Afferent Circuit Map
Roy V. Sillitoe;Michael W. Vogel;Alexandra L. Joyner.
The Journal of Neuroscience (2010)
Pumilio1 Haploinsufficiency Leads to SCA1-like Neurodegeneration by Increasing Wild-Type Ataxin1 Levels
Vincenzo A. Gennarino;Vincenzo A. Gennarino;Ravi K. Singh;Joshua J. White;Joshua J. White;Antonia De Maio;Antonia De Maio.
SGCE missense mutations that cause myoclonus-dystonia syndrome impair ε-sarcoglycan trafficking to the plasma membrane: modulation by ubiquitination and torsinA
Christopher T. Esapa;Adrian James Waite;Matthew Locke;Matthew A. Benson.
Human Molecular Genetics (2007)
Emerging connections between cerebellar development, behaviour and complex brain disorders.
Aaron Sathyanesan;Joy Zhou;Joseph Scafidi;Joseph Scafidi;Detlef H. Heck.
Nature Reviews Neuroscience (2019)
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