World's Best Scientists 2026 revealed!

D-Index & Metrics

Genetics

D-Index
104
Citations
40047
World Ranking
645
National Ranking
327

Medicine

D-Index
104
Citations
40214
World Ranking
7048
National Ranking
3707

Neuroscience

D-Index
102
Citations
39059
World Ranking
709
National Ranking
398

Harry T. Orr publication distribution in Genetics in 2026

The chart shows the distribution of publications by all Research.com ranked scientists in the field of Genetics in 2026. The highlighted bar marks where Harry T. Orr sits on this spectrum.

45–54 publications: 6 scientists 55–64 publications: 10 scientists 65–74 publications: 35 scientists 75–84 publications: 84 scientists 85–94 publications: 102 scientists 95–104 publications: 151 scientists 105–114 publications: 175 scientists 115–124 publications: 203 scientists 125–134 publications: 217 scientists 135–144 publications: 205 scientists 145–154 publications: 193 scientists 155–164 publications: 188 scientists 165–174 publications: 170 scientists 175–184 publications: 178 scientists 185–194 publications: 164 scientists 195–204 publications: 173 scientists 205–214 publications: 159 scientists 215–224 publications: 134 scientists 225–234 publications: 143 scientists 235–244 publications: 105 scientists 245–254 publications: 114 scientists 255–264 publications: 92 scientists 265–274 publications: 88 scientists 275–284 publications: 87 scientists 285–294 publications: 80 scientists 295–304 publications: 62 scientists 305–314 publications: 75 scientists 315–324 publications: 67 scientists 325–334 publications: 60 scientists 335–344 publications: 52 scientists 345–354 publications: 40 scientists 355–364 publications: 48 scientists 365–374 publications: 47 scientists 375–384 publications: 46 scientists 385–394 publications: 31 scientists 395–404 publications: 27 scientists 405–414 publications: 40 scientists 415–424 publications: 30 scientists 425–434 publications: 43 scientists 435–444 publications: 29 scientists 445–454 publications: 14 scientists 455–464 publications: 28 scientists 465–474 publications: 21 scientists 475–484 publications: 21 scientists 485–494 publications: 22 scientists 495–504 publications: 17 scientists 505–514 publications: 12 scientists 515–524 publications: 11 scientists 525–534 publications: 8 scientists 535–544 publications: 8 scientists 545–554 publications: 14 scientists 555–564 publications: 4 scientists 565–574 publications: 11 scientists 575–584 publications: 5 scientists 585–594 publications: 11 scientists 595–604 publications: 12 scientists 605–614 publications: 7 scientists 615–624 publications: 6 scientists 625–634 publications: 10 scientists 635–644 publications: 9 scientists 645–654 publications: 10 scientists 655–664 publications: 6 scientists 665–674 publications: 6 scientists 675–684 publications: 6 scientists 685–694 publications: 4 scientists 695–702 publications: 6 scientists 703+ publications: 100 scientists
45 publications 703+

This scientist: 295 publications — 75th percentile

75% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 703 publications or more.

Harry T. Orr D-index placement in Genetics in 2026

The chart shows the D-index (discipline H-index) distribution of Genetics scientists ranked by Research.com in 2026. The highlighted bar marks where Harry T. Orr sits on this spectrum.

40–41 D-Index: 24 scientists 42–43 D-Index: 52 scientists 44–45 D-Index: 84 scientists 46–47 D-Index: 112 scientists 48–49 D-Index: 118 scientists 50–51 D-Index: 141 scientists 52–53 D-Index: 143 scientists 54–55 D-Index: 145 scientists 56–57 D-Index: 179 scientists 58–59 D-Index: 162 scientists 60–61 D-Index: 175 scientists 62–63 D-Index: 191 scientists 64–65 D-Index: 172 scientists 66–67 D-Index: 184 scientists 68–69 D-Index: 164 scientists 70–71 D-Index: 158 scientists 72–73 D-Index: 150 scientists 74–75 D-Index: 136 scientists 76–77 D-Index: 127 scientists 78–79 D-Index: 127 scientists 80–81 D-Index: 111 scientists 82–83 D-Index: 110 scientists 84–85 D-Index: 110 scientists 86–87 D-Index: 84 scientists 88–89 D-Index: 102 scientists 90–91 D-Index: 66 scientists 92–93 D-Index: 72 scientists 94–95 D-Index: 70 scientists 96–97 D-Index: 54 scientists 98–99 D-Index: 60 scientists 100–101 D-Index: 49 scientists 102–103 D-Index: 55 scientists 104–105 D-Index: 45 scientists 106–107 D-Index: 42 scientists 108–109 D-Index: 28 scientists 110–111 D-Index: 39 scientists 112–113 D-Index: 25 scientists 114–115 D-Index: 31 scientists 116–117 D-Index: 29 scientists 118–119 D-Index: 34 scientists 120–121 D-Index: 29 scientists 122–123 D-Index: 29 scientists 124–125 D-Index: 18 scientists 126–127 D-Index: 27 scientists 128–129 D-Index: 22 scientists 130–131 D-Index: 16 scientists 132–133 D-Index: 11 scientists 134–135 D-Index: 17 scientists 136–137 D-Index: 12 scientists 138–139 D-Index: 21 scientists 140–141 D-Index: 4 scientists 142–143 D-Index: 9 scientists 144–145 D-Index: 14 scientists 146–147 D-Index: 6 scientists 148–149 D-Index: 10 scientists 150–151 D-Index: 7 scientists 152–153 D-Index: 9 scientists 154–155 D-Index: 8 scientists 156–157 D-Index: 8 scientists 158–159 D-Index: 9 scientists 160+ D-Index: 96 scientists
40 D-Index 160+

This scientist: 104 D-Index — 85th percentile

85% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 160 D-Index or more.

Overview

Harry T. Orr is affiliated with the University of Minnesota in the United States. Their research primarily spans the fields of Biochemistry, Genetics and Molecular Biology, and Neuroscience. Within these main fields, they focus on subfields including Molecular Biology, Cellular and Molecular Neuroscience, Neurology, Genetics, and Cognitive Neuroscience.

The scientist's work concentrates on various main topics, notably Genetic Neurodegenerative Diseases, Mitochondrial Function and Pathology, RNA Research and Splicing, Genetics and Neurodevelopmental Disorders, Autism Spectrum Disorder Research, DNA Repair Mechanisms, and Muscle Physiology and Disorders.

Harry T. Orr has contributed to several recent publications, including:

  • Patterns of CAG repeat instability in the central nervous system and periphery in Huntington's disease and in spinocerebellar ataxia type 1, 2020, Human Molecular Genetics
  • Autistic-like behavior and cerebellar dysfunction in Bmal1 mutant mice ameliorated by mTORC1 inhibition, 2022, Molecular Psychiatry
  • Targeting inhibitory cerebellar circuitry to alleviate behavioral deficits in a mouse model for studying idiopathic autism, 2020, Neuropsychopharmacology
  • Consensus Paper: Strengths and Weaknesses of Animal Models of Spinocerebellar Ataxias and Their Clinical Implications, 2021, The Cerebellum
  • Altered Capicua expression drives regional Purkinje neuron vulnerability through ion channel gene dysregulation in spinocerebellar ataxia type 1, 2020, Human Molecular Genetics

Frequent co-authors in Harry T. Orr's publications include Lisa Duvick, Huda Y. Zoghbi, Hillary P. Handler, Praseuth Yang, and Marija Cvetanović.

Common venues for Harry T. Orr's publications include bioRxiv (Cold Spring Harbor Laboratory), Neuron, Human Molecular Genetics, JCI Insight, and The Cerebellum.

Best Publications

  • Expansion of an unstable trinucleotide CAG repeat in spinocerebellar ataxia type 1.

    Harry T. Orr;Ming yi Chung;Sandro Banfi;Thomas J. Kwiatkowski

  • Trinucleotide Repeat Disorders

    Harry T. Orr;Huda Y. Zoghbi

  • Glutamine Repeats and Neurodegeneration

    Huda Y. Zoghbi;Harry T. Orr

  • Genetic linkage evidence for a familial Alzheimer's disease locus on chromosome 14.

    Gerard D. Schellenberg;Thomas D. Bird;Ellen M. Wijsman;Harry T. Orr

  • Ataxin-1 nuclear localization and aggregation: role in polyglutamine-induced disease in SCA1 transgenic mice.

    Ivan A Klement;Pamela J Skinner;Michael D Kaytor;Hong Yi

  • Chaperone suppression of ataxin-1 aggregation and altered subcellular proteasome localization imply protein misfolding in sca1

    Huda Y Zoghbi;Harry T Orr;Donald B Defranco;Michael A Mancini

  • RNAi suppresses polyglutamine-induced neurodegeneration in a model of spinocerebellar ataxia

    Haibin Xia;Qinwen Mao;Steven L Eliason;Scott Q Harper

  • Identification of genes that modify ataxin-1-induced neurodegeneration

    Pedro Fernandez-Funez;Maria Laura Nino-Rosales;Beatrice De Gouyon;Wei Chi She

  • SCA1 transgenic mice: A model for neurodegeneration caused by an expanded CAG trinucleotide repeat

    Eric N Burright;H Brent Clark;Antonio Servadio;Toni Matilla

  • A HUMAN MAJOR HISTOCOMPATIBILITY COMPLEX CLASS I GENE THAT ENCODES A PROTEIN WITH A SHORTENED CYTOPLASMIC SEGMENT

    Daniel E. Geraghty;Beverly H. Koller;Harry T. Orr

  • Major histocompatibility antigens: The human (HLA-A,-B,-C) and murine (H-2K, H-2D) class I molecules

    Hidde L. Ploegh;Harry T. Orr;Jack L. Strominger

  • Ataxin-1 with an expanded glutamine tract alters nuclear matrix-associated structures

    Pamela J. Skinner;Beena T. Koshy;Christopher J. Cummings;Ivan A. Klement

  • Over-expression of inducible HSP70 chaperone suppresses neuropathology and improves motor function in SCA1 mice

    Christopher J. Cummings;Yaling Sun;Puneet Opal;Barbara Antalffy

  • Mutation of the E6-AP Ubiquitin Ligase Reduces Nuclear Inclusion Frequency While Accelerating Polyglutamine-Induced Pathology in SCA1 Mice

    Christopher J Cummings;Eyal Reinstein;Yaling Sun;Barbara Antalffy

  • Evidence for a mechanism predisposing to intergenerational CAG repeat instability in spinocerebellar ataxia type I

    Ming Yi Chung;Laura P.W. Ranum;Lisa A. Duvick;Antonio Servadio

  • Interaction of Akt-Phosphorylated Ataxin-1 with 14-3-3 Mediates Neurodegeneration in Spinocerebellar Ataxia Type 1

    Hung Kai Chen;Hung Kai Chen;Pedro Fernandez-Funez;Summer F. Acevedo;Yung C. Lam

  • Genetic linkage studies suggest that Alzheimer's disease is not a single homogeneous disorder

    P. H. St George-Hyslop;J. L. Haines;L. A. Farrer;R. Polinsky

  • Linkage map of the human major histocompatibility complex including the tumor necrosis factor genes

    Michael C. Carroll;Philip Katzman;Elizabeth M. Alicot;Beverly H. Koller

  • Identification and characterization of the gene causing type 1 spinocerebellar ataxia

    Sandro Banfi;Antonio Servadio;Ming yi Chung;Thomas J. Kwiatkowski

  • Polyglutamine expansion down-regulates specific neuronal genes before pathologic changes in SCA1

    Xi Lin;Barbara Antalffy;Dongcheul Kang;Harry T. Orr

Frequent Co-Authors

Huda Y. Zoghbi
Huda Y. Zoghbi Baylor College of Medicine
Jack L. Strominger
Jack L. Strominger Harvard University
Beverly H. Koller
Beverly H. Koller University of North Carolina at Chapel Hill
Laura P.W. Ranum
Laura P.W. Ranum University of Florida
Peter Parham
Peter Parham Stanford University
Stephen S. Rich
Stephen S. Rich University of Virginia
Juan Botas
Juan Botas Baylor College of Medicine
Hidde L. Ploegh
Hidde L. Ploegh Boston Children's Hospital
Thomas D. Bird
Thomas D. Bird University of Washington
Daniel E. Geraghty
Daniel E. Geraghty Fred Hutchinson Cancer Research Center

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