His main research concerns Genetics, Alzheimer's disease, Locus, Gene and Degenerative disease. His work on Age of onset expands to the thematically related Genetics. His Alzheimer's disease research integrates issues from Dementia, Physiology and Genetic testing.
Thomas D. Bird works mostly in the field of Locus, limiting it down to topics relating to Charcot-Marie-Tooth Disease Type 1A and, in certain cases, Peripheral myelin protein 22, as a part of the same area of interest. The various areas that he examines in his Degenerative disease study include Genetic determinism and Progressive supranuclear palsy. His Mutation research is multidisciplinary, incorporating perspectives in Molecular biology, Microtubule, Neuroscience and Exon.
Thomas D. Bird spends much of his time researching Genetics, GeneReviews, Pathology, Internal medicine and Disease. His is doing research in Locus, Gene, Mutation, Genetic linkage and Allele, both of which are found in Genetics. His studies deal with areas such as Genetic heterogeneity and Gene mapping as well as Locus.
His research in Allele intersects with topics in Apolipoprotein E and Genotype. Thomas D. Bird interconnects Dermatology, Molecular biology and Pediatrics in the investigation of issues within GeneReviews. His Internal medicine research includes elements of Gastroenterology and Endocrinology.
His scientific interests lie mostly in GeneReviews, Internal medicine, Pediatrics, Pathology and Dermatology. His GeneReviews research entails a greater understanding of Genetics. His study in Internal medicine is interdisciplinary in nature, drawing from both Gastroenterology, Endocrinology and Oncology.
All of his Pathology and Spinocerebellar ataxia and Neuropathology investigations are sub-components of the entire Pathology study.
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Secreted amyloid beta-protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease.
D. Scheuner;C. Eckman;C. Eckman;M. Jensen;X. Song.
Nature Medicine (1996)
Candidate gene for the chromosome 1 familial Alzheimer's disease locus
Ephrat Levy-Lahad;Wilma Wasco;Parvoneh Poorkaj;Donna M. Romano.
Common variants at MS4A4/MS4A6E, CD2AP, CD33 and EPHA1 are associated with late-onset Alzheimer's disease.
Adam C. Naj;Gyungah Jun;Gary W. Beecham;Li-San Wang.
Nature Genetics (2011)
Tau is a candidate gene for chromosome 17 frontotemporal dementia.
Parvoneh Poorkaj;Thomas D. Bird;Ellen Wijsman;Ellen Nemens.
Annals of Neurology (1998)
Genetic linkage evidence for a familial Alzheimer's disease locus on chromosome 14.
Gerard D. Schellenberg;Thomas D. Bird;Ellen M. Wijsman;Harry T. Orr.
Mutation-Specific Functional Impairments in Distinct Tau Isoforms of Hereditary FTDP-17
Ming Hong;Victoria Zhukareva;Vanessa Vogelsberg-Ragaglia;Zbigniew Wszolek.
A familial Alzheimer's disease locus on chromosome 1
Ephrat Levy-Lahad;Ellen M. Wijsman;Ellen Nemens;Leojean Anderson.
Genetics of Alzheimer Disease
Lynn M. Bekris;Chang-En Yu;Thomas D. Bird;Debby W. Tsuang.
Journal of Geriatric Psychiatry and Neurology (2010)
DNA deletion associated with hereditary neuropathy with liability to pressure palsies
Phillip F. Chance;Phillip F. Chance;Mary Kathryn Alderson;Mary Kathryn Alderson;Mary Kathryn Alderson;Kathleen A. Leppig;M.William Lensch.
TARDBP mutations in amyotrophic lateral sclerosis with TDP-43 neuropathology: a genetic and histopathological analysis
Vivianna M. Van Deerlin;James B. Leverenz;James B. Leverenz;Lynn M. Bekris;Thomas D. Bird;Thomas D. Bird.
Lancet Neurology (2008)
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