H. Phillip Koeffler mostly deals with Cancer research, Molecular biology, Internal medicine, Leukemia and Endocrinology. His Cancer research research includes themes of Carcinogenesis, Cancer, Cell cycle, Tumor suppressor gene and Cell growth. His biological study spans a wide range of topics, including Myeloid, Chronic myelogenous leukemia, Ccaat-enhancer-binding proteins, Myeloid leukemia and Gene.
His studies in Myeloid integrate themes in fields like Tyrosine kinase and Haematopoiesis. His Leukemia research incorporates themes from Mutation, Neuroblastoma RAS viral oncogene homolog and Lymphoma. His Endocrinology study which covers Apoptosis that intersects with Tumor necrosis factor alpha.
The scientist’s investigation covers issues in Cancer research, Molecular biology, Cell growth, Internal medicine and Gene. The various areas that H. Phillip Koeffler examines in his Cancer research study include Cell culture, Cancer, Cell cycle, Apoptosis and Leukemia. His research in Molecular biology intersects with topics in Gene expression, Myeloid, Transcription factor, Ccaat-enhancer-binding proteins and Loss of heterozygosity.
His Myeloid research focuses on Haematopoiesis and how it relates to Bone marrow. H. Phillip Koeffler combines subjects such as Signal transduction and Pathology with his study of Cell growth. His Internal medicine study incorporates themes from Endocrinology and Oncology.
His primary scientific interests are in Cancer research, Transcription factor, Cell biology, Enhancer and Gene silencing. H. Phillip Koeffler has researched Cancer research in several fields, including Apoptosis, Epigenomics, Epigenetics, Bromodomain and In vivo. His research investigates the connection between Apoptosis and topics such as Cell culture that intersect with problems in Kinase.
H. Phillip Koeffler works mostly in the field of Epigenetics, limiting it down to topics relating to Cancer and, in certain cases, Bioinformatics. His research integrates issues of Chromatin, Transcriptome and Myeloid in his study of Cell biology. His Haematopoiesis research incorporates elements of RNA splicing and Intron.
H. Phillip Koeffler spends much of his time researching Cancer research, Transcription factor, Regulation of gene expression, Cell biology and Transcription. The Cancer research study combines topics in areas such as Cell growth, Apoptosis, Cell cycle, Epigenomics and Bromodomain. The study incorporates disciplines such as Histone deacetylase inhibitor and Belinostat in addition to Cell cycle.
His research in Transcription factor intersects with topics in Chromatin immunoprecipitation, Receptor tyrosine kinase, Gene silencing and Long non-coding RNA. He usually deals with Cell biology and limits it to topics linked to Transcriptome and Ccaat-enhancer-binding proteins, Bone marrow, Effector, Inflammation and Progenitor. His Pathology study combines topics in areas such as Cell culture and Kinase.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Frequent pathway mutations of splicing machinery in myelodysplasia
Kenichi Yoshida;Masashi Sanada;Yuichi Shiraishi;Daniel Nowak.
Human cathelicidin antimicrobial peptide (CAMP) gene is a direct target of the vitamin D receptor and is strongly up-regulated in myeloid cells by 1,25-dihydroxyvitamin D3
Adrian F. Gombart;Niels Borregaard;H. Phillip Koeffler.
The FASEB Journal (2005)
The Circadian Gene Per1 Plays an Important Role in Cell Growth and DNA Damage Control in Human Cancer Cells
Sigal Gery;Naoki Komatsu;Lilit Baldjyan;Andrew Yu.
Molecular Cell (2006)
Molecular Analysis of the Cyclin-dependent Kinase Inhibitor Gene p27/Kip1 in Human Malignancies
Norihiko Kawamata;Roberta Morosetti;Carl W. Miller;Dorothy Park.
Cancer Research (1995)
Peroxisome proliferator-activated receptor γ and cancers
H. Phillip Koeffler.
Clinical Cancer Research (2003)
Landscape Of Genetic Lesions In 944 Patients With Myelodysplastic Syndromes
Yasunobu Nagata;Vera Grossmann;Yusuke Okuno;Ulrike Bacher.
Maftools: efficient and comprehensive analysis of somatic variants in cancer.
Anand Mayakonda;Anand Mayakonda;De Chen Lin;Yassen Assenov;Yassen Assenov;Christoph Plass;Christoph Plass.
Genome Research (2018)
Gain-of-function of mutated C-CBL tumour suppressor in myeloid neoplasms.
Masashi Sanada;Takahiro Suzuki;Lee-Yung Shih;Makoto Otsu.
Capsaicin, a Component of Red Peppers, Inhibits the Growth of Androgen-Independent, p53 Mutant Prostate Cancer Cells
Akio Mori;Sören Lehmann;James O'Kelly;Takashi Kumagai.
Cancer Research (2006)
Genomic and molecular characterization of esophageal squamous cell carcinoma
De-Chen Lin;De-Chen Lin;Jia-Jie Hao;Yasunobu Nagata;Liang Xu.
Nature Genetics (2014)
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