The scientist’s investigation covers issues in Alzheimer's disease, Dementia, Internal medicine, Pathology and Disease. His Alzheimer's disease research is multidisciplinary, relying on both Clinical trial, Psychiatry, Immunology, Parkinsonism and Degenerative disease. The Clinical trial study which covers Randomized controlled trial that intersects with Neuroimaging, Neurocognitive, REM sleep behavior disorder and Intensive care medicine.
His Dementia research incorporates elements of Neuropathology, Neuroscience and Gerontology. His Internal medicine research includes elements of Endocrinology and Oncology. Disease is closely attributed to Genetic variation in his work.
His primary areas of investigation include Dementia, Internal medicine, Disease, Alzheimer's disease and Pathology. His Dementia study incorporates themes from Neuropathology, Psychiatry, Cognition and Parkinson's disease. His Internal medicine research integrates issues from Endocrinology and Oncology.
James B. Leverenz focuses mostly in the field of Disease, narrowing it down to matters related to Immunology and, in some cases, Neurodegeneration. The concepts of his Alzheimer's disease study are interwoven with issues in Age of onset, Single-nucleotide polymorphism, Neuroscience, Cognitive decline and Degenerative disease. His Senile plaques, Autopsy, Amyloid and Pathogenesis investigations are all subjects of Pathology research.
James B. Leverenz focuses on Disease, Dementia, Internal medicine, Oncology and Immunology. His Disease research is mostly focused on the topic Dementia with Lewy bodies. James B. Leverenz combines subjects such as Clinical trial, Gerontology, Geriatric psychiatry, Randomized controlled trial and Intensive care medicine with his study of Dementia with Lewy bodies.
He works in the field of Dementia, namely Lewy body. His Oncology research is multidisciplinary, incorporating perspectives in Alzheimer's disease, Clinical Dementia Rating, Plasma biomarkers, Inflammatory pathways and Mild cognitive impairment. His research in Alzheimer's disease tackles topics such as Genetics which are related to areas like Presenilin.
His primary areas of study are Disease, Dementia, Internal medicine, Dementia with Lewy bodies and Cognitive decline. He performs integrative study on Disease and Epiphenomenon. His research in Dementia intersects with topics in TREM2 and Neurology.
James B. Leverenz frequently studies issues relating to Oncology and Internal medicine. His study in Dementia with Lewy bodies is interdisciplinary in nature, drawing from both Geriatric psychiatry, Gerontology and Intensive care medicine. His Alzheimer's Disease Neuroimaging Initiative study also includes fields such as
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Diagnosis and management of dementia with Lewy bodies Fourth consensus report of the DLB Consortium
Ian G. McKeith;Bradley F. Boeve;Dennis W. DIckson;Glenda Halliday.
Neurology (2017)
Correlation of Alzheimer Disease Neuropathologic Changes With Cognitive Status: A Review of the Literature
Peter T Nelson;Irina Alafuzoff;Eileen H Bigio;Constantin Bouras.
Journal of Neuropathology and Experimental Neurology (2012)
Intranasal Insulin Therapy for Alzheimer Disease and Amnestic Mild Cognitive Impairment
Suzanne Craft;Laura D. Baker;Thomas J. Montine;Satoshi Minoshima.
JAMA Neurology (2012)
Analysis of shared heritability in common disorders of the brain
Verneri Anttila;Verneri Anttila;Brendan Bulik-Sullivan;Brendan Bulik-Sullivan;Hilary K. Finucane;Raymond K. Walters;Raymond K. Walters.
Science (2018)
The Parkinson Progression Marker Initiative (PPMI)
Kenneth Marek;Danna Jennings;Shirley Lasch;Andrew Siderowf.
Progress in Neurobiology (2011)
Neuropathological assessment of Parkinson's disease: refining the diagnostic criteria.
Dennis W Dickson;Heiko Braak;John E Duda;Charles Duyckaerts.
Lancet Neurology (2009)
TARDBP mutations in amyotrophic lateral sclerosis with TDP-43 neuropathology: a genetic and histopathological analysis
Vivianna M. Van Deerlin;James B. Leverenz;James B. Leverenz;Lynn M. Bekris;Thomas D. Bird;Thomas D. Bird.
Lancet Neurology (2008)
Rare coding variants in PLCG2, ABI3, and TREM2 implicate microglial-mediated innate immunity in Alzheimer's disease
Rebecca Sims;Sven J. Van Der Lee;Adam C. Naj;Céline Bellenguez;Céline Bellenguez.
Nature Genetics (2017)
DJ-1 and α-synuclein in human cerebrospinal fluid as biomarkers of Parkinson's disease
Zhen Hong;Min Shi;Kathryn A. Chung;Joseph F. Quinn.
Brain (2010)
DLB and PDD boundary issues: Diagnosis, treatment, molecular pathology, and biomarkers
C. F. Lippa;J. E. Duda;M. Grossman;H. I. Hurtig.
Neurology (2007)
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