2002 - Fellow of the American Association for the Advancement of Science (AAAS)
1996 - Fellow of the American Academy of Arts and Sciences
1994 - Member of the National Academy of Medicine (NAM)
1994 - Paul Ehrlich and Ludwig Darmstaedter Prize
1993 - Member of the National Academy of Sciences
His primary areas of investigation include Molecular biology, Cell biology, Genetics, Gene and Ubiquitin. His Molecular biology research incorporates elements of Plasma protein binding, Retinoblastoma protein, Tumor suppressor gene, Gene product and Papillomavirus E7 Proteins. He interconnects Retinoblastoma-Like Protein p107 and Papillomaviridae in the investigation of issues within Gene product.
In his work, Ubiquitin-conjugating enzyme, Protein degradation and Ubiquitin-Protein Ligases is strongly intertwined with F-box protein, which is a subfield of Cell biology. His Ubiquitin research includes themes of Cell cycle, Kinase and Proteasome. His Mutation research is multidisciplinary, incorporating perspectives in Carcinogenesis, Cancer research and Phosphorylation.
Peter M. Howley mostly deals with Molecular biology, Cell biology, Genetics, Gene and Bovine papillomavirus. His work in Molecular biology covers topics such as Virus which are related to areas like Restriction enzyme. As a part of the same scientific family, Peter M. Howley mostly works in the field of Cell biology, focusing on Ubiquitin and, on occasion, Proteasome.
Specifically, his work in Gene is concerned with the study of Transcription. His Bovine papillomavirus research is multidisciplinary, incorporating elements of Enhancer, Repressor and Transactivation. His Papovavirus study in the realm of Virology interacts with subjects such as BK virus.
His main research concerns Type 1 diabetes, Insulin, Internal medicine, Postprandial and Demographic economics. The study incorporates disciplines such as Cross-sectional study, Pandemic, Glycemic and Pediatrics in addition to Type 1 diabetes. His Postprandial research incorporates themes from Insulin dose, Meal and Bolus.
His work is dedicated to discovering how Demographic economics, Unemployment are connected with Personality, Life satisfaction and Quality of life and other disciplines. His research in Cancer intersects with topics in Molecular biology, Ubiquitin, Point mutation and Proteasome. His primary area of study in Ubiquitin is in the field of Ubiquitin ligase.
His scientific interests lie mostly in Type 1 diabetes, Pediatrics, Life satisfaction, Ubiquitin ligase and Diabetes mellitus. The various areas that Peter M. Howley examines in his Type 1 diabetes study include Newly diagnosed, Emergency department and Appetite. His Life satisfaction study combines topics in areas such as Psychological intervention, Risk-seeking and Confounding.
His Ubiquitin ligase research integrates issues from Angelman syndrome, RIG-I, Immune system and Cell biology, Proteasome. His biological study spans a wide range of topics, including Young adult, Overweight, Obesity and Cross-sectional study. As part of his studies on Papillomavirus E7 Proteins, he often connects relevant areas like Molecular biology.
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The E6 oncoprotein encoded by human papillomavirus types 16 and 18 promotes the degradation of p53
Martin Scheffner;Bruce A. Werness;Jon M. Huibregtse;Arnold J. Levine.
The human papilloma virus-16 E7 oncoprotein is able to bind to the retinoblastoma gene product
Nicholas Dyson;Peter M. Howley;Karl Münger;Ed Harlow.
Association of human papillomavirus types 16 and 18 E6 proteins with p53.
Bruce A. Werness;Arnold J. Levine;Peter M. Howley.
The HPV-16 E6 and E6-AP complex functions as a ubiquitin-protein ligase in the ubiquitination of p53
Martin Scheffner;Jon M. Huibregtse;Richard D. Vierstra;Peter M. Howley.
The E6 and E7 genes of the human papillomavirus type 16 together are necessary and sufficient for transformation of primary human keratinocytes.
K Münger;W C Phelps;V Bubb;P M Howley.
Journal of Virology (1989)
Complex formation of human papillomavirus E7 proteins with the retinoblastoma tumor suppressor gene product.
K Münger;B A Werness;N Dyson;W C Phelps.
The EMBO Journal (1989)
A family of proteins structurally and functionally related to the E6-AP ubiquitin-protein ligase.
Jon M. Huibregtse;Martin Scheffner;Sylvie Beaudenon;Peter M. Howley.
Proceedings of the National Academy of Sciences of the United States of America (1995)
The Molecular Basis of Cancer
John Mendelsohn;Peter M. Howley;Mark A. Israel;Joe W. Gray.
The state of the p53 and retinoblastoma genes in human cervical carcinoma cell lines
Martin Scheffner;Karl Münger;Janet C. Byrne;Peter M. Howley.
Proceedings of the National Academy of Sciences of the United States of America (1991)
Human papillomavirus immortalization and transformation functions.
Karl Münger;Peter M Howley.
Virus Research (2002)
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