1996 - Fellow of the American Association for the Advancement of Science (AAAS)
His primary areas of investigation include Cancer research, Molecular biology, Papillomaviridae, Immunology and Cancer. His Cancer research study frequently draws connections between related disciplines such as Pathology. His Molecular biology research incorporates themes from Cellular differentiation, DNA synthesis, Promoter, Transcription and DNA replication.
His biological study spans a wide range of topics, including Genetically modified mouse and Head and neck cancer. He has researched Genetically modified mouse in several fields, including Carcinogenesis, Papillomavirus E7 Proteins and Epidermis, Cell biology. His Cancer research is multidisciplinary, relying on both Mutagenesis and Receptor.
Paul F. Lambert mostly deals with Cancer research, Cancer, Carcinogenesis, Immunology and Molecular biology. His work carried out in the field of Cancer research brings together such families of science as Cell, Cervical cancer, Oncogene, Genetically modified mouse and Papillomavirus E7 Proteins. Many of his studies involve connections with topics such as Pathology and Cancer.
His research investigates the connection between Carcinogenesis and topics such as PI3K/AKT/mTOR pathway that intersect with issues in Protein kinase B. His Immunology research incorporates elements of Papillomaviridae and Fanconi anemia. His work is dedicated to discovering how Molecular biology, Cellular differentiation are connected with Cell culture and Cell biology and other disciplines.
His primary scientific interests are in Cancer research, Carcinogenesis, Cancer, Oncogene and PI3K/AKT/mTOR pathway. Paul F. Lambert interconnects Cell, Gene expression, Head and neck squamous-cell carcinoma, Downregulation and upregulation and HPV infection in the investigation of issues within Cancer research. His studies deal with areas such as Merkel cell, Transgene, Pathogenesis and Carcinogen as well as Carcinogenesis.
The study incorporates disciplines such as Carcinoma and Notch signaling pathway in addition to Cancer. His research in Oncogene intersects with topics in Cervical cancer, Cyclin A2, HeLa, Cyclin E2 and microRNA. Paul F. Lambert usually deals with Mutant and limits it to topics linked to Kinase and Gene.
Paul F. Lambert mainly focuses on Cancer research, Carcinogenesis, Pathogenesis, Downregulation and upregulation and Virus. Paul F. Lambert has included themes like Cancer, Cervical cancer, Cell culture, Gene expression and Oncogene in his Cancer research study. His research integrates issues of Transforming growth factor, Epidermodysplasia verruciformis and Notch signaling pathway in his study of Cancer.
His research is interdisciplinary, bridging the disciplines of Transgene and Carcinogenesis. His Pathogenesis study incorporates themes from Human papillomavirus and Virology. His study in the fields of Viral pathogenesis under the domain of Virology overlaps with other disciplines such as Rodent.
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Integration of human papillomavirus type 16 into the human genome correlates with a selective growth advantage of cells.
Saewha Jeon;B. L. Allen-Hoffmann;P. F. Lambert.
Journal of Virology (1995)
Integration of human papillomavirus type 16 DNA into the human genome leads to increased stability of E6 and E7 mRNAs: implications for cervical carcinogenesis.
Saewha Jeon;Paul F. Lambert.
Proceedings of the National Academy of Sciences of the United States of America (1995)
Fundamental differences in cell cycle deregulation in human papillomavirus-positive and human papillomavirus-negative head/neck and cervical cancers.
Dohun Pyeon;Michael A. Newton;Paul F. Lambert;Johan A. den Boon.
Cancer Research (2007)
Key Characteristics of Carcinogens as a Basis for Organizing Data on Mechanisms of Carcinogenesis.
Martyn T. Smith;Kathryn Z. Guyton;Catherine F. Gibbons;Jason M. Fritz.
Environmental Health Perspectives (2015)
A transcriptional repressor encoded by BPV-1 shares a common carboxy-terminal domain with the E2 transactivator
Paul F. Lambert;Barbara A. Spalholz;Peter M. Howley.
Establishment of Human Papillomavirus Infection Requires Cell Cycle Progression
Dohun Pyeon;Shane M. Pearce;Simon M. Lank;Paul Ahlquist.
PLOS Pathogens (2009)
Organization of Human Papillomavirus Productive Cycle during Neoplastic Progression Provides a Basis for Selection of Diagnostic Markers
Kate Middleton;Woei Peh;Shirley Southern;Heather Griffin.
Journal of Virology (2003)
Dissection of Human Papillomavirus E6 and E7 Function in Transgenic Mouse Models of Cervical Carcinogenesis
Rebeccah R Riley;Stefan Duensing;Tiffany Brake;Karl Münger.
Cancer Research (2003)
Enhanced Radiation Sensitivity in HPV-Positive Head and Neck Cancer
Randall J. Kimple;Molly A. Smith;Grace C. Blitzer;Alexandra D. Torres.
Cancer Research (2013)
The Human Papillomavirus Type 16 E6 Gene Alone Is Sufficient To Induce Carcinomas in Transgenic Animals
Shiyu Song;Henry C. Pitot;Paul F. Lambert.
Journal of Virology (1999)
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