D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Microbiology D-index 77 Citations 18,359 227 World Ranking 821 National Ranking 392

Research.com Recognitions

Awards & Achievements

1996 - Fellow of the American Association for the Advancement of Science (AAAS)

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cancer
  • DNA

His primary areas of investigation include Cancer research, Molecular biology, Papillomaviridae, Immunology and Cancer. His Cancer research study frequently draws connections between related disciplines such as Pathology. His Molecular biology research incorporates themes from Cellular differentiation, DNA synthesis, Promoter, Transcription and DNA replication.

His biological study spans a wide range of topics, including Genetically modified mouse and Head and neck cancer. He has researched Genetically modified mouse in several fields, including Carcinogenesis, Papillomavirus E7 Proteins and Epidermis, Cell biology. His Cancer research is multidisciplinary, relying on both Mutagenesis and Receptor.

His most cited work include:

  • Integration of human papillomavirus type 16 into the human genome correlates with a selective growth advantage of cells. (473 citations)
  • Integration of human papillomavirus type 16 DNA into the human genome leads to increased stability of E6 and E7 mRNAs: implications for cervical carcinogenesis. (444 citations)
  • Fundamental differences in cell cycle deregulation in human papillomavirus-positive and human papillomavirus-negative head/neck and cervical cancers. (331 citations)

What are the main themes of his work throughout his whole career to date?

Paul F. Lambert mostly deals with Cancer research, Cancer, Carcinogenesis, Immunology and Molecular biology. His work carried out in the field of Cancer research brings together such families of science as Cell, Cervical cancer, Oncogene, Genetically modified mouse and Papillomavirus E7 Proteins. Many of his studies involve connections with topics such as Pathology and Cancer.

His research investigates the connection between Carcinogenesis and topics such as PI3K/AKT/mTOR pathway that intersect with issues in Protein kinase B. His Immunology research incorporates elements of Papillomaviridae and Fanconi anemia. His work is dedicated to discovering how Molecular biology, Cellular differentiation are connected with Cell culture and Cell biology and other disciplines.

He most often published in these fields:

  • Cancer research (40.24%)
  • Cancer (24.70%)
  • Carcinogenesis (23.90%)

What were the highlights of his more recent work (between 2017-2021)?

  • Cancer research (40.24%)
  • Carcinogenesis (23.90%)
  • Cancer (24.70%)

In recent papers he was focusing on the following fields of study:

His primary scientific interests are in Cancer research, Carcinogenesis, Cancer, Oncogene and PI3K/AKT/mTOR pathway. Paul F. Lambert interconnects Cell, Gene expression, Head and neck squamous-cell carcinoma, Downregulation and upregulation and HPV infection in the investigation of issues within Cancer research. His studies deal with areas such as Merkel cell, Transgene, Pathogenesis and Carcinogen as well as Carcinogenesis.

The study incorporates disciplines such as Carcinoma and Notch signaling pathway in addition to Cancer. His research in Oncogene intersects with topics in Cervical cancer, Cyclin A2, HeLa, Cyclin E2 and microRNA. Paul F. Lambert usually deals with Mutant and limits it to topics linked to Kinase and Gene.

Between 2017 and 2021, his most popular works were:

  • Loss of Function of Canonical Notch Signaling Drives Head and Neck Carcinogenesis (20 citations)
  • A Human Papillomavirus-Independent Cervical Cancer Animal Model Reveals Unconventional Mechanisms of Cervical Carcinogenesis. (18 citations)
  • Inhibition of TGF-β and NOTCH Signaling by Cutaneous Papillomaviruses. (13 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • DNA

Paul F. Lambert mainly focuses on Cancer research, Carcinogenesis, Pathogenesis, Downregulation and upregulation and Virus. Paul F. Lambert has included themes like Cancer, Cervical cancer, Cell culture, Gene expression and Oncogene in his Cancer research study. His research integrates issues of Transforming growth factor, Epidermodysplasia verruciformis and Notch signaling pathway in his study of Cancer.

His research is interdisciplinary, bridging the disciplines of Transgene and Carcinogenesis. His Pathogenesis study incorporates themes from Human papillomavirus and Virology. His study in the fields of Viral pathogenesis under the domain of Virology overlaps with other disciplines such as Rodent.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Integration of human papillomavirus type 16 into the human genome correlates with a selective growth advantage of cells.

Saewha Jeon;B. L. Allen-Hoffmann;P. F. Lambert.
Journal of Virology (1995)

707 Citations

Integration of human papillomavirus type 16 DNA into the human genome leads to increased stability of E6 and E7 mRNAs: implications for cervical carcinogenesis.

Saewha Jeon;Paul F. Lambert.
Proceedings of the National Academy of Sciences of the United States of America (1995)

656 Citations

Fundamental differences in cell cycle deregulation in human papillomavirus-positive and human papillomavirus-negative head/neck and cervical cancers.

Dohun Pyeon;Michael A. Newton;Paul F. Lambert;Johan A. den Boon.
Cancer Research (2007)

469 Citations

Key Characteristics of Carcinogens as a Basis for Organizing Data on Mechanisms of Carcinogenesis.

Martyn T. Smith;Kathryn Z. Guyton;Catherine F. Gibbons;Jason M. Fritz.
Environmental Health Perspectives (2015)

415 Citations

A transcriptional repressor encoded by BPV-1 shares a common carboxy-terminal domain with the E2 transactivator

Paul F. Lambert;Barbara A. Spalholz;Peter M. Howley.
Cell (1987)

392 Citations

Establishment of Human Papillomavirus Infection Requires Cell Cycle Progression

Dohun Pyeon;Shane M. Pearce;Simon M. Lank;Paul Ahlquist.
PLOS Pathogens (2009)

391 Citations

Organization of Human Papillomavirus Productive Cycle during Neoplastic Progression Provides a Basis for Selection of Diagnostic Markers

Kate Middleton;Woei Peh;Shirley Southern;Heather Griffin.
Journal of Virology (2003)

380 Citations

Dissection of Human Papillomavirus E6 and E7 Function in Transgenic Mouse Models of Cervical Carcinogenesis

Rebeccah R Riley;Stefan Duensing;Tiffany Brake;Karl Münger.
Cancer Research (2003)

364 Citations

Enhanced Radiation Sensitivity in HPV-Positive Head and Neck Cancer

Randall J. Kimple;Molly A. Smith;Grace C. Blitzer;Alexandra D. Torres.
Cancer Research (2013)

363 Citations

The Human Papillomavirus Type 16 E6 Gene Alone Is Sufficient To Induce Carcinomas in Transgenic Animals

Shiyu Song;Henry C. Pitot;Paul F. Lambert.
Journal of Virology (1999)

354 Citations

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