D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Genetics and Molecular Biology D-index 70 Citations 18,325 172 World Ranking 1523 National Ranking 783

Research.com Recognitions

Awards & Achievements

2017 - Fellow of the American Association for the Advancement of Science (AAAS)

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Enzyme

Reuben S. Harris mostly deals with Genetics, APOBEC3A, APOBEC3G, DNA and Cytidine deaminase. His Genetics research focuses on Somatic hypermutation, Adaptive mutation, Mutation, APOBEC1 and APOBEC Deaminases. His work deals with themes such as Cancer, Cancer research and DNA damage, which intersect with Mutation.

Reuben S. Harris studied APOBEC3A and RNA editing that intersect with Cytidine deaminase activity. His studies deal with areas such as Complementary DNA and Retrotransposon as well as APOBEC3G. His DNA study integrates concerns from other disciplines, such as Gene, Deamination and DNA deamination.

His most cited work include:

  • AID mutates E. coli suggesting a DNA deamination mechanism for antibody diversification (796 citations)
  • APOBEC3B is an enzymatic source of mutation in breast cancer (550 citations)
  • Retroviral restriction by APOBEC proteins (518 citations)

What are the main themes of his work throughout his whole career to date?

Reuben S. Harris focuses on DNA, Genetics, APOBEC3G, Cytidine deaminase and Virology. Reuben S. Harris has researched DNA in several fields, including Molecular biology and Cell biology. His Genetics study is mostly concerned with Mutation, Somatic hypermutation, Gene, Mutation and Adaptive mutation.

His Mutation research is multidisciplinary, relying on both Cancer, Breast cancer, Cancer research and Kataegis. His biological study spans a wide range of topics, including Complementary DNA, APOBEC1 and Infectivity. His Cytidine deaminase study incorporates themes from Plasma protein binding and Ubiquitin.

He most often published in these fields:

  • DNA (37.00%)
  • Genetics (34.80%)
  • APOBEC3G (26.74%)

What were the highlights of his more recent work (between 2018-2021)?

  • DNA (37.00%)
  • Cell biology (11.36%)
  • APOBEC (9.89%)

In recent papers he was focusing on the following fields of study:

His main research concerns DNA, Cell biology, APOBEC, Cancer research and APOBEC3G. His work carried out in the field of DNA brings together such families of science as Monoclonal antibody, Small molecule and Enzyme. His APOBEC research incorporates elements of RNA, Cas9 and DNA sequencing.

His Cancer research study combines topics from a wide range of disciplines, such as Immune checkpoint, Cancer, Immunotherapy, Antigen and Lung cancer. His studies examine the connections between APOBEC3G and genetics, as well as such issues in Simian immunodeficiency virus, with regards to Lentivirus, Viral infectivity factor, Viral evolution and Cell. His APOBEC3A study introduces a deeper knowledge of Genetics.

Between 2018 and 2021, his most popular works were:

  • Epstein-Barr virus BORF2 inhibits cellular APOBEC3B to preserve viral genome integrity. (43 citations)
  • Lobular Carcinomas In Situ Display Intralesion Genetic Heterogeneity and Clonal Evolution in the Progression to Invasive Lobular Carcinoma. (26 citations)
  • A panel of eGFP reporters for single base editing by APOBEC-Cas9 editosome complexes (17 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Enzyme

Reuben S. Harris mainly investigates Cell biology, APOBEC, Cancer research, DNA and Cancer. His research integrates issues of RNA and Cytosine in his study of APOBEC. His Cancer research study also includes fields such as

  • Tumor Escape and related Immune checkpoint, Cytidine deaminase, Cancer immunotherapy, Melanoma and Epitope,
  • T cell that connect with fields like Antigen, Small hairpin RNA, Oncolytic virus and Oncolytic Virus Therapy.

His DNA research focuses on APOBEC3A in particular. His APOBEC3A study is associated with Genetics. He combines subjects such as genomic DNA, Molecular biology, Innate immune system and Monoclonal with his study of Cancer.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

AID mutates E. coli suggesting a DNA deamination mechanism for antibody diversification

Svend K. Petersen-Mahrt;Reuben S. Harris;Michael S. Neuberger.
Nature (2002)

1234 Citations

Retroviral restriction by APOBEC proteins

Reuben S. Harris;Mark T. Liddament.
Nature Reviews Immunology (2004)

791 Citations

RNA editing enzyme APOBEC1 and some of its homologs can act as DNA mutators.

Reuben S. Harris;Svend K. Petersen-Mahrt;Michael S. Neuberger.
Molecular Cell (2002)

743 Citations

The Vif Protein of HIV Triggers Degradation of the Human Antiretroviral DNA Deaminase APOBEC3G

Silvestro G Conticello;Reuben S Harris;Michael S Neuberger.
Current Biology (2003)

671 Citations

APOBEC3B is an enzymatic source of mutation in breast cancer

Michael B. Burns;Lela Lackey;Michael A. Carpenter;Anurag Rathore.
Nature (2013)

668 Citations

APOBEC3F properties and hypermutation preferences indicate activity against HIV-1 in vivo.

Mark T. Liddament;William L. Brown;April J. Schumacher;Reuben S. Harris.
Current Biology (2004)

623 Citations

Evidence for APOBEC3B mutagenesis in multiple human cancers

Michael B Burns;Nuri A Temiz;Reuben S Harris.
Nature Genetics (2013)

595 Citations

Comparison of the differential context-dependence of DNA deamination by APOBEC enzymes: correlation with mutation spectra in vivo.

Rupert C.L. Beale;Svend K. Petersen-Mahrt;Svend K. Petersen-Mahrt;Ian N. Watt;Reuben S. Harris.
Journal of Molecular Biology (2004)

460 Citations

APOBECs and virus restriction

Reuben S. Harris;Jaquelin P. Dudley.
Virology (2015)

378 Citations

Vif hijacks CBF-β to degrade APOBEC3G and promote HIV-1 infection

Stefanie Jäger;Dong Young Kim;Judd F. Hultquist;Keisuke Shindo.
Nature (2012)

345 Citations

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