What is he best known for?
The fields of study he is best known for:
His primary scientific interests are in Virology, Virus, Gene, Genetics and Cell culture.
He is interested in Murine leukemia virus, which is a branch of Virology.
His Virus research is multidisciplinary, relying on both Long terminal repeat and Provirus.
In general Gene, his work in Protein domain, TRIM5 Gene, Endogenous retrovirus and Tripartite motif family is often linked to Membrane protein linking many areas of study.
Michael Emerman has researched Cell culture in several fields, including Jurkat cells and Clone.
His Cell cycle study incorporates themes from Mitosis, Cell division and Cell growth.
His most cited work include:
- A SARS-CoV-2 protein interaction map reveals targets for drug repurposing. (1317 citations)
- Detection of replication-competent and pseudotyped human immunodeficiency virus with a sensitive cell line on the basis of activation of an integrated beta-galactosidase gene. (865 citations)
- The Vpr protein of human immunodeficiency virus type 1 influences nuclear localization of viral nucleic acids in nondividing host cells (799 citations)
What are the main themes of his work throughout his whole career to date?
His main research concerns Virology, Virus, Genetics, Gene and Viral replication.
His Virology research includes elements of Cell cycle and Cell culture.
His research integrates issues of Mitosis, Cell division and Long terminal repeat in his study of Cell cycle.
His Virus research integrates issues from Mutation, Molecular biology and Provirus.
As a part of the same scientific study, Michael Emerman usually deals with the Gene, concentrating on Innate immune system and frequently concerns with Ubiquitin.
His Viral replication research incorporates elements of Phenotype, Cell nucleus, Nuclear localization sequence and DNA.
He most often published in these fields:
- Virology (66.27%)
- Virus (38.46%)
- Genetics (34.91%)
What were the highlights of his more recent work (between 2016-2021)?
- Gene (31.95%)
- Genetics (34.91%)
- Virology (66.27%)
In recent papers he was focusing on the following fields of study:
The scientist’s investigation covers issues in Gene, Genetics, Virology, Interferon and Antiviral protein.
While the research belongs to areas of Gene, Michael Emerman spends his time largely on the problem of Innate immune system, intersecting his research to questions surrounding Enzyme, Vero cell, Plasma protein binding and Medical microbiology.
His research brings together the fields of ZINC FINGER ANTIVIRAL PROTEIN and Virology.
His study in Interferon is interdisciplinary in nature, drawing from both Transmembrane protein, Viral replication, CRISPR and Cell biology.
His studies in CRISPR integrate themes in fields like Vector, Virus, Tetherin, Viral vector and Lentivirus.
Michael Emerman combines subjects such as Mutagenesis, Amino acid, Influenza A virus and Target protein with his study of Antiviral protein.
Between 2016 and 2021, his most popular works were:
- A SARS-CoV-2 protein interaction map reveals targets for drug repurposing. (1317 citations)
- A SARS-CoV-2-Human Protein-Protein Interaction Map Reveals Drug Targets and Potential Drug-Repurposing. (187 citations)
- A Virus-Packageable CRISPR Screen Identifies Host Factors Mediating Interferon Inhibition of HIV (30 citations)
In his most recent research, the most cited papers focused on:
His scientific interests lie mostly in Interferon, Reverse transcriptase, Lentivirus, Virology and HEK 293 cells.
The subject of his Interferon research is within the realm of Genetics.
His work deals with themes such as Protein structure, Cytosine, DNA and Enzyme, which intersect with Reverse transcriptase.
Michael Emerman works mostly in the field of Lentivirus, limiting it down to concerns involving CRISPR and, occasionally, Virus, Alpha interferon and Tissue tropism.
The various areas that he examines in his Virology study include Nuclear transport and Phosphorylation.
His HEK 293 cells research includes themes of Cytoplasm, Gene knockdown, Mutant, Capsid and Viral replication.
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