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Immunology

D-Index
52
Citations
16343
World Ranking
4023
National Ranking
1837

Overview

Vineet N. KewalRamani is affiliated with the National Institutes of Health in the United States. Their research primarily focuses on the areas of Biochemistry, Genetics and Molecular Biology, Immunology and Microbiology, and Medicine. Within these fields, their work concentrates notably on subfields such as Molecular Biology, Virology, Infectious Diseases, Epidemiology, and Ecology.

The scientist has contributed extensively to several topics, with a significant emphasis on HIV research. The key topics covered in their work include:

  • HIV Research and Treatment
  • RNA Research and Splicing
  • HIV/AIDS Drug Development and Treatment
  • Bacteriophages and Microbial Interactions
  • Cytomegalovirus and Herpesvirus Research
  • Nuclear Structure and Function
  • RNA Modifications and Cancer

Vineet N. KewalRamani's recent papers demonstrate an ongoing engagement with the molecular mechanisms of HIV and related virological studies. These include:

  • "Nuclear Import of the HIV-1 Core Precedes Reverse Transcription and Uncoating" (2020) published in Cell Reports
  • "Sec24C is an HIV-1 host dependency factor crucial for virus replication" (2021) published in Nature Microbiology
  • "CPSF6-Dependent Targeting of Speckle-Associated Domains Distinguishes Primate from Nonprimate Lentiviral Integration" (2020) published in mBio
  • "Prion-like low complexity regions enable avid virus-host interactions during HIV-1 infection" (2022) published in Nature Communications
  • "GS-CA1 and lenacapavir stabilize the HIV-1 core and modulate the core interaction with cellular factors" (2021) published in iScience

Their work has appeared frequently in journals such as Nature Communications, bioRxiv (Cold Spring Harbor Laboratory), Cell Reports, Nature Microbiology, and mBio. The scientist has multiple publications in Nature Communications and bioRxiv, indicating a sustained contribution to these venues.

Collaboration features prominently in their research with coauthors including:

  • Kyeongeun Lee
  • Felipe Diaz-Griffero
  • Hyun Jae Yu
  • Anastasia Selyutina
  • Ashwanth C. Francis

These collaborations reflect the interdisciplinary and multi-institutional nature of their research projects.

Best Publications

  • DC-SIGN, a dendritic cell-specific HIV-1-binding protein that enhances trans-infection of T cells

    T.B.H. Geijtenbeek;D.S. Kwon;R. Torensma;S.J. van Vliet

  • The Vpr protein of human immunodeficiency virus type 1 influences nuclear localization of viral nucleic acids in nondividing host cells

    Nina K. Heinzinger;Michael I. Bukrinsky;Sheryl A. Haggerty;Anna M. Ragland

  • Recruitment of HIV and its receptors to dendritic cell-T cell junctions.

    David McDonald;Li Wu;Stacy M. Bohks;Vineet N. KewalRamani

  • Expression cloning of new receptors used by simian and human immunodeficiency viruses

    HongKui Deng;Derya Unutmaz;Vineet N. KewalRamani;Dan R. Littman

  • Dendritic-cell interactions with HIV: infection and viral dissemination

    Li Wu;Vineet N. KewalRamani

  • Flexible Use of Nuclear Import Pathways by HIV-1

    KyeongEun Lee;Zandrea Ambrose;Thomas D. Martin;Ilker Oztop

  • HIV-1 capsid-cyclophilin interactions determine nuclear import pathway, integration targeting and replication efficiency.

    Torsten Schaller;Karen E. Ocwieja;Jane Rasaiyaah;Amanda J. Price

  • Cytokine Signals Are Sufficient for HIV-1 Infection of Resting Human T Lymphocytes

    Derya Unutmaz;Vineet N. KewalRamani;Shana Marmon;Dan R. Littman

  • The interaction between HIV-1 Tat and human cyclin T1 requires zinc and a critical cysteine residue that is not conserved in the murine CycT1 protein

    Mitchell E. Garber;Ping Wei;Vineet N. KewalRamani;Timothy P. Mayall

  • Primary Human Immunodeficiency Virus Type 2 (HIV-2) Isolates, Like HIV-1 Isolates, Frequently Use CCR5 but Show Promiscuity in Coreceptor Usage

    Andreas Mörner;Åsa Björndal;Jan Albert;Vineet N. KewalRamani

  • A Dendritic Cell–Specific Intercellular Adhesion Molecule 3–Grabbing Nonintegrin (Dc-Sign)–Related Protein Is Highly Expressed on Human Liver Sinusoidal Endothelial Cells and Promotes HIV-1 Infection

    Arman A Bashirova;Teunis B.H. Geijtenbeek;Gerard C.F. van Duijnhoven;Sandra J. van Vliet

  • CPSF6 defines a conserved capsid interface that modulates HIV-1 replication.

    Amanda J. Price;Adam J. Fletcher;Torsten Schaller;Tom Elliott

  • X-ray crystal structures of native HIV-1 capsid protein reveal conformational variability

    Anna T. Gres;Karen A. Kirby;Vineet N. KewalRamani;John J. Tanner

  • Neutralization Profiles of Primary Human Immunodeficiency Virus Type 1 Isolates in the Context of Coreceptor Usage

    D. Cecilia;Vineet N. KewalRamani;Jeanne O’Leary;Barbara Volsky

  • Exclusive and Persistent Use of the Entry Coreceptor CXCR4 by Human Immunodeficiency Virus Type 1 from a Subject Homozygous for CCR5 Δ32

    Nelson L. Michael;Julie A. E. Nelson;Vineet N. KewalRamani;George Chang

  • Envelope glycoproteins from human immunodeficiency virus types 1 and 2 and simian immunodeficiency virus can use human CCR5 as a coreceptor for viral entry and make direct CD4-dependent interactions with this chemokine receptor.

    C M Hill;H Deng;D Unutmaz;V N Kewalramani

  • The Requirement for Cellular Transportin 3 (TNPO3 or TRN-SR2) during Infection Maps to Human Immunodeficiency Virus Type 1 Capsid and Not Integrase

    Lavanya Krishnan;Kenneth A. Matreyek;Ilker Oztop;Kyeongeun Lee

  • Neutralization Sensitivity of Human Immunodeficiency Virus Type 1 Primary Isolates to Antibodies and CD4-Based Reagents Is Independent of Coreceptor Usage

    Alexandra Trkola;Tom Ketas;Vineet N. KewalRamani;Fred Endorf

  • Use of Coreceptors Other Than CCR5 by Non-Syncytium-Inducing Adult and Pediatric Isolates of Human Immunodeficiency Virus Type 1 Is Rare In Vitro

    Yi-jun Zhang;Tatjana Dragic;Yunzhen Cao;Leondios Kostrikis

  • A macaque model of HIV-1 infection

    Theodora Hatziioannou;Zandrea Ambrose;Zandrea Ambrose;Nancy P. Y. Chung;Michael Piatak

Frequent Co-Authors

Dan R. Littman
Dan R. Littman New York University
Derya Unutmaz
Derya Unutmaz University of Connecticut
Paul D. Bieniasz
Paul D. Bieniasz Rockefeller University
Li Wu
Li Wu University of Iowa
John M. Coffin
John M. Coffin Tufts University
Jeffrey D. Lifson
Jeffrey D. Lifson Leidos (United States)
Michael Emerman
Michael Emerman Fred Hutchinson Cancer Research Center
Frank Maldarelli
Frank Maldarelli National Institutes of Health
Theodora Hatziioannou
Theodora Hatziioannou Rockefeller University
Mary F. Kearney
Mary F. Kearney National Institutes of Health

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