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Microbiology

D-Index
51
Citations
12848
World Ranking
4286
National Ranking
1652

Overview

Mary F. Kearney is affiliated with the National Institutes of Health in the United States. Their research primarily focuses on the fields of Medicine and Immunology and Microbiology, with a strong specialization in infectious diseases and virology. Their work spans molecular biology, epidemiology, and immunology, reflecting a multidisciplinary approach to studying complex viral infections.

The scientist's main research topics include:

  • HIV Research and Treatment
  • HIV/AIDS drug development and treatment
  • HIV/AIDS Research and Interventions
  • SARS-CoV-2 and COVID-19 Research
  • Cytomegalovirus and herpesvirus research
  • Immune Cell Function and Interaction
  • CRISPR and Genetic Engineering

Mary F. Kearney has contributed to multiple publications, often in collaboration with frequent co-authors. Notable co-authors include John W. Mellors, John M. Coffin, Michael J. Bale, Jason W. Rausch, and Adam A. Capoferri.

Frequent venues for publication include:

  • bioRxiv (Cold Spring Harbor Laboratory)
  • Viruses
  • Proceedings of the National Academy of Sciences
  • mBio
  • UNC Libraries

Significant recent papers authored or co-authored by Mary F. Kearney include:

  • HIV-1 viremia not suppressible by antiretroviral therapy can originate from large T cell clones producing infectious virus, 2020, Journal of Clinical Investigation
  • Severe Acute Respiratory Syndrome Coronavirus 2 Viremia Is Associated With Coronavirus Disease 2019 Severity and Predicts Clinical Outcomes, 2021, Clinical Infectious Diseases
  • Low genetic diversity may be an Achilles heel of SARS-CoV-2, 2020, Proceedings of the National Academy of Sciences
  • Integration in oncogenes plays only a minor role in determining the in vivo distribution of HIV integration sites before or during suppressive antiretroviral therapy, 2021, PLoS Pathogens
  • Dynamic Shifts in the HIV Proviral Landscape During Long Term Combination Antiretroviral Therapy: Implications for Persistence and Control of HIV Infections, 2020, Viruses

Best Publications

  • Administration of vorinostat disrupts HIV-1 latency in patients on antiretroviral therapy

    N. M. Archin;A. L. Liberty;A. D. Kashuba;S. K. Choudhary

  • Specific HIV integration sites are linked to clonal expansion and persistence of infected cells

    F. Maldarelli;X. Wu;L. Su;F. R. Simonetti

  • Single-strand specificity of APOBEC3G accounts for minus-strand deamination of the HIV genome.

    Qin Yu;Renate König;Satish Pillai;Kristopher Chiles

  • Multiple, Linked Human Immunodeficiency Virus Type 1 Drug Resistance Mutations in Treatment-Experienced Patients Are Missed by Standard Genotype Analysis

    Sarah Palmer;Mary Kearney;Frank Maldarelli;Elias K. Halvas

  • Treatment intensification does not reduce residual HIV-1 viremia in patients on highly active antiretroviral therapy

    J. B. Dinoso;S. Y. Kim;A. M. Wiegand;S. E. Palmer;S. E. Palmer

  • B-108 Specific HIV integration sites are linked to clonal expansion and persistence of infected cells

    Frank Maldarelli;Xiaolin Wu;Ling Su;Francesco Simonetti

  • Clonally expanded CD4+ T cells can produce infectious HIV-1 in vivo

    Francesco R. Simonetti;Michele D. Sobolewski;Elizabeth Fyne;Wei Shao

  • Histone deacetylase inhibitor romidepsin induces HIV expression in CD4 T cells from patients on suppressive antiretroviral therapy at concentrations achieved by clinical dosing.

    Datsen George Wei;Vicki Chiang;Elizabeth Fyne;Mini Balakrishnan

  • The Effect of Raltegravir Intensification on Low-level Residual Viremia in HIV-Infected Patients on Antiretroviral Therapy: A Randomized Controlled Trial

    Rajesh T. Gandhi;Lu Zheng;Ronald J. Bosch;Ellen S. Chan

  • Proviruses with identical sequences comprise a large fraction of the replication-competent HIV reservoir.

    John K. Bui;John K. Bui;Michele D. Sobolewski;Brandon F. Keele;Jonathan Spindler

  • HIV-1 Expression Within Resting CD4+ T Cells After Multiple Doses of Vorinostat

    Nancy M. Archin;Rosalie Bateson;Manoj K. Tripathy;Amanda M. Crooks

  • Immediate antiviral therapy appears to restrict resting CD4+ cell HIV-1 infection without accelerating the decay of latent infection.

    Nancie M. Archin;Naveen K. Vaidya;Naveen K. Vaidya;Jo Ann D. Kuruc;Abigail L. Liberty

  • Short-course raltegravir intensification does not reduce persistent low-level viremia in patients with HIV-1 suppression during receipt of combination antiretroviral therapy

    D. McMahon;J. Jones;A. Wiegand;S. J. Gange

  • Lack of Detectable HIV-1 Molecular Evolution during Suppressive Antiretroviral Therapy

    Mary F. Kearney;Jonathan Spindler;Wei Shao;Sloane Yu

  • HIV-1 persistence following extremely early initiation of antiretroviral therapy (ART) during acute HIV-1 infection: An observational study

    Timothy J. Henrich;Hiroyu Hatano;Oliver Bacon;Louise E. Hogan

  • Human Immunodeficiency Virus Type 1 Population Genetics and Adaptation in Newly Infected Individuals

    M. Kearney;M. Kearney;F. Maldarelli;W. Shao;J. B. Margolick

  • Majority of CD4+ T cells from peripheral blood of HIV-1–infected individuals contain only one HIV DNA molecule

    Lina Josefsson;Martin S. King;Barbro Makitalo;Johan Brännström

  • Single-cell analysis of HIV-1 transcriptional activity reveals expression of proviruses in expanded clones during ART.

    Ann Wiegand;Jonathan Spindler;Feiyu F. Hong;Wei Shao

  • HIV populations are large and accumulate high genetic diversity in a nonlinear fashion

    Frank Maldarelli;Mary Kearney;Sarah Palmer;Robert Stephens

  • Blinded, Multicenter Comparison of Methods To Detect a Drug-Resistant Mutant of Human Immunodeficiency Virus Type 1 at Low Frequency

    Elias K. Halvas;Grace M. Aldrovandi;Peter Balfe;Ingrid A. Beck

Frequent Co-Authors

John M. Coffin
John M. Coffin Tufts University
John W. Mellors
John W. Mellors University of Pittsburgh
Frank Maldarelli
Frank Maldarelli National Institutes of Health
Sarah Palmer
Sarah Palmer University of Sydney
Stephen Hughes
Stephen Hughes National Cancer Institute
Brandon F. Keele
Brandon F. Keele National Institutes of Health
Rebecca Hoh
Rebecca Hoh University of California, San Francisco
David M. Margolis
David M. Margolis University of North Carolina at Chapel Hill
Joseph J. Eron
Joseph J. Eron University of North Carolina at Chapel Hill
Vineet N. KewalRamani
Vineet N. KewalRamani National Institutes of Health

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