D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Microbiology D-index 55 Citations 14,220 98 World Ranking 1551 National Ranking 648

Research.com Recognitions

Awards & Achievements

Member of the Association of American Physicians

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Enzyme

His primary areas of investigation include Virology, Molecular biology, Viral replication, Cyclophilin A and Cypa. His Innate immune system research extends to Virology, which is thematically connected. His Molecular biology research is multidisciplinary, incorporating elements of RNA, Induced pluripotent stem cell, Rex1, Monocyte and Cytotoxic T cell.

His study on Viral replication is covered under Virus. His Cyclophilin A study combines topics from a wide range of disciplines, such as Infectivity, Cyclophilin, Peptidylprolyl isomerase and Cell biology. His work carried out in the field of Cypa brings together such families of science as Mutation, Gene targeting, Gene, Apoptosis and Isomerase activity.

His most cited work include:

  • Human immunodeficiency virus type 1 Gag protein binds to cyclophilins A and B (695 citations)
  • Specific incorporation of cyclophilin A into HIV-1 virions (657 citations)
  • The retinoblastoma protein and BRG1 form a complex and cooperate to induce cell cycle arrest (576 citations)

What are the main themes of his work throughout his whole career to date?

His scientific interests lie mostly in Virology, Cell biology, Capsid, Viral replication and Cyclophilin A. The concepts of his Virology study are interwoven with issues in Cell culture and Innate immune system. His Cell biology study also includes fields such as

  • Reverse transcriptase that intertwine with fields like Complementary DNA, Ubiquitin ligase and Mutant,
  • Interferon that connect with fields like Transduction.

As part of the same scientific family, Jeremy Luban usually focuses on Capsid, concentrating on Peptide sequence and intersecting with Binding site. Jeremy Luban combines subjects such as RNA and Nuclear transport with his study of Viral replication. His studies deal with areas such as Cyclophilin, Peptidylprolyl isomerase and Biochemistry as well as Cyclophilin A.

He most often published in these fields:

  • Virology (52.20%)
  • Cell biology (27.80%)
  • Capsid (20.49%)

What were the highlights of his more recent work (between 2017-2021)?

  • Cell biology (27.80%)
  • Virology (52.20%)
  • Virus (16.59%)

In recent papers he was focusing on the following fields of study:

Jeremy Luban spends much of his time researching Cell biology, Virology, Virus, Inflammation and Immunology. Jeremy Luban interconnects Reverse transcriptase, Ebola virus, Mutant, Protein structure and Transcription in the investigation of issues within Cell biology. The Reverse transcriptase study combines topics in areas such as Cytoplasm, Cypa, Cyclophilin A, Viral replication and Infectivity.

Virology is frequently linked to Monoclonal antibody in his study. His Virus research incorporates elements of Antibody and Ectopic expression. His Inflammation study combines topics in areas such as Cell and Wnt signaling pathway.

Between 2017 and 2021, his most popular works were:

  • Structural and Functional Analysis of the D614G SARS-CoV-2 Spike Protein Variant. (237 citations)
  • SARS-CoV-2 Spike protein variant D614G increases infectivity and retains sensitivity to antibodies that target the receptor binding domain [preprint] (58 citations)
  • Primate immunodeficiency virus proteins Vpx and Vpr counteract transcriptional repression of proviruses by the HUSH complex. (43 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Enzyme

The scientist’s investigation covers issues in Cell biology, Virus, Coronavirus disease 2019, Severe acute respiratory syndrome coronavirus 2 and Geography. Jeremy Luban has researched Cell biology in several fields, including Reverse transcriptase, Cypa, RNA interference, Zebrafish and Reverse genetics. His Virus study deals with the bigger picture of Virology.

His Virology study integrates concerns from other disciplines, such as Virus classification and Ubiquitin. The study incorporates disciplines such as Infectivity and Genome in addition to Ectopic expression. When carried out as part of a general Antibody research project, his work on Monoclonal antibody is frequently linked to work in Protein trimer, Virion membrane and Susceptible individual, therefore connecting diverse disciplines of study.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Human immunodeficiency virus type 1 Gag protein binds to cyclophilins A and B

Jeremy Luban;Karen Leigh Bossolt;Ettaly K. Franke;Ganjam V. Kalpana.
Cell (1993)

1046 Citations

Specific incorporation of cyclophilin A into HIV-1 virions

Ettaly Kara Franke;Hannah En Hui Yuan;Jeremy Luban.
Nature (1994)

991 Citations

Cyclophilin A retrotransposition into TRIM5 explains owl monkey resistance to HIV-1

David M. Sayah;Elena Sokolskaja;Lionel Berthoux;Jeremy Luban.
Nature (2004)

787 Citations

The retinoblastoma protein and BRG1 form a complex and cooperate to induce cell cycle arrest

Joshua L. Dunaief;Bruce E. Strober;Sushovan Guha;Paul A. Khavari.
Cell (1994)

778 Citations

TRIM5 is an innate immune sensor for the retrovirus capsid lattice

Thomas Pertel;Stéphane Hausmann;Damien Morger;Sara Züger.
Nature (2011)

627 Citations

Cyclophilin A modulates the sensitivity of HIV-1 to host restriction factors.

Greg J Towers;Theodora Hatziioannou;Simone Cowan;Stephen P Goff.
Nature Medicine (2003)

465 Citations

Human immunodeficiency virus type 1 Vpr arrests the cell cycle in G2 by inhibiting the activation of p34cdc2-cyclin B.

F Re;D Braaten;E K Franke;J Luban.
Journal of Virology (1995)

448 Citations

Cyclophilin A is required for an early step in the life cycle of human immunodeficiency virus type 1 before the initiation of reverse transcription.

D Braaten;E K Franke;J Luban.
Journal of Virology (1996)

438 Citations

TCR stimulation with modified anti-CD3 mAb expands CD8+ T cell population and induces CD8+CD25+ Tregs

Brygida Bisikirska;John Colgan;Jeremy Luban;Jeffrey A. Bluestone.
Journal of Clinical Investigation (2005)

398 Citations

Relative efficacy of human monocytes and dendritic cells as accessory cells for T cell replication.

W C Van Voorhis;J Valinsky;E Hoffman;J Luban.
Journal of Experimental Medicine (1983)

362 Citations

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