Chen Liang mainly investigates Virus, Virology, RNA, Genetics and Viral envelope. The concepts of his Virus study are interwoven with issues in Interferon, Small hairpin RNA and COS cells. He focuses mostly in the field of Interferon, narrowing it down to matters related to Innate immune system and, in some cases, Effector, Cell and Viral protein.
Chen Liang has included themes like Membrane protein, RNA-binding protein, Mutant and DNA in his Virology study. His work carried out in the field of Mutant brings together such families of science as Cypa and Cyclophilin A. He interconnects Molecular biology and Genome in the investigation of issues within RNA.
Chen Liang spends much of his time researching Virology, Virus, Viral replication, Molecular biology and RNA. His Virology study which covers Genetics that intersects with Protein structure. His Virus study combines topics in areas such as Innate immune system, RNA-binding protein and Effector.
As a part of the same scientific family, Chen Liang mostly works in the field of Viral replication, focusing on Cell biology and, on occasion, HEK 293 cells, Stress granule and SAMHD1. His Molecular biology research integrates issues from Primer binding site, Reverse transcriptase, Point mutation, Capsid and Group-specific antigen. His RNA study combines topics from a wide range of disciplines, such as Transcription and Mutant.
His primary scientific interests are in Virology, Cell biology, Interferon, Virus and Viral replication. His work deals with themes such as Polymerase, Gene and RNA polymerase, which intersect with Virology. His Cell biology research includes elements of Stress granule, Nuclear export signal and Cytosol.
His Interferon study incorporates themes from Translation, Viral translation, Cell culture and Protein biosynthesis. His Virus study integrates concerns from other disciplines, such as Innate immune system and Immune system. His Viral replication research includes themes of RNA and Dengue virus.
Chen Liang mostly deals with Cell biology, Virus, Viral replication, DNA and Signal transduction. His Cell biology research incorporates themes from Stress granule and APOBEC3G. His work deals with themes such as Interferon, Drug development, Small molecule, Protein structure and Biological evaluation, which intersect with Virus.
The study incorporates disciplines such as Non-coding RNA and RNA polymerase complex, Polymerase, RNA polymerase, RNA-dependent RNA polymerase in addition to Viral replication. His study on DNA also encompasses disciplines like
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The IFITM Proteins Inhibit HIV-1 Infection
Jennifer Lu;Jennifer Lu;Qinghua Pan;Liwei Rong;Shan-Lu Liu;Shan-Lu Liu.
Journal of Virology (2011)
The Interferon-Inducible MxB Protein Inhibits HIV-1 Infection
Zhenlong Liu;Qinghua Pan;Shilei Ding;Shilei Ding;Jin Qian;Jin Qian.
Cell Host & Microbe (2013)
IFITM Proteins Restrict Viral Membrane Hemifusion
Kun Li;Ruben M. Markosyan;Yi-Min Zheng;Ottavia Golfetto.
PLOS Pathogens (2013)
CRISPR/Cas9-Derived Mutations Both Inhibit HIV-1 Replication and Accelerate Viral Escape.
Zhen Wang;Zhen Wang;Qinghua Pan;Patrick Gendron;Weijun Zhu.
Cell Reports (2016)
Methylation of Tat by PRMT6 Regulates Human Immunodeficiency Virus Type 1 Gene Expression
Marie Chloé Boulanger;Chen Liang;Rodney S. Russell;Rongtuan Lin.
Journal of Virology (2005)
Mutations in the kissing-loop hairpin of human immunodeficiency virus type 1 reduce viral infectivity as well as genomic RNA packaging and dimerization.
Michael Laughrea;Louis Jette;Johnson Mak;Lawrence Kleiman.
Journal of Virology (1997)
The CRISPR/Cas9 system inactivates latent HIV-1 proviral DNA
Weijun Zhu;Rongyue Lei;Yann Le Duff;Yann Le Duff;Jian Li.
The N-Terminal Region of IFITM3 Modulates Its Antiviral Activity by Regulating IFITM3 Cellular Localization
Rui Jia;Qinghua Pan;Shilei Ding;Shilei Ding;Liwei Rong.
Journal of Virology (2012)
IFITM Proteins Incorporated into HIV-1 Virions Impair Viral Fusion and Spread
Alex A. Compton;Alex A. Compton;Timothée Bruel;Timothée Bruel;Françoise Porrot;Françoise Porrot;Adeline Mallet.
Cell Host & Microbe (2014)
The Transmembrane Domain of BST-2 Determines Its Sensitivity to Down-Modulation by Human Immunodeficiency Virus Type 1 Vpu
Liwei Rong;Jianyong Zhang;Jennifer Lu;Qinghua Pan.
Journal of Virology (2009)
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