SMN1, Genetics, Spinal muscular atrophy, Molecular biology and Exon are his primary areas of study. His SMN1 research is classified as research in Motor neuron. As part of one scientific family, Elliot J. Androphy deals mainly with the area of Genetics, narrowing it down to issues related to the Cell biology, and often Transcription factor.
His work carried out in the field of Spinal muscular atrophy brings together such families of science as Exonic splicing silencer and SnRNP Biogenesis. His study in Molecular biology is interdisciplinary in nature, drawing from both Messenger RNA, Mutant, Gene, Bovine papillomavirus and Tumor suppressor gene. His RNA splicing research extends to the thematically linked field of Exon.
His primary areas of investigation include Molecular biology, Cell biology, Spinal muscular atrophy, Transcription and SMN1. His research in Molecular biology intersects with topics in Cell culture, Mutant, Gene, Repressor and Bovine papillomavirus. His Cell biology study combines topics from a wide range of disciplines, such as Transcription factor, Viral replication and Gene knockdown.
His Spinal muscular atrophy research incorporates themes from Bioinformatics, Motor neuron and Alternative splicing, Exon. To a larger extent, Elliot J. Androphy studies Genetics with the aim of understanding Exon. Elliot J. Androphy works in the field of SMN1, focusing on Survival of motor neuron in particular.
Elliot J. Androphy spends much of his time researching Cell biology, Spinal muscular atrophy, Viral replication, Transcription and Motor neuron. Elliot J. Androphy is involved in the study of Spinal muscular atrophy that focuses on SMN1 in particular. His research on SMN1 concerns the broader Exon.
His research in Exon focuses on subjects like Disease, which are connected to Genetics. His Viral replication research includes themes of Mutant, Molecular biology, Fibroblast growth factor receptor, Phosphorylation and Bovine papillomavirus. As a part of the same scientific family, Elliot J. Androphy mostly works in the field of Molecular biology, focusing on Fibroblast growth factor receptor 3 and, on occasion, Fibroblast growth factor receptor 2.
His primary areas of investigation include Cell biology, Spinal muscular atrophy, Transcription, Viral replication and DNA replication. The various areas that Elliot J. Androphy examines in his Cell biology study include Cell culture and Gene knockdown. His Spinal muscular atrophy study focuses on SMN1 in particular.
As part of the same scientific family, Elliot J. Androphy usually focuses on Transcription, concentrating on Virology and intersecting with Bovine papillomavirus and Mutant. His studies deal with areas such as Tyrosine kinase, Molecular biology, Viral protein and Phosphorylation as well as Viral replication. His Molecular biology research is multidisciplinary, incorporating perspectives in DCPS, Regulation of gene expression, Messenger RNA and Transcriptome.
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A single nucleotide in the SMN gene regulates splicing and is responsible for spinal muscular atrophy
Christian L. Lorson;Eric Hahnen;Elliot J. Androphy;Brunhilde Wirth.
Proceedings of the National Academy of Sciences of the United States of America (1999)
A Single Nucleotide Difference That Alters Splicing Patterns Distinguishes the SMA Gene SMN1 From the Copy Gene SMN2
Umrao R. Monani;Christian L. Lorson;D. William Parsons;Thomas W. Prior.
Human Molecular Genetics (1999)
The Survival Motor Neuron Protein in Spinal Muscular Atrophy
Daniel D. Coovert;Thanh T. Le;Patricia E. McAndrew;John Strasswimmer.
Human Molecular Genetics (1997)
SMN oligomerization defect correlates with spinal muscular atrophy severity.
Christian L. Lorson;John Strasswimmer;Jun Mei Yao;James D. Baleja.
Nature Genetics (1998)
Targeting the E1 replication protein to the papillomavirus origin of replication by complex formation with the E2 transactivator.
Ian J. Mohr;Robin Clark;Shaw Sun;Elliot J. Androphy.
An exonic enhancer is required for inclusion of an essential exon in the SMA-determining gene SMN.
Christian L. Lorson;Elliot J. Androphy.
Human Molecular Genetics (2000)
Bovine papillomavirus E2 trans -activating gene product binds to specific sites in papillomavirus DNA
Elliot J. Androphy;Douglas R. Lowy;John T. Schiller.
Splicing of a Critical Exon of Human Survival Motor Neuron Is Regulated by a Unique Silencer Element Located in the Last Intron
Nirmal K. Singh;Natalia N. Singh;Elliot J. Androphy;Ravindra N. Singh.
Molecular and Cellular Biology (2006)
Interaction of papillomavirus E6 oncoproteins with a putative calcium-binding protein
Jason J. Chen;Carl E. Reid;Vimla Band;Elliot J. Androphy.
Htra2-β1 stimulates an exonic splicing enhancer and can restore full-length SMN expression to survival motor neuron 2 (SMN2)
Yvonne Hofmann;Christian L. Lorson;Stefan Stamm;Elliot J. Androphy.
Proceedings of the National Academy of Sciences of the United States of America (2000)
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