What is he best known for?
The fields of study he is best known for:
SMN1, Genetics, Spinal muscular atrophy, Molecular biology and Exon are his primary areas of study.
His SMN1 research is classified as research in Motor neuron.
As part of one scientific family, Elliot J. Androphy deals mainly with the area of Genetics, narrowing it down to issues related to the Cell biology, and often Transcription factor.
His work carried out in the field of Spinal muscular atrophy brings together such families of science as Exonic splicing silencer and SnRNP Biogenesis.
His study in Molecular biology is interdisciplinary in nature, drawing from both Messenger RNA, Mutant, Gene, Bovine papillomavirus and Tumor suppressor gene.
His RNA splicing research extends to the thematically linked field of Exon.
His most cited work include:
- A single nucleotide in the SMN gene regulates splicing and is responsible for spinal muscular atrophy (1116 citations)
- A Single Nucleotide Difference That Alters Splicing Patterns Distinguishes the SMA Gene SMN1 From the Copy Gene SMN2 (712 citations)
- The Survival Motor Neuron Protein in Spinal Muscular Atrophy (593 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Molecular biology, Cell biology, Spinal muscular atrophy, Transcription and SMN1.
His research in Molecular biology intersects with topics in Cell culture, Mutant, Gene, Repressor and Bovine papillomavirus.
His Cell biology study combines topics from a wide range of disciplines, such as Transcription factor, Viral replication and Gene knockdown.
His Spinal muscular atrophy research incorporates themes from Bioinformatics, Motor neuron and Alternative splicing, Exon.
To a larger extent, Elliot J. Androphy studies Genetics with the aim of understanding Exon.
Elliot J. Androphy works in the field of SMN1, focusing on Survival of motor neuron in particular.
He most often published in these fields:
- Molecular biology (41.52%)
- Cell biology (24.11%)
- Spinal muscular atrophy (24.55%)
What were the highlights of his more recent work (between 2016-2021)?
- Cell biology (24.11%)
- Spinal muscular atrophy (24.55%)
- Viral replication (11.61%)
In recent papers he was focusing on the following fields of study:
Elliot J. Androphy spends much of his time researching Cell biology, Spinal muscular atrophy, Viral replication, Transcription and Motor neuron.
Elliot J. Androphy is involved in the study of Spinal muscular atrophy that focuses on SMN1 in particular.
His research on SMN1 concerns the broader Exon.
His research in Exon focuses on subjects like Disease, which are connected to Genetics.
His Viral replication research includes themes of Mutant, Molecular biology, Fibroblast growth factor receptor, Phosphorylation and Bovine papillomavirus.
As a part of the same scientific family, Elliot J. Androphy mostly works in the field of Molecular biology, focusing on Fibroblast growth factor receptor 3 and, on occasion, Fibroblast growth factor receptor 2.
Between 2016 and 2021, his most popular works were:
- How the discovery of ISS-N1 led to the first medical therapy for spinal muscular atrophy. (72 citations)
- Metagenomic Discovery of 83 New Human Papillomavirus Types in Patients with Immunodeficiency. (35 citations)
- SMN deficiency negatively impacts red pulp macrophages and spleen development in mouse models of spinal muscular atrophy (15 citations)
In his most recent research, the most cited papers focused on:
His primary areas of investigation include Cell biology, Spinal muscular atrophy, Transcription, Viral replication and DNA replication.
The various areas that Elliot J. Androphy examines in his Cell biology study include Cell culture and Gene knockdown.
His Spinal muscular atrophy study focuses on SMN1 in particular.
As part of the same scientific family, Elliot J. Androphy usually focuses on Transcription, concentrating on Virology and intersecting with Bovine papillomavirus and Mutant.
His studies deal with areas such as Tyrosine kinase, Molecular biology, Viral protein and Phosphorylation as well as Viral replication.
His Molecular biology research is multidisciplinary, incorporating perspectives in DCPS, Regulation of gene expression, Messenger RNA and Transcriptome.
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