What is he best known for?
The fields of study he is best known for:
Molecular biology, Dystrophin, Duchenne muscular dystrophy, Cell biology and Spinal muscular atrophy are his primary areas of study.
His research integrates issues of Mutation, Gene, Emerin, Emery–Dreifuss muscular dystrophy and Gene product in his study of Molecular biology.
His work deals with themes such as Alternative splicing and Exon, which intersect with Dystrophin.
His Duchenne muscular dystrophy research includes elements of Messenger RNA, Immune system and Locus.
The Cell biology study combines topics in areas such as Genetics and Anatomy.
He has included themes like SMN complex, snRNP, Motor neuron and Coilin in his Spinal muscular atrophy study.
His most cited work include:
- The human centromeric survival motor neuron gene (SMN2) rescues embryonic lethality in Smn–/– mice and results in a mouse with spinal muscular atrophy (615 citations)
- Functional amounts of dystrophin produced by skipping the mutated exon in the mdx dystrophic mouse. (377 citations)
- The Emery-Dreifuss Muscular Dystrophy Protein, Emerin, is a Nuclear Membrane Protein (325 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Molecular biology, Dystrophin, Cell biology, Monoclonal antibody and Muscular dystrophy.
His research on Molecular biology also deals with topics like
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Epitope that intertwine with fields like Amino acid,
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Epitope mapping which is related to area like Binding site.
His Dystrophin study also includes
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Exon, which have a strong connection to RNA splicing,
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Pathology which intersects with area such as Anatomy.
His Cell biology research is multidisciplinary, incorporating perspectives in Genetics, Spinal muscular atrophy and snRNP.
His Muscular dystrophy research incorporates elements of Emerin and Skeletal muscle.
Glenn E. Morris interconnects Emery–Dreifuss muscular dystrophy and Mutation in the investigation of issues within Emerin.
He most often published in these fields:
- Molecular biology (47.03%)
- Dystrophin (26.73%)
- Cell biology (25.25%)
What were the highlights of his more recent work (between 2012-2020)?
- Molecular biology (47.03%)
- Monoclonal antibody (20.79%)
- Cell biology (25.25%)
In recent papers he was focusing on the following fields of study:
Glenn E. Morris mostly deals with Molecular biology, Monoclonal antibody, Cell biology, Gene isoform and Spinal muscular atrophy.
The concepts of his Molecular biology study are interwoven with issues in Alternative splicing, Blot, Muscular dystrophy, Protein kinase C and Epitope.
His Monoclonal antibody study combines topics in areas such as Sarcolemma, Transmembrane protein, Dystrophin and Virology.
His research on Dystrophin concerns the broader Duchenne muscular dystrophy.
His studies deal with areas such as Exon and Nesprin as well as Gene isoform.
Glenn E. Morris combines subjects such as Neuromuscular disease, Motor neuron and Ubiquitin with his study of Spinal muscular atrophy.
Between 2012 and 2020, his most popular works were:
- Dysregulation of ubiquitin homeostasis and β-catenin signaling promote spinal muscular atrophy (132 citations)
- Specific nuclear envelope transmembrane proteins can promote the location of chromosomes to and from the nuclear periphery (90 citations)
- Nesprins: Tissue-Specific Expression of Epsilon and Other Short Isoforms (53 citations)
In his most recent research, the most cited papers focused on:
His primary areas of study are Cell biology, Molecular biology, Motor neuron, Spinal muscular atrophy and Ubiquitin.
His Cell biology research includes themes of Genetics, Actin cytoskeleton and Alternative splicing, Gene isoform.
Glenn E. Morris performs multidisciplinary study in Molecular biology and MSH3 in his work.
His work carried out in the field of Motor neuron brings together such families of science as Neuromuscular disease, Proteomics, Neuroscience and Induced pluripotent stem cell.
The study incorporates disciplines such as Neuromuscular junction and Zebrafish in addition to Spinal muscular atrophy.
His work focuses on many connections between Cell nucleus and other disciplines, such as Transmembrane protein, that overlap with his field of interest in Lamin.
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