2019 - Fellow of the American Academy of Arts and Sciences
2008 - Fellow of the American Association for the Advancement of Science (AAAS)
Denise A. Galloway mainly investigates Virology, Molecular biology, Immunology, HPV infection and Cancer research. Her studies deal with areas such as Antigen and Merkel cell polyomavirus as well as Virology. Denise A. Galloway interconnects DNA damage, Cell growth, Replication factor C, Control of chromosome duplication and Origin recognition complex in the investigation of issues within Molecular biology.
In general Immunology study, her work on Antibody often relates to the realm of Polyomavirus Infections, thereby connecting several areas of interest. Denise A. Galloway has included themes like Physiology, Gynecology, Oncology and Cervix in her HPV infection study. Her Cancer research research includes elements of Tumor suppressor gene, Cell cycle, Kinase and CpG site.
The scientist’s investigation covers issues in Virology, Immunology, Molecular biology, Antibody and Internal medicine. Her studies in Virology integrate themes in fields like DNA and Monoclonal antibody. Her research in Immunology intersects with topics in Young adult and Cervical cancer, HPV infection.
As a member of one scientific family, Denise A. Galloway mostly works in the field of Molecular biology, focusing on Telomerase reverse transcriptase and, on occasion, Cell biology and Protein subunit. The Antibody study combines topics in areas such as Viral disease, Immune system and Antigen. Her Internal medicine research incorporates elements of Gynecology and Oncology.
Her primary areas of investigation include Immunology, Internal medicine, Virology, Oncology and Young adult. Her Immunology research is multidisciplinary, incorporating elements of Cervical cancer, HPV infection and Cohort. Her research investigates the connection with Internal medicine and areas like Genotype which intersect with concerns in Head and neck squamous-cell carcinoma.
Her work on Virus as part of general Virology study is frequently connected to Polyomavirus Infections, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them. The study incorporates disciplines such as Gynecology and Cervical intraepithelial neoplasia in addition to Oncology. Her Young adult study also includes fields such as
Her primary scientific interests are in Immunology, DNA damage, Virology, Carcinogenesis and Genetics. The concepts of her Immunology study are interwoven with issues in Internal medicine, Oncology, Merkel cell polyomavirus and Skin cancer. Her DNA damage study incorporates themes from Molecular biology, Beta and Cell cycle checkpoint.
Her Molecular biology research integrates issues from Pyrimidine dimer and Ataxia Telangiectasia Mutated Proteins. Denise A. Galloway combines subjects such as Antibody, B cell and Human papillomavirus with her study of Virology. Her work deals with themes such as Virus, Innate immune system, Disease and HPV infection, which intersect with Human papillomavirus.
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Both Rb/p16INK4a inactivation and telomerase activity are required to immortalize human epithelial cells
Tohru Kiyono;Scott A. Foster;Jenn I. Koop;James K. McDougall.
A cohort study of the risk of cervical intraepithelial neoplasia grade 2 or 3 in relation to papillomavirus infection.
LA Koutsky;KK Holmes;CW Critchlow;CE Stevens.
The New England Journal of Medicine (1992)
Loss of normal p53 function confers sensitization to Taxol by increasing G2/M arrest and apoptosis
A F Wahl;K L Donaldson;C Fairchild;F Y Lee.
Nature Medicine (1996)
EPIDEMIOLOGY OF GENITAL HUMAN PAPILLOMAVIRUS INFECTION
Laura A. Koutsky;Denise A. Galloway;King K. Holmes.
Epidemiologic Reviews (1988)
The E7 gene of human papillomavirus type 16 is sufficient for immortalization of human epithelial cells.
C L Halbert;G W Demers;D A Galloway.
Journal of Virology (1991)
Comparison of human papillomavirus types 16, 18, and 6 capsid antibody responses following incident infection.
Joseph J. Carter;Laura A. Koutsky;James P. Hughes;Shu Kuang Lee.
The Journal of Infectious Diseases (2000)
Oral Cancer Risk in Relation to Sexual History and Evidence of Human Papillomavirus Infection
Stephen M. Schwartz;Janet R. Daling;Margaret M. Madeleine;David R. Doody.
Journal of the National Cancer Institute (1998)
Non-transcriptional control of DNA replication by c-Myc
David Dominguez-Sola;Carol Y. Ying;Carla Grandori;Carla Grandori;Luca Ruggiero.
Inhibition of CDK activity and PCNA-dependent DNA replication by p21 is blocked by interaction with the HPV-16 E7 oncoprotein
Jens Oliver Funk;Shou Waga;Jo Beth Harry;Erik Espling.
Genes & Development (1997)
c-Myc binds to human ribosomal DNA and stimulates transcription of rRNA genes by RNA polymerase I.
Carla Grandori;Natividad Gomez-Roman;Zoe A. Felton-Edkins;Celine Ngouenet.
Nature Cell Biology (2005)
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