D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Molecular Biology D-index 51 Citations 7,301 136 World Ranking 1757 National Ranking 890

Research.com Recognitions

Awards & Achievements

2015 - Fellow of the American Association for the Advancement of Science (AAAS)

Overview

What is she best known for?

The fields of study she is best known for:

  • Gene
  • DNA
  • Gene expression

Michelle Craig Barton mainly investigates Molecular biology, Cancer research, Psychological repression, Histone and Chromatin. Her study in Molecular biology is interdisciplinary in nature, drawing from both Transcription, TRIM24 and Promoter, RNA polymerase II, Transcription factor II D. Her studies in Cancer research integrate themes in fields like Cancer cell, Histone methyltransferase and Bladder cancer.

Michelle Craig Barton works mostly in the field of Psychological repression, limiting it down to topics relating to Transcription factor and, in certain cases, Derepression, as a part of the same area of interest. Her Histone research includes themes of Regulation of gene expression, Epigenetics, Pharmacology and Cell biology. Chromatin immunoprecipitation is closely connected to Transcriptional regulation in her research, which is encompassed under the umbrella topic of Chromatin.

Her most cited work include:

  • TRIM24 links a non-canonical histone signature to breast cancer (276 citations)
  • Kaiso/p120-Catenin and TCF/β-Catenin Complexes Coordinately Regulate Canonical Wnt Gene Targets (203 citations)
  • Kaiso/p120-Catenin and TCF/β-Catenin Complexes Coordinately Regulate Canonical Wnt Gene Targets (203 citations)

What are the main themes of her work throughout her whole career to date?

Her primary areas of investigation include Cell biology, Chromatin, Cancer research, Molecular biology and Histone. She has included themes like Cell cycle checkpoint, Genetics, Embryonic stem cell, Induced pluripotent stem cell and Xenopus in her Cell biology study. Her Chromatin research includes elements of Chromatin immunoprecipitation, Epigenetics and Cellular differentiation.

Her research in Cancer research intersects with topics in TRIM24, Cancer cell, Transcription factor, Signal transduction and Regulation of gene expression. Her biological study spans a wide range of topics, including Transcription, Psychological repression, Gene expression, General transcription factor and DNA replication. Her work deals with themes such as Gene silencing and Acetylation, which intersect with Histone.

She most often published in these fields:

  • Cell biology (38.19%)
  • Chromatin (36.11%)
  • Cancer research (31.94%)

What were the highlights of her more recent work (between 2013-2021)?

  • Cancer research (31.94%)
  • Cell biology (38.19%)
  • Chromatin (36.11%)

In recent papers she was focusing on the following fields of study:

Michelle Craig Barton mainly investigates Cancer research, Cell biology, Chromatin, Histone and Epigenetics. Michelle Craig Barton combines subjects such as TRIM24, Cancer cell, Transcription factor, Breast cancer and Androgen receptor with her study of Cancer research. She has researched Cell biology in several fields, including Embryonic stem cell, Histone H3, Cellular differentiation and Cytosol.

Her Chromatin study combines topics from a wide range of disciplines, such as Molecular biology, Chromatin immunoprecipitation and Mdm2. Her Histone research incorporates themes from Cell cycle and Acetylation. As a part of the same scientific study, Michelle Craig Barton usually deals with the Epigenetics, concentrating on Gene silencing and frequently concerns with Regulator, Endogeny and Death-associated protein 6.

Between 2013 and 2021, her most popular works were:

  • ZMYND11 links histone H3.3K36me3 to transcription elongation and tumour suppression (186 citations)
  • TET1 is a maintenance DNA demethylase that prevents methylation spreading in differentiated cells (84 citations)
  • LncPRESS1 Is a p53-Regulated LncRNA that Safeguards Pluripotency by Disrupting SIRT6-Mediated De-acetylation of Histone H3K56 (79 citations)

In her most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Gene expression

Michelle Craig Barton mainly focuses on Cancer research, Chromatin, Histone, Cellular differentiation and Regulation of gene expression. Her Cancer research research is multidisciplinary, incorporating elements of Carcinogenesis, Epigenomics, Epigenetics and Apoptosis. Her Chromatin study incorporates themes from Transcription, Acetylation and DNA damage.

Her study with Histone involves better knowledge in Genetics. Her research integrates issues of Reprogramming and Cell biology in her study of Cellular differentiation. Her work carried out in the field of Regulation of gene expression brings together such families of science as Transcription factor and Gene expression.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

TRIM24 links a non-canonical histone signature to breast cancer

Wen Wei Tsai;Zhanxin Wang;Teresa T. Yiu;Teresa T. Yiu;Kadir C. Akdemir.
Nature (2010)

403 Citations

Kaiso/p120-Catenin and TCF/β-Catenin Complexes Coordinately Regulate Canonical Wnt Gene Targets

Jae Il Park;Jae Il Park;Si Wan Kim;Jon P. Lyons;Jon P. Lyons;Hong Ji.
Developmental Cell (2005)

304 Citations

ZMYND11 links histone H3.3K36me3 to transcription elongation and tumour suppression

Hong Wen;Yuanyuan Li;Yuanxin Xi;Shiming Jiang.
Nature (2014)

280 Citations

Hypoxia induces a novel signature of chromatin modifications and global repression of transcription.

Amber Buescher Johnson;Nicholas Denko;Michelle Craig Barton.
Mutation Research (2008)

278 Citations

Trim24 targets endogenous p53 for degradation

Kendra Allton;Abhinav K. Jain;Hans-Martin Herz;Wen-Wei Tsai;Wen-Wei Tsai.
Proceedings of the National Academy of Sciences of the United States of America (2009)

266 Citations

p53 Regulates Cell Cycle and MicroRNAs to Promote Differentiation of Human Embryonic Stem Cells

Abhinav K. Jain;Kendra Allton;Michelina Iacovino;Elisabeth Mahen.
PLOS Biology (2012)

235 Citations

p53-Mediated repression of alpha-fetoprotein gene expression by specific DNA binding

Kathleen C. Lee;Alison J. Crowe;Michelle Craig Barton.
Molecular and Cellular Biology (1999)

226 Citations

3-Hydroxy-3-methylglutaryl-coenzyme A reductase is present in peroxisomes in normal rat liver cells

Gilbert-A. Keller;Michelle C. Barton;David J. Shapiro;S. J. Singer.
Proceedings of the National Academy of Sciences of the United States of America (1985)

220 Citations

UV-induced inhibition of transcription involves repression of transcription initiation and phosphorylation of RNA polymerase II

Davy A. P. Rockx;Rebecca Mason;Rebecca Mason;Anneke van Hoffen;Michelle Craig Barton.
Proceedings of the National Academy of Sciences of the United States of America (2000)

204 Citations

The p63 Protein Isoform ΔNp63α Inhibits Epithelial-Mesenchymal Transition in Human Bladder Cancer Cells ROLE OF MIR-205

Mai N. Tran;Woonyoung Choi;Matthew F. Wszolek;Neema Navai.
Journal of Biological Chemistry (2013)

152 Citations

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