Markus Löbrich integrates many fields in his works, including DNA and Histone. Markus Löbrich integrates many fields in his works, including Histone and DNA. He incorporates Genetics and Computational biology in his studies. He undertakes multidisciplinary investigations into Computational biology and Genetics in his work. In his works, he conducts interdisciplinary research on Cell biology and Gene. He brings together Gene and Cell biology to produce work in his papers. He regularly ties together related areas like Double strand in his DNA repair studies. Many of his studies involve connections with topics such as Double strand and DNA damage. Markus Löbrich combines Molecular biology and Biochemistry in his research.
Markus Löbrich integrates Genetics and Cancer research in his studies. His work often combines Cancer research and Genetics studies. In his papers, he integrates diverse fields, such as DNA repair and Genome instability. His DNA damage study frequently links to related topics such as Genome instability. In his works, Markus Löbrich conducts interdisciplinary research on Cell biology and Molecular biology. He brings together Molecular biology and Gene to produce work in his papers. He integrates Gene and Cell biology in his studies. Markus Löbrich incorporates Homologous recombination and Non-homologous end joining in his research. In his work, he performs multidisciplinary research in Non-homologous end joining and Homologous recombination.
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Evidence for a lack of DNA double-strand break repair in human cells exposed to very low x-ray doses
Kai Rothkamm;Markus Löbrich.
Proceedings of the National Academy of Sciences of the United States of America (2003)
Pathways of DNA double-strand break repair during the mammalian cell cycle.
Kai Rothkamm;Ines Krüger;Larry H. Thompson;Markus Löbrich.
Molecular and Cellular Biology (2003)
ATM and DNA-PK function redundantly to phosphorylate H2AX after exposure to ionizing radiation.
Tom Stiff;Mark O’Driscoll;Nicole Rief;Kuniyoshi Iwabuchi.
Cancer Research (2004)
A pathway of double-strand break rejoining dependent upon ATM, Artemis, and proteins locating to gamma-H2AX foci.
Enriqueta Riballo;Martin Kühne;Nicole Rief;Aidan Doherty.
Molecular Cell (2004)
ATM signaling facilitates repair of DNA double-strand breaks associated with heterochromatin.
Aaron A. Goodarzi;Angela T. Noon;Dorothee Deckbar;Yael Ziv.
Molecular Cell (2008)
The impact of a negligent G2/M checkpoint on genomic instability and cancer induction.
Markus Löbrich;Penny A. Jeggo.
Nature Reviews Cancer (2007)
γH2AX foci analysis for monitoring DNA double-strand break repair: strengths, limitations and optimization
Markus Löbrich;Atsushi Shibata;Andrea Beucher;Anna Fisher.
Cell Cycle (2010)
ATM and Artemis promote homologous recombination of radiation-induced DNA double-strand breaks in G2.
Andrea Beucher;Julie Birraux;Leopoldine Tchouandong;Olivia Barton.
The EMBO Journal (2009)
In vivo formation and repair of DNA double-strand breaks after computed tomography examinations
Markus Löbrich;Nicole Rief;Martin Kühne;Martina Heckmann.
Proceedings of the National Academy of Sciences of the United States of America (2005)
Factors determining DNA double-strand break repair pathway choice in G2 phase.
Atsushi Shibata;Sandro Conrad;Julie Birraux;Verena Geuting.
The EMBO Journal (2011)
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