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Medicine

D-Index
94
Citations
29192
World Ranking
10568
National Ranking
320

Overview

David G. Allen is affiliated with the University of Sydney in Australia. Their research primarily spans the fields of Medicine and Biochemistry, Genetics and Molecular Biology, with a strong focus on Cardiology and Cardiovascular Medicine, Molecular Biology, Physiology, Cell Biology, and Pulmonary and Respiratory Medicine.

The scientist's research topics include:

  • Cardiac electrophysiology and arrhythmias
  • Ion channel regulation and function
  • Cardiovascular effects of exercise
  • Muscle metabolism and nutrition
  • Adipose tissue and metabolism
  • Diet and metabolism studies
  • Renal cell carcinoma treatment

Frequent co-authors of David G. Allen include:

  • Santiago Pineda
  • Vesna Nikolova-Krstevski
  • Christiana Leimena
  • Andrew Atkinson
  • Ann-Kristin Alteköester

Their recent papers demonstrate contributions to multiple areas, including cardiac ion channel function and cancer treatment. Notable publications are:

  • "Conserved Role of the Large Conductance Calcium-Activated Potassium Channel, K Ca 1.1, in Sinus Node Function and Arrhythmia Risk," 2021, Circulation Genomic and Precision Medicine
  • "The spatial distribution of glycogen and glycogen consumption in muscle cells," 2022, The Journal of General Physiology
  • "Conserved Role of the Large Conductance Calcium-Activated Potassium Channel, KCa1.1, in Sinus Node Function and Arrhythmia Risk," 2020, bioRxiv (Cold Spring Harbor Laboratory)
  • "Abstract No. 225 External Validation of the Ability of the mRENAL Nephrometry Score to Identify Patients at Risk for Major Adverse Events or Local Tumor Recurrence After Percutaneous Renal Cryoablation," 2024, Journal of Vascular and Interventional Radiology
  • "A circle RNA regulatory axis promotes lung squamous metastasis via CDR1-mediated regulation of golgi trafficking," 2024, UNC Libraries

Publication venues where their work frequently appears include:

  • Circulation Genomic and Precision Medicine
  • The Journal of General Physiology
  • bioRxiv (Cold Spring Harbor Laboratory)
  • Journal of Vascular and Interventional Radiology
  • UNC Libraries

Best Publications

  • Skeletal Muscle Fatigue: Cellular Mechanisms

    D. G. Allen;G. D. Lamb;H. Westerblad

  • Muscle Fatigue: Lactic Acid or Inorganic Phosphate the Major Cause?

    Håkan Westerblad;David G. Allen;Jan Lännergren

  • The effects of muscle length on intracellular calcium transients in mammalian cardiac muscle.

    D G Allen;S Kurihara

  • The cellular basis of the length-tension relation in cardiac muscle

    D.G. Allen;J.C. Kentish

  • Myocardial contractile function during ischemia and hypoxia.

    D G Allen;C H Orchard

  • Calcium transients in aequorin-injected frog cardiac muscle.

    David G. Allen;David G. Allen;John R. Blinks

  • Skeletal muscle hypertrophy is mediated by a Ca2+-dependent calcineurin signalling pathway.

    Christopher Semsarian;Ming-Jie Wu;Yue-Kun Ju;Tadeusz Marciniec

  • Cellular mechanisms of fatigue in skeletal muscle.

    H. Westerblad;J. A. Lee;J. Lannergren;D. G. Allen

  • Effect of hydrogen peroxide and dithiothreitol on contractile function of single skeletal muscle fibres from the mouse.

    Francisco H. Andrade;Francisco H. Andrade;Michael B. Reid;David G. Allen;Håkan Westerblad

  • Early events in stretch-induced muscle damage.

    David L Morgan;D G Allen

  • MUSCLE CELL FUNCTION DURING PROLONGED ACTIVITY: CELLULAR MECHANISMS OF FATIGUE

    DG Allen;J Lannergren;H Westerblad

  • Changes of myoplasmic calcium concentration during fatigue in single mouse muscle fibers.

    H Westerblad;D G Allen

  • Role of phosphate and calcium stores in muscle fatigue.

    D. G. Allen;H. Westerblad

  • Eccentric muscle damage: mechanisms of early reduction of force

    D. G. Allen

  • Fibroblasts Can Be Genetically Modified to Produce Excitable Cells Capable of Electrical Coupling

    Eddy Kizana;Samantha L. Ginn;David G. Allen;David L. Ross

  • Absence of Dystrophin Disrupts Skeletal Muscle Signaling: Roles of Ca2+, Reactive Oxygen Species, and Nitric Oxide in the Development of Muscular Dystrophy

    David G. Allen;Nicholas P. Whitehead;Stanley C. Froehner

  • Muscle damage in mdx (dystrophic) mice: role of calcium and reactive oxygen species.

    Nicholas P Whitehead;Ella W Yeung;David G Allen

  • Intracellular calcium concentration during low-frequency fatigue in isolated single fibers of mouse skeletal muscle

    H. Westerblad;S. Duty;D. G. Allen

  • A nuclear magnetic resonance study of metabolism in the ferret heart during hypoxia and inhibition of glycolysis.

    D G Allen;P G Morris;C H Orchard;J S Pirolo

  • Impaired calcium release during fatigue

    David G. Allen;Graham D. Lamb;Hakan Westerblad

Frequent Co-Authors

Håkan Westerblad
Håkan Westerblad Karolinska Institute
David A. Eisner
David A. Eisner University of Manchester
Robert M. Graham
Robert M. Graham Victor Chang Cardiac Research Institute
John Aldo Lee
John Aldo Lee Université Catholique de Louvain
Mark B. Cannell
Mark B. Cannell University of Bristol
Richard P. Harvey
Richard P. Harvey Victor Chang Cardiac Research Institute
Ian E. Alexander
Ian E. Alexander University of Sydney
Christopher Semsarian
Christopher Semsarian University of Sydney
Godfrey L. Smith
Godfrey L. Smith University of Glasgow
Peter C. M. Molenaar
Peter C. M. Molenaar Pennsylvania State University

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