What is he best known for?
The fields of study he is best known for:
- Internal medicine
- Biochemistry
- Endocrinology
David G. Allen mainly investigates Calcium, Internal medicine, Biophysics, Calcium metabolism and Endocrinology.
The various areas that he examines in his Calcium study include Endoplasmic reticulum, Biochemistry, Myofibril and Muscle contraction, Anatomy.
His biological study focuses on Skeletal muscle.
David G. Allen studied Skeletal muscle and Muscular dystrophy that intersect with Duchenne muscular dystrophy and Cell biology.
His study in Biophysics is interdisciplinary in nature, drawing from both Isometric exercise, Calcium in biology, Intracellular, Aequorin and Contraction.
His Calcium in biology research is multidisciplinary, incorporating perspectives in Cardiac muscle and Myogenesis.
His most cited work include:
- Skeletal Muscle Fatigue: Cellular Mechanisms (1508 citations)
- The cellular basis of the length-tension relation in cardiac muscle (485 citations)
- The effects of muscle length on intracellular calcium transients in mammalian cardiac muscle. (469 citations)
What are the main themes of his work throughout his whole career to date?
His primary scientific interests are in Internal medicine, Endocrinology, Calcium, Biophysics and Skeletal muscle.
His Internal medicine study frequently draws connections to other fields, such as Cardiology.
In his research, Muscular dystrophy is intimately related to Duchenne muscular dystrophy, which falls under the overarching field of Endocrinology.
David G. Allen combines subjects such as Endoplasmic reticulum and Muscle contraction with his study of Calcium.
His research on Biophysics also deals with topics like
- Biochemistry that intertwine with fields like Fluorescence spectrometry,
- Anatomy and related Sarcomere.
His work carried out in the field of Skeletal muscle brings together such families of science as Reactive oxygen species, Cell biology and Myofibril.
He most often published in these fields:
- Internal medicine (41.95%)
- Endocrinology (36.02%)
- Calcium (31.36%)
What were the highlights of his more recent work (between 2006-2021)?
- Internal medicine (41.95%)
- Endocrinology (36.02%)
- Cell biology (13.14%)
In recent papers he was focusing on the following fields of study:
David G. Allen mainly focuses on Internal medicine, Endocrinology, Cell biology, Skeletal muscle and Intracellular.
His Endocrinology research is multidisciplinary, relying on both Endoplasmic reticulum, TRPC1, Duchenne muscular dystrophy and Calcium.
David G. Allen studies Calcium in biology, a branch of Calcium.
In the field of Cell biology, his study on Function and Cell signaling overlaps with subjects such as Sinoatrial node and Scavenger receptor.
His Skeletal muscle research incorporates themes from Biophysics and Biochemistry.
He interconnects Extracellular, Ischemia, Rat heart and Amiloride in the investigation of issues within Intracellular.
Between 2006 and 2021, his most popular works were:
- Skeletal Muscle Fatigue: Cellular Mechanisms (1508 citations)
- Impaired calcium release during fatigue (189 citations)
- Absence of Dystrophin Disrupts Skeletal Muscle Signaling: Roles of Ca2+, Reactive Oxygen Species, and Nitric Oxide in the Development of Muscular Dystrophy (177 citations)
In his most recent research, the most cited papers focused on:
- Internal medicine
- Biochemistry
- Endocrinology
His main research concerns Internal medicine, Endocrinology, Biochemistry, Cell biology and Dystrophin.
When carried out as part of a general Internal medicine research project, his work on Mean age, Endometrium and Neuroendocrine carcinoma is frequently linked to work in Large cell, therefore connecting diverse disciplines of study.
His Endocrinology study incorporates themes from TRPC1, Calcium and Anatomy.
He is interested in Calcium metabolism, which is a field of Calcium.
His study looks at the intersection of Biochemistry and topics like Skeletal muscle with Membrane permeability.
His Calcium in biology research extends to Dystrophin, which is thematically connected.
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