D-Index & Metrics Best Publications
Aristidis Moustakas

Aristidis Moustakas

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Molecular Biology D-index 70 Citations 22,578 170 World Ranking 865 National Ranking 16

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Transcription factor

Aristidis Moustakas focuses on SMAD, Transforming growth factor beta, Cell biology, Transcription factor and Signal transduction. His work deals with themes such as TGF beta signaling pathway, Cancer research and Kinase, which intersect with SMAD. His biological study spans a wide range of topics, including Epithelial–mesenchymal transition and Immunology.

His work in Transforming growth factor beta covers topics such as Cellular differentiation which are related to areas like Bone morphogenetic protein. His studies examine the connections between Cell biology and genetics, as well as such issues in Ubiquitin ligase, with regards to Tissue homeostasis. Aristidis Moustakas interconnects Platelet-derived growth factor receptor, Phosphorylation, Regulation of gene expression, Adherens junction and Mesenchymal–epithelial transition in the investigation of issues within Signal transduction.

His most cited work include:

  • Non-Smad TGF-β signals (875 citations)
  • Smad regulation in TGF-beta signal transduction. (826 citations)
  • The regulation of TGFβ signal transduction (645 citations)

What are the main themes of his work throughout his whole career to date?

Cell biology, SMAD, Transforming growth factor, Signal transduction and Cancer research are his primary areas of study. He is interested in Transforming growth factor beta, which is a branch of Cell biology. His studies deal with areas such as TGF beta signaling pathway, Molecular biology, Transcription factor and Kinase as well as SMAD.

As part of the same scientific family, he usually focuses on Transforming growth factor, concentrating on Stromal cell and intersecting with Extracellular matrix. His Signal transduction study incorporates themes from R-SMAD, Downregulation and upregulation and Protein kinase A, Phosphorylation. His Cancer research research is multidisciplinary, relying on both Epithelial–mesenchymal transition, Cancer, Metastasis, Cell growth and Immunology.

He most often published in these fields:

  • Cell biology (78.28%)
  • SMAD (53.54%)
  • Transforming growth factor (54.55%)

What were the highlights of his more recent work (between 2015-2021)?

  • Cell biology (78.28%)
  • Transforming growth factor (54.55%)
  • Cancer research (47.47%)

In recent papers he was focusing on the following fields of study:

The scientist’s investigation covers issues in Cell biology, Transforming growth factor, Cancer research, Signal transduction and SMAD. His Cell biology study integrates concerns from other disciplines, such as Epithelial–mesenchymal transition and Bone morphogenetic protein, Gene, Cellular differentiation, Transcription factor. His Transforming growth factor study combines topics in areas such as Autocrine signalling, Extracellular, Crosstalk, Molecular biology and Gene isoform.

His work carried out in the field of Cancer research brings together such families of science as Carcinogenesis, Fibroblast, Metastasis and Immunology. Aristidis Moustakas has included themes like Cell surface receptor and Recombinant DNA in his Signal transduction study. His SMAD research integrates issues from Cell signaling, Tissue homeostasis, Kinase and WNT3A.

Between 2015 and 2021, his most popular works were:

  • Signaling Receptors for TGF-β Family Members (232 citations)
  • Signaling Receptors for TGF-β Family Members (232 citations)
  • Mechanisms of TGFβ-Induced Epithelial–Mesenchymal Transition (128 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • DNA

His primary areas of investigation include Cell biology, Transforming growth factor, Epithelial–mesenchymal transition, Cancer research and Signal transduction. He is interested in SMAD, which is a field of Cell biology. His research investigates the connection between Transforming growth factor and topics such as Crosstalk that intersect with issues in HCCS.

His study in Epithelial–mesenchymal transition is interdisciplinary in nature, drawing from both Serglycin, PI3K/AKT/mTOR pathway, Cellular differentiation and Pathology. Aristidis Moustakas combines subjects such as Extracellular matrix, Metastasis and Immunology with his study of Cancer research. In general Signal transduction, his work in Transforming growth factor beta is often linked to Janus kinase 1 and Cyclin-dependent kinase 9 linking many areas of study.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Non-Smad TGF-β signals

Aristidis Moustakas;Carl-Henrik Heldin.
Journal of Cell Science (2005)

1452 Citations

Smad regulation in TGF-beta signal transduction.

Aristidis Moustakas;Serhiy Souchelnytskyi;Carl-Henrik Heldin.
Journal of Cell Science (2001)

1357 Citations

The regulation of TGFβ signal transduction

Aristidis Moustakas;Carl-Henrik Heldin.
Development (2009)

1085 Citations

Signaling networks guiding epithelial–mesenchymal transitions during embryogenesis and cancer progression

Aristidis Moustakas;Carl Henrik Heldin.
Cancer Science (2007)

1016 Citations

Mechanism of TGF-beta signaling to growth arrest, apoptosis, and epithelial-mesenchymal transition.

Carl-Henrik Heldin;Maréne Landström;Aristidis Moustakas.
Current Opinion in Cell Biology (2009)

829 Citations

TGF-β and the Smad Signaling Pathway Support Transcriptomic Reprogramming during Epithelial-Mesenchymal Cell Transition

Ulrich Valcourt;Marcin Kowanetz;Hideki Niimi;Carl-Henrik Heldin.
Molecular Biology of the Cell (2005)

752 Citations

Actions of TGF-beta as tumor suppressor and pro-metastatic factor in human cancer.

Katerina Pardali;Aristidis Moustakas.
Biochimica et Biophysica Acta (2007)

736 Citations

A SNAIL1-SMAD3/4 transcriptional repressor complex promotes TGF-beta mediated epithelial-mesenchymal transition.

Theresa Vincent;Theresa Vincent;Etienne P. A. Neve;Etienne P. A. Neve;Jill R. Johnson;Alexander Kukalev.
Nature Cell Biology (2009)

695 Citations

Mechanisms of TGF-beta signaling in regulation of cell growth and differentiation.

Aristidis Moustakas;Katerina Pardali;Annamaria Gaal;Carl Henrik Heldin.
Immunology Letters (2002)

686 Citations

Transforming growth factor-β employs HMGA2 to elicit epithelial–mesenchymal transition

Sylvie Thuault;Ulrich Valcourt;Maj Petersen;Guidalberto Manfioletti.
Journal of Cell Biology (2006)

566 Citations

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