Cell biology, Signal transduction, SMAD, Transforming growth factor and Transcription factor are her primary areas of study. The various areas that Caroline S. Hill examines in her Cell biology study include Cell cycle and Transcription. Caroline S. Hill has researched Signal transduction in several fields, including TGF beta signaling pathway, ACVR2B, Cancer research and Gene expression.
Her studies deal with areas such as R-SMAD, Response element and MAPK/ERK pathway as well as SMAD. Her work deals with themes such as SUPERFAMILY, Receptor, Extracellular signal and Signalling, which intersect with Transforming growth factor. Her research in Transcription factor intersects with topics in Extracellular and Cell membrane.
Caroline S. Hill mainly focuses on Cell biology, SMAD, Transforming growth factor, Signal transduction and Molecular biology. She has included themes like Receptor, Transcription factor and R-SMAD in her Cell biology study. The study incorporates disciplines such as Epithelial–mesenchymal transition, Xenopus and Peptide sequence in addition to SMAD.
Her Transforming growth factor research includes elements of SUPERFAMILY, Cell culture, Transcription and Signalling. Her Signal transduction study combines topics from a wide range of disciplines, such as Extracellular, Cancer research and Autocrine signalling. Her work deals with themes such as DNA, Regulation of gene expression, Bone morphogenetic protein and Mesoderm, which intersect with Molecular biology.
Her primary scientific interests are in Cell biology, Transforming growth factor, Phosphorylation, Receptor and SMAD. She studies Cell biology, focusing on Nodal signaling in particular. Her Transforming growth factor research incorporates elements of Carcinogenesis, Cancer research, Signal transduction and Downregulation and upregulation.
Her Signal transduction research includes elements of Extracellular, Signalling and In vivo. Her ACVR1, ACVR2A and Receptor clustering study in the realm of Receptor interacts with subjects such as Fibrodysplasia ossificans progressiva. Her SMAD study integrates concerns from other disciplines, such as Transcription, Kinase, Cell signaling and Gene isoform.
The scientist’s investigation covers issues in Cell biology, Nodal signaling, NODAL, Transforming growth factor and Signal transduction. She works on Cell biology which deals in particular with Lefty. Her Lefty research is multidisciplinary, incorporating elements of Signalling and Cell fate determination.
Her Nodal signaling research includes themes of Chromatin, Transcription factor, Chromatin binding and Activin type 2 receptors. As a part of the same scientific family, Caroline S. Hill mostly works in the field of Epithelial–mesenchymal transition, focusing on Cell and, on occasion, SMAD. Her Morphogen study combines topics in areas such as Nodal Signaling Ligands, Endoderm, Mesoderm and Fibroblast growth factor.
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The Rho family GTPases RhoA, Racl , and CDC42Hsregulate transcriptional activation by SRF
Caroline S. Hill;Judy Wynne;Richard Treisman.
SB-431542 is a potent and specific inhibitor of transforming growth factor-beta superfamily type I activin receptor-like kinase (ALK) receptors ALK4, ALK5, and ALK7.
Gareth J. Inman;Francisco J. Nicolás;James F. Callahan;John D. Harling.
Molecular Pharmacology (2002)
Transcriptional Regulation by Extracellular Signals: Mechanisms and Specificity
Caroline S Hill;Richard Treisman.
New insights into TGF-beta-Smad signalling.
Peter ten Dijke;Caroline S Hill.
Trends in Biochemical Sciences (2004)
TGFβ–SMAD signal transduction: molecular specificity and functional flexibility
Bernhard Schmierer;Caroline S. Hill.
Nature Reviews Molecular Cell Biology (2007)
TGF-β Superfamily Signaling in Embryonic Development and Homeostasis
Mary Y. Wu;Caroline S. Hill.
Developmental Cell (2009)
Localized and reversible TGFbeta signalling switches breast cancer cells from cohesive to single cell motility.
Silvia Giampieri;Cerys Manning;Steven Hooper;Louise Jones.
Nature Cell Biology (2009)
Alterations in components of the TGF-β superfamily signaling pathways in human cancer
Laurence Levy;Caroline S. Hill.
Cytokine & Growth Factor Reviews (2006)
Nucleocytoplasmic Shuttling of Smads 2, 3, and 4 Permits Sensing of TGF-β Receptor Activity
Gareth J. Inman;Francisco J. Nicolás;Caroline S. Hill.
Molecular Cell (2002)
Structure of the HMG box motif in the B-domain of HMG1.
H.M. Weir;P.J. Kraulis;C.S. Hill;A.R.C. Raine.
The EMBO Journal (1994)
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