D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Molecular Biology D-index 73 Citations 22,199 180 World Ranking 779 National Ranking 429

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Transcription factor

The scientist’s investigation covers issues in Signal transduction, Cell biology, SMAD, Transforming growth factor beta and Molecular biology. His Signal transduction research is multidisciplinary, incorporating perspectives in Receptor, Receptor complex, Cellular differentiation and R-SMAD. Xin-Hua Feng has researched Cell biology in several fields, including Genetics, Autocrine signalling and SOCS2.

His SMAD research incorporates elements of Transcription, Kinase and Ectopic expression. His Transforming growth factor beta study combines topics in areas such as Cancer research, Immunology, DNA-binding protein, TGF beta signaling pathway and Phosphorylation. He combines subjects such as Peptide sequence, Transcription factor, Protein kinase domain and Mitogen-activated protein kinase kinase with his study of Molecular biology.

His most cited work include:

  • SPECIFICITY AND VERSATILITY IN TGF-β SIGNALING THROUGH SMADS (1563 citations)
  • Transcriptional Activators of TGF-β Responses: Smads (925 citations)
  • Molecular Antagonism and Plasticity of Regulatory and Inflammatory T Cell Programs (894 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of investigation include Cell biology, Signal transduction, Phosphorylation, SMAD and Cancer research. His work carried out in the field of Cell biology brings together such families of science as Genetics, Ubiquitin and Cellular differentiation. His Signal transduction research is multidisciplinary, relying on both Molecular biology, Receptor and Ubiquitin ligase.

His studies deal with areas such as BMPR2 and Kinase as well as Phosphorylation. His SMAD study incorporates themes from Growth factor receptor, Transcription factor and Transcription. The Transforming growth factor beta study combines topics in areas such as R-SMAD and DNA-binding protein.

He most often published in these fields:

  • Cell biology (70.47%)
  • Signal transduction (43.01%)
  • Phosphorylation (37.31%)

What were the highlights of his more recent work (between 2015-2021)?

  • Cell biology (70.47%)
  • Phosphorylation (37.31%)
  • Signal transduction (43.01%)

In recent papers he was focusing on the following fields of study:

His scientific interests lie mostly in Cell biology, Phosphorylation, Signal transduction, Cancer research and Transforming growth factor. His Cell biology study focuses mostly on SMAD, Transforming growth factor beta, Kinase, Cell signaling and TANK-binding kinase 1. His SMAD study improves the overall literature in Genetics.

Xin-Hua Feng interconnects Tyrosine and Function in the investigation of issues within Phosphorylation. His research in Signal transduction intersects with topics in Molecular biology, Ubiquitin and R-SMAD. His work deals with themes such as Suppressor and Receptor, which intersect with Transforming growth factor.

Between 2015 and 2021, his most popular works were:

  • PPM1A Functions as a Smad Phosphatase to Terminate TGFβ Signaling (377 citations)
  • Smad7 Protein Interacts with Receptor-regulated Smads (R-Smads) to Inhibit Transforming Growth Factor-β (TGF-β)/Smad Signaling. (95 citations)
  • Single tumor-initiating cells evade immune clearance by recruiting type II macrophages (81 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Transcription factor

Xin-Hua Feng mainly focuses on Cell biology, Signal transduction, Cancer research, SMAD and Transforming growth factor beta. The various areas that Xin-Hua Feng examines in his Cell biology study include Innate immune system and Immunity. The concepts of his Signal transduction study are interwoven with issues in Protein kinase domain, Ubiquitin and Immunology.

The study incorporates disciplines such as Carcinogenesis, Transcription factor, STAT3, Gene silencing and Immunotherapy in addition to Cancer research. His Transforming growth factor beta research is multidisciplinary, incorporating elements of Acetylation, Cell morphology, Tubulin, Microtubule and Cytoskeleton. His research integrates issues of Receptor, Gene expression and Transforming growth factor in his study of Phosphorylation.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

SPECIFICITY AND VERSATILITY IN TGF-β SIGNALING THROUGH SMADS

Xin-Hua Feng;Rik Derynck.
Annual Review of Cell and Developmental Biology (2005)

2294 Citations

Smads: transcriptional activators of TGF-beta responses.

Rik Derynck;Ying Zhang;Xin-Hua Feng.
Cell (1998)

1524 Citations

Molecular Antagonism and Plasticity of Regulatory and Inflammatory T Cell Programs

Xuexian O. Yang;Roza Nurieva;Gustavo J. Martinez;Hong Soon Kang.
Immunity (2008)

1281 Citations

Receptor-associated Mad homologues synergize as effectors of the TGF-β response

Ying Zhang;Xin-Hua Feng;Rui-Yun Wu;Rik Derynck.
Nature (1996)

1114 Citations

Smad3 and Smad4 cooperate with c-Jun/c-Fos to mediate TGF-beta-induced transcription.

Ying Zhang;Xin-Hua Feng;Rik Derynck.
Nature (1998)

916 Citations

TGF-beta receptor signaling.

Rik Derynck;Xin-Hua Feng.
Biochimica et Biophysica Acta (1997)

864 Citations

The tumor suppressor Smad4/DPC4 and transcriptional adaptor CBP/p300 are coactivators for Smad3 in TGF-β-induced transcriptional activation

Xin-Hua Feng;Ying Zhang;Rui-Yun Wu;Rik Derynck.
Genes & Development (1998)

672 Citations

Smurf2 Is a Ubiquitin E3 Ligase Mediating Proteasome-dependent Degradation of Smad2 in Transforming Growth Factor-β Signaling

Xia Lin;Min Liang;Xin-Hua Feng.
Journal of Biological Chemistry (2000)

585 Citations

PPM1A Functions as a Smad Phosphatase to Terminate TGFβ Signaling

Xia Lin;Xueyan Duan;Yao Yun Liang;Ying Su.
Cell (2016)

575 Citations

Smad2, Smad3 and Smad4 cooperate with Sp1 to induce p15Ink4B transcription in response to TGF‐β

Xin‐Hua Feng;Xin‐Hua Feng;Xia Lin;Rik Derynck.
The EMBO Journal (2000)

518 Citations

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