What is he best known for?
The fields of study he is best known for:
- Internal medicine
- Neuron
- Biochemistry
Joe E. Springer focuses on Internal medicine, Endocrinology, Spinal cord injury, Cell biology and Spinal cord.
His Biochemistry research extends to Internal medicine, which is thematically connected.
His study on Endocrinology is mostly dedicated to connecting different topics, such as Nerve growth factor.
In his study, which falls under the umbrella issue of Spinal cord injury, 4-Hydroxynonenal, Amino acid and Ischemia is strongly linked to Lesion.
The study incorporates disciplines such as Caspase 3 and Apoptosis in addition to Cell biology.
His Spinal cord study combines topics in areas such as Programmed cell death and Pathology.
His most cited work include:
- Acute Inflammatory Response in Spinal Cord Following Impact Injury (434 citations)
- Activation of the caspase-3 apoptotic cascade in traumatic spinal cord injury. (384 citations)
- Prolonged survival and decreased abnormal movements in transgenic model of Huntington disease, with administration of the transglutaminase inhibitor cystamine. (355 citations)
What are the main themes of his work throughout his whole career to date?
His primary scientific interests are in Neuroscience, Internal medicine, Endocrinology, Spinal cord and Spinal cord injury.
His Neuroscience research incorporates themes from Amyotrophic lateral sclerosis, Signal transduction and Nerve growth factor.
His Endocrinology research focuses on subjects like Glutamate receptor, which are linked to Glutamic acid.
His Spinal cord course of study focuses on Pathology and Microglia.
His studies in Spinal cord injury integrate themes in fields like Caspase 3, Anesthesia, Neuroprotection, Pharmacology and Mitochondrion.
His studies deal with areas such as Lipid peroxidation, Oxidative stress, Reactive oxygen species and Abscess as well as Anesthesia.
He most often published in these fields:
- Neuroscience (28.05%)
- Internal medicine (28.05%)
- Endocrinology (25.61%)
What were the highlights of his more recent work (between 2012-2021)?
- Mitochondrion (20.73%)
- Cell biology (13.41%)
- Neuroscience (28.05%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include Mitochondrion, Cell biology, Neuroscience, Pediatrics and Endoplasmic reticulum.
His Mitochondrion research incorporates themes from Inflammation, Central nervous system, Tumor necrosis factor alpha and Crosstalk.
The Central nervous system study combines topics in areas such as Adenosine triphosphate, Function and Mitochondrial permeability transition pore.
His work on Mitochondrial protein is typically connected to Mouse cortex as part of general Cell biology study, connecting several disciplines of science.
His study deals with a combination of Neuroscience and mir-223.
He focuses mostly in the field of Pediatrics, narrowing it down to matters related to Incidence and, in some cases, Anesthesia and Abscess.
Between 2012 and 2021, his most popular works were:
- Mitochondria-Associated MicroRNAs in Rat Hippocampus Following Traumatic Brain Injury (76 citations)
- Mitochondria and microRNA crosstalk in traumatic brain injury. (14 citations)
- Fractionated mitochondrial magnetic separation for isolation of synaptic mitochondria from brain tissue. (13 citations)
In his most recent research, the most cited papers focused on:
- Internal medicine
- Neuron
- Biochemistry
The scientist’s investigation covers issues in Mitochondrion, Neuroscience, Cell biology, mir-223 and Traumatic brain injury.
His Mitochondrion research incorporates elements of Central nervous system, NIM811, Mitochondrial permeability transition pore and Adenosine triphosphate.
His work on Mitochondrial protein as part of general Cell biology research is frequently linked to Mouse cortex, thereby connecting diverse disciplines of science.
His study of mir-223 brings together topics like Dicer, Gene silencing, Argonaute and Neurodegeneration.
Traumatic brain injury is intertwined with Crosstalk, Bioinformatics, Oxidative damage and Cellular activity in his research.
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