D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 40 Citations 5,811 76 World Ranking 3668 National Ranking 1693

Overview

What is she best known for?

The fields of study she is best known for:

  • Gene
  • Immune system
  • Cytokine

Phyllis B. Silver mostly deals with Immunology, Autoimmunity, Antigen, Interferon gamma and Retinal. The study of Immunology is intertwined with the study of Interleukin 12 in a number of ways. Her work deals with themes such as Innate immune system and Peptide sequence, which intersect with Autoimmunity.

Her Antigen study integrates concerns from other disciplines, such as T cell, Uveitis and Immunity. Her research is interdisciplinary, bridging the disciplines of Interleukin 17 and Interferon gamma. Her studies in Cytokine integrate themes in fields like Delayed hypersensitivity and Adoptive cell transfer.

Her most cited work include:

  • Either a Th17 or a Th1 effector response can drive autoimmunity: conditions of disease induction affect dominant effector category. (545 citations)
  • Calcitriol Suppresses Antiretinal Autoimmunity through Inhibitory Effects on the Th17 Effector Response (243 citations)
  • Interleukin 12 protects from a T helper type 1-mediated autoimmune disease, experimental autoimmune uveitis, through a mechanism involving interferon gamma, nitric oxide, and apoptosis. (201 citations)

What are the main themes of her work throughout her whole career to date?

Phyllis B. Silver spends much of her time researching Immunology, Autoimmunity, Antigen, Autoimmune uveitis and Uveitis. Her study in Immunology focuses on Autoimmune disease, Effector, Cytokine, Interleukin 17 and Immune system. Phyllis B. Silver has included themes like Lymphocyte proliferation, Adoptive cell transfer and Pertussis toxin in her Autoimmune disease study.

She interconnects Inflammation and Priming in the investigation of issues within Cytokine. Her research in Autoimmunity intersects with topics in Pathogenesis, Retinal, Immune tolerance, Endogeny and T-cell receptor. Phyllis B. Silver focuses mostly in the field of Antigen, narrowing it down to topics relating to T cell and, in certain cases, Immunization.

She most often published in these fields:

  • Immunology (80.56%)
  • Autoimmunity (29.17%)
  • Antigen (22.22%)

What were the highlights of her more recent work (between 2013-2020)?

  • Immunology (80.56%)
  • Autoimmunity (29.17%)
  • Uveitis (16.67%)

In recent papers she was focusing on the following fields of study:

Her primary areas of study are Immunology, Autoimmunity, Uveitis, Autoimmune uveitis and Cytokine. She regularly links together related areas like Transgene in her Immunology studies. Her Autoimmunity study combines topics from a wide range of disciplines, such as Multiple sclerosis and Endogeny.

The Uveitis study combines topics in areas such as Innate immune system, Mediator, Crosstalk and C-type lectin. Her Cytokine research includes elements of Interleukin 12, Janus kinase 3, Interleukin 21, Neuroinflammation and Innate lymphoid cell. Her T cell research focuses on Interleukin 17 and how it relates to Candida albicans.

Between 2013 and 2020, her most popular works were:

  • Microbiota-Dependent Activation of an Autoreactive T Cell Receptor Provokes Autoimmunity in an Immunologically Privileged Site (196 citations)
  • An Ocular Commensal Protects against Corneal Infection by Driving an Interleukin-17 Response from Mucosal γδ T Cells (112 citations)
  • Retina-Specific T Regulatory Cells Bring About Resolution and Maintain Remission of Autoimmune Uveitis (46 citations)

In her most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Cytokine

Phyllis B. Silver mainly investigates Immunology, Uveitis, Antigen, T cell and Signal transduction. Much of her study explores Immunology relationship to In vivo. Her Antigen research is multidisciplinary, incorporating perspectives in Adjuvant, Pertussis toxin, Transcription factor and Haplotype.

Her biological study spans a wide range of topics, including Retinal, Interleukin 17, Transgene and Immunization. The various areas that Phyllis B. Silver examines in her Signal transduction study include Innate immune system, Autoimmune disease and C-type lectin. Her Autoimmunity research integrates issues from Receptor, Endogeny and Immune tolerance.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Either a Th17 or a Th1 effector response can drive autoimmunity: conditions of disease induction affect dominant effector category.

Dror Luger;Phyllis B Silver;Jun Tang;Daniel J. Cua.
Journal of Experimental Medicine (2008)

673 Citations

Calcitriol Suppresses Antiretinal Autoimmunity through Inhibitory Effects on the Th17 Effector Response

Jun Tang;Ru Zhou;Dror Luger;Wei Zhu.
Journal of Immunology (2009)

322 Citations

Interleukin 12 Protects from a T Helper Type 1–mediated Autoimmune Disease, Experimental Autoimmune Uveitis, through a Mechanism Involving Interferon γ, Nitric Oxide, and Apoptosis

Teresa K. Tarrant;Phyllis B. Silver;Jennifer L. Wahlsten;Luiz Vicente Rizzo.
Journal of Experimental Medicine (1999)

234 Citations

Endogenous systemic IFN-gamma has a protective role against ocular autoimmunity in mice.

RR Caspi;CC Chan;BG Grubbs;PB Silver.
Journal of Immunology (1994)

213 Citations

Microbiota-Dependent Activation of an Autoreactive T Cell Receptor Provokes Autoimmunity in an Immunologically Privileged Site

Reiko Horai;Carlos R. Zárate-Bladés;Patricia Dillenburg-Pilla;Jun Chen;Jun Chen.
Immunity (2015)

194 Citations

Essential Role of the MyD88 Pathway, but Nonessential Roles of TLRs 2, 4, and 9, in the Adjuvant Effect Promoting Th1-Mediated Autoimmunity

Shao Bo Su;Phyllis B. Silver;Rafael S. Grajewski;Rajeev K. Agarwal.
Journal of Immunology (2005)

188 Citations

Genetic susceptibility to experimental autoimmune uveoretinitis in the rat is associated with an elevated Th1 response.

R R Caspi;P B Silver;C C Chan;B Sun.
Journal of Immunology (1996)

180 Citations

Identification of a new epitope of human IRBP that induces autoimmune uveoretinitis in mice of the H-2b haplotype.

Dody Avichezer;Phyllis B. Silver;Chi-Chao Chan;Barbara Wiggert.
Investigative Ophthalmology & Visual Science (2000)

168 Citations

Rodent models of experimental autoimmune uveitis.

Rajeev K Agarwal;Phyllis B Silver;Rachel R Caspi.
Methods of Molecular Biology (2012)

141 Citations

Autoimmune Uveitis Elicited with Antigen-Pulsed Dendritic Cells Has a Distinct Clinical Signature and Is Driven by Unique Effector Mechanisms: Initial Encounter with Autoantigen Defines Disease Phenotype

Jun Tang;Wei Zhu;Phyllis B. Silver;Shao Bo Su.
Journal of Immunology (2007)

136 Citations

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