Rachel R. Caspi focuses on Immunology, Autoimmunity, Antigen, Autoimmune disease and Cytokine. The Immunology study combines topics in areas such as Retina and Retinal. The concepts of her Autoimmunity study are interwoven with issues in IL-2 receptor, Inflammation, Interferon gamma, Adjuvant and Innate immune system.
Her Antigen research is multidisciplinary, relying on both Oral administration, T cell and Antibody. Her Autoimmune disease research incorporates elements of Gene and Immunopathology. Her Cytokine research includes themes of Delayed hypersensitivity and Lymphocyte proliferation.
Her main research concerns Immunology, Antigen, Autoimmunity, Uveitis and Immune system. Her study in Autoimmune disease, Autoimmune uveitis, T cell, Effector and Cytokine is carried out as part of her Immunology studies. Her research in Autoimmune disease intersects with topics in Lymphocyte proliferation and Immunotherapy.
Rachel R. Caspi has researched Antigen in several fields, including Molecular biology and Adoptive cell transfer. Her Autoimmunity research integrates issues from Pathogenesis, Interleukin 17, Interferon gamma, Immune tolerance and T-cell receptor. Her Uveitis study which covers Retina that intersects with Pathology.
Rachel R. Caspi mainly focuses on Immunology, Uveitis, Autoimmunity, Autoimmune uveitis and Immune system. T cell, Antigen, Immunity, T-cell receptor and Cytokine are among the areas of Immunology where she concentrates her study. The Uveitis study combines topics in areas such as Retinal degeneration, Retina, Retinal, Immune privilege and Autoimmune disease.
Her research in Autoimmunity focuses on subjects like Gut flora, which are connected to Antibiotics. Her Autoimmune uveitis research is multidisciplinary, incorporating elements of Effector cell, Lipoxin a4, Pathogenesis and Complement. Her Immune system research incorporates themes from Interleukin, Multiple sclerosis, Lymphatic system and Disease.
Her primary areas of investigation include Immunology, Uveitis, Antigen, T cell and Immune system. Her Immunology study frequently intersects with other fields, such as Microbiome. Rachel R. Caspi has included themes like Cell, Cytokine, Pathology, Retina and Innate immune system in her Uveitis study.
Her work investigates the relationship between Antigen and topics such as Virology that intersect with problems in Apoptosis. Her studies in T cell integrate themes in fields like Inflammation, Interleukin 17, Receptor and Retinal. Her work carried out in the field of Immune system brings together such families of science as Multiple sclerosis and Gut flora.
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Either a Th17 or a Th1 effector response can drive autoimmunity: conditions of disease induction affect dominant effector category.
Dror Luger;Phyllis B Silver;Jun Tang;Daniel J. Cua.
Journal of Experimental Medicine (2008)
The Rd8 Mutation of the Crb1 Gene Is Present in Vendor Lines of C57BL/6N Mice and Embryonic Stem Cells, and Confounds Ocular Induced Mutant Phenotypes
Mary J. Mattapallil;Eric F. Wawrousek;Chi-Chao Chan;Hui Zhao.
Investigative Ophthalmology & Visual Science (2012)
A new model of autoimmune disease. Experimental autoimmune uveoretinitis induced in mice with two different retinal antigens.
R. R. Caspi;F. G. Roberge;Chi-Chao Chan;B. Wiggert.
Journal of Immunology (1988)
Th1 and Th17 cells
Jesse M. Damsker;Anna M. Hansen;Rachel R. Caspi.
Annals of the New York Academy of Sciences (2010)
Cutting Edge: NKT Cells Constitutively Express IL-23 Receptor and RORγt and Rapidly Produce IL-17 upon Receptor Ligation in an IL-6-Independent Fashion
Aleksandra V. Rachitskaya;Anna M. Hansen;Reiko Horai;Zhuqing Li.
Journal of Immunology (2008)
A look at autoimmunity and inflammation in the eye
Rachel R. Caspi.
Journal of Clinical Investigation (2010)
T cell lines mediating experimental autoimmune uveoretinitis (EAU) in the rat.
R R Caspi;F G Roberge;C G McAllister;M el-Saied.
Journal of Immunology (1986)
Calcitriol Suppresses Antiretinal Autoimmunity through Inhibitory Effects on the Th17 Effector Response
Jun Tang;Ru Zhou;Dror Luger;Wei Zhu.
Journal of Immunology (2009)
Eosinophil-derived neurotoxin acts as an alarmin to activate the TLR2–MyD88 signal pathway in dendritic cells and enhances Th2 immune responses
De-Xuan Yang;Qian-Qian Chen;Shao Bo Su;Ping Zhang.
Journal of Experimental Medicine (2008)
Inhibition of S-antigen induced experimental autoimmune uveoretinitis by oral induction of tolerance with S-antigen.
R B Nussenblatt;R R Caspi;R Mahdi;C C Chan.
Journal of Immunology (1990)
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