2003 - Fellow of the Royal Society of Edinburgh
John V. Forrester spends much of his time researching Immunology, Cell biology, Pathology, Retinal and Retina. His studies in Inflammation, Cytokine, Antigen, Microglia and Immune privilege are all subfields of Immunology research. His Antigen research is multidisciplinary, incorporating perspectives in Uveitis, Antigen-presenting cell and Immune system, Immunotherapy.
His Cell biology research is multidisciplinary, incorporating elements of Wound healing, Cell migration and Cell polarity. His work carried out in the field of Pathology brings together such families of science as Blood–retinal barrier and Dendritic cell. John V. Forrester is interested in Fundus, which is a branch of Retinal.
Immunology, Cell biology, Retinal, Pathology and Retina are his primary areas of study. His study in Inflammation, Uveitis, Antigen, Immune system and Autoimmunity are all subfields of Immunology. The Uveitis study which covers Surgery that intersects with Internal medicine.
John V. Forrester interconnects Wound healing, Cell migration, Cytokine and Antigen-presenting cell in the investigation of issues within Cell biology. His work deals with themes such as Cornea and Corneal transplantation, which intersect with Pathology. Retina and Microglia are frequently intertwined in his study.
John V. Forrester spends much of his time researching Immunology, Immune system, Inflammation, Pathology and Cell biology. He undertakes interdisciplinary study in the fields of Immunology and Context through his works. His study on T cell and Innate immune system is often connected to Population as part of broader study in Immune system.
John V. Forrester combines subjects such as Pathogen, CD8, Retina, Viral replication and Iris with his study of Inflammation. His Pathology research incorporates themes from Chemokine, Angiogenesis and Erg. John V. Forrester has researched Cell biology in several fields, including Retinal pigment epithelium, Retinal and Inflammasome.
John V. Forrester mainly investigates Immunology, Immune system, Cell biology, Acquired immune system and Inflammation. John V. Forrester has included themes like MPTP and Corneal transplantation in his Immunology study. His studies in Cell biology integrate themes in fields like Proliferative vitreoretinopathy, Retinal pigment epithelium, Photoreceptor outer segment, Retinal and Cell division.
His research in Acquired immune system intersects with topics in T cell and Interleukin 17. His Inflammation research includes elements of Pathogen, Congenital cytomegalovirus infection, CD8, Retina and Viral replication. His research investigates the connection between Pathogenesis and topics such as Choroid that intersect with issues in Pathology.
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Electrical signals control wound healing through phosphatidylinositol-3-OH kinase-γ and PTEN
Min Zhao;Bing Song;Jin Pu;Teiji Wada.
Nature (2006)
Para-inflammation in the aging retina.
Heping Xu;Mei Chen;John Vincent Forrester.
Progress in Retinal and Eye Research (2009)
Epidemiology of diabetic retinopathy and macular oedema: a systematic review
R Williams;M Airey;H Baxter;J Forrester.
Eye (2004)
Electrical stimulation directly induces pre-angiogenic responses in vascular endothelial cells by signaling through VEGF receptors
Min Zhao;Huai Bai;Entong Wang;John Vincent Forrester.
Journal of Cell Science (2003)
The Eye: Basic Sciences in Practice
J.V. Forrester;J.V. Forrester;A.D. Dick;Paul Mcmenamin;F. Roberts.
(2002)
An image-processing strategy for the segmentation and quantification of microaneurysms in fluorescein angiograms of the ocular fundus
Timothy Spencer;John A. Olson;Kenneth C. McHardy;Peter F. Sharp.
Computers and Biomedical Research (1996)
Lay perspectives: advantages for health research
Vikki A Entwistle;Mary J Renfrew;Steven Yearley;John Forrester.
BMJ (1998)
A comparison of computer based classification methods applied to the detection of microaneurysms in ophthalmic fluorescein angiograms.
Allan J. Frame;Peter E. Undrill;Michael J. Cree;John A. Olson.
Computers in Biology and Medicine (1998)
Inducible nitric oxide synthase isoform is a key mediator of leukostasis and blood-retinal barrier breakdown in diabetic retinopathy.
Ermelindo C Leal;Ayyakkannu Manivannan;Ken-Ichi Hosoya;Tetsuya Terasaki.
Investigative Ophthalmology & Visual Science (2007)
The corneoscleral limbus in human corneal epithelial wound healing.
Harminder S. Dua;John V. Forrester.
American Journal of Ophthalmology (1990)
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