Masashi Kato mostly deals with Melanoma, Immunology, Cancer research, Cell biology and Molecular biology. His research integrates issues of Tumor microenvironment, CD8, Pathology, Cytotoxic T cell and In vivo in his study of Melanoma. His studies in Immunology integrate themes in fields like Cancer cell and Tumor progression.
His Cancer research study integrates concerns from other disciplines, such as Genetically modified mouse, Transgene, Transcription factor, Epigenetics and Immunotherapy. His Tyrosine phosphorylation, Signal transduction and Phosphorylation study in the realm of Cell biology interacts with subjects such as 4-Hydroxynonenal. His Molecular biology study incorporates themes from Gene expression, MAPK14, Mitogen-activated protein kinase kinase, Akt/PKB signaling pathway and Kinase.
The scientist’s investigation covers issues in Optoelectronics, Melanoma, Cancer research, Cell biology and Carrier lifetime. His Optoelectronics research incorporates elements of Epitaxy and Analytical chemistry. His studies deal with areas such as Cancer, Immunology, Pathology, Genetically modified mouse and In vivo as well as Melanoma.
Masashi Kato interconnects HaCaT, Protein kinase B and Metastasis in the investigation of issues within Cancer research. His work on Biochemistry expands to the thematically related Cell biology. The Carrier lifetime study combines topics in areas such as Molecular physics, Recombination, Free carrier absorption and Photoconductivity.
Optoelectronics, Carrier lifetime, Photoconductivity, Cancer research and Epitaxy are his primary areas of study. His research in Optoelectronics focuses on subjects like Surface roughness, which are connected to Etching, Energy conversion efficiency, Absorption, Photocathode and Texture. In his study, which falls under the umbrella issue of Carrier lifetime, High voltage and Image resolution is strongly linked to Free carrier absorption.
Masashi Kato has researched Photoconductivity in several fields, including Molecular physics, Time constant and Photoluminescence. Masashi Kato studies Malignant transformation, a branch of Cancer research. His research in Epitaxy intersects with topics in Ion implantation, Resolution and Voltage.
Masashi Kato spends much of his time researching In vitro, Cancer research, Malignant transformation, Hyperpigmented skin and Optoelectronics. The concepts of his In vitro study are interwoven with issues in Morphogenesis and Period. His study in Cancer research is interdisciplinary in nature, drawing from both Protein kinase B, Lithium, Carcinogenesis, Toxicity and PI3K/AKT/mTOR pathway.
His Malignant transformation research is multidisciplinary, incorporating elements of In vitro study, Human lung and MAPK/ERK pathway. His work deals with themes such as Chronic exposure and Reflectivity, which intersect with Hyperpigmented skin. The various areas that Masashi Kato examines in his Optoelectronics study include Surface roughness, Stacking, Epitaxy and Etching.
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Tumor cells disseminate early, but immunosurveillance limits metastatic outgrowth, in a mouse model of melanoma
Jo Eyles;Anne Laure Puaux;Xiaojie Wang;Benjamin Toh.
Journal of Clinical Investigation (2010)
NCRs and DNAM-1 mediate NK cell recognition and lysis of human and mouse melanoma cell lines in vitro and in vivo
Tadepally Lakshmikanth;Shannon Burke;Talib Hassan Ali;Silvia Kimpfler.
Journal of Clinical Investigation (2009)
IL-18 induces PD-1-dependent immunosuppression in cancer.
Magali Terme;Evelyn Ullrich;Laetitia Aymeric;Kathrin Meinhardt.
Cancer Research (2011)
Chronic inflammation promotes myeloid-derived suppressor cell activation blocking antitumor immunity in transgenic mouse melanoma model
Meyer C;Sevko A;Ramacher M;Bazhin Av.
Proceedings of the National Academy of Sciences of the United States of America (2011)
Mesenchymal Transition and Dissemination of Cancer Cells Is Driven by Myeloid-Derived Suppressor Cells Infiltrating the Primary Tumor
Benjamin Toh;Xiaojie Wang;Jo Keeble;Wen Jing Sim.
PLOS Biology (2011)
4-hydroxynonenal induces a cellular redox status-related activation of the caspase cascade for apoptotic cell death.
Wei Liu;Masashi Kato;Anwaral A. Akhand;Akemi Hayakawa.
Journal of Cell Science (2000)
Transgenic mouse model for skin malignant melanoma
Masashi Kato;Masahide Takahashi;Anwarul A Akhand;Wei Liu.
Oncogene (1998)
Genomic analysis of homotypic vacuole fusion.
E. Scott Seeley;Masashi Kato;Nathan Margolis;William Wickner.
Molecular Biology of the Cell (2002)
Antitumor effect of paclitaxel is mediated by inhibition of myeloid-derived suppressor cells and chronic inflammation in the spontaneous melanoma model.
Alexandra Sevko;Alexandra Sevko;Tillmann Michels;Tillmann Michels;Melissa Vrohlings;Melissa Vrohlings;Ludmila Umansky.
Journal of Immunology (2013)
Chemotherapy Induces Intratumoral Expression of Chemokines in Cutaneous Melanoma, Favoring T-cell Infiltration and Tumor Control
Michelle Hong;Anne Laure Puaux;Caleb Huang;Laure Loumagne.
Cancer Research (2011)
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