His primary areas of study are Internal medicine, Endocrinology, Immunology, Rheumatoid arthritis and Arthritis. Cytokine, Dexamethasone, Hydrocortisone, Catecholamine and Rheumatology are subfields of Internal medicine in which his conducts study. His Rheumatology research includes themes of Dermatology, Anti–citrullinated protein antibody and Rheumatoid nodule.
The concepts of his Immunology study are interwoven with issues in Genetics and Molecular biology. His Rheumatoid arthritis research incorporates themes from Pregnancy and Postpartum period. His research in Arthritis intersects with topics in Immunostaining, Corticosterone and Pathology.
Ronald L. Wilder spends much of his time researching Immunology, Internal medicine, Arthritis, Rheumatoid arthritis and Endocrinology. In his research, Lymphocyte and T lymphocyte is intimately related to Peripheral blood mononuclear cell, which falls under the overarching field of Immunology. His Rheumatology, Adrenocorticotropic hormone, Inflammation, Cytokine and Dexamethasone investigations are all subjects of Internal medicine research.
His Arthritis study combines topics in areas such as Quantitative trait locus, Major histocompatibility complex and Pathology. His studies deal with areas such as Gastroenterology, Surgery and Disease as well as Rheumatoid arthritis. Ronald L. Wilder combines subjects such as Molecular biology and Cyclosporin a with his study of Antigen.
Immunology, Internal medicine, Genetics, Rheumatoid arthritis and Arthritis are his primary areas of study. His Immunology study combines topics from a wide range of disciplines, such as Genetic determinism and Allele. His Internal medicine research is multidisciplinary, incorporating elements of Endocrinology and Surgery.
The study incorporates disciplines such as Diabetes mellitus and Insulin in addition to Surgery. His work deals with themes such as Gastroenterology, Postpartum period, Human leukocyte antigen, Disease and Immunopathology, which intersect with Rheumatoid arthritis. While the research belongs to areas of Arthritis, Ronald L. Wilder spends his time largely on the problem of Major histocompatibility complex, intersecting his research to questions surrounding X chromosome and Candidate gene.
Ronald L. Wilder mainly focuses on Immunology, Internal medicine, Rheumatoid arthritis, Endocrinology and Immune system. His Genetics research extends to Immunology, which is thematically connected. Ronald L. Wilder works in the field of Rheumatoid arthritis, namely Rheumatoid factor.
His research integrates issues of Chromosome and Antigen in his study of Rheumatoid factor. Receptor, Agonist and Phosphodiesterase inhibitor is closely connected to Pharmacology in his research, which is encompassed under the umbrella topic of Endocrinology. Ronald L. Wilder interconnects Chronic stress, Postpartum period and Estrogen in the investigation of issues within Immune system.
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The American Rheumatism Association 1987 revised criteria for the classification of rheumatoid arthritis.
Frank C. Arnett;Steven M. Edworthy;Daniel A. Bloch;Dennis J. Mcshane.
Arthritis & Rheumatism (1988)
The Sympathetic Nerve—An Integrative Interface between Two Supersystems: The Brain and the Immune System
Elenkov Ij;Wilder Rl;Chrousos Gp;Vizi Es.
Pharmacological Reviews (2000)
Expression of Cyclooxygenase-1 and -2 in Human Colorectal Cancer
Hajime Sano;Yutaka Kawahito;Ronald L. Wilder;Akira Hashiramoto.
Cancer Research (1995)
The Pathophysiologic Roles of Interleukin-6 in Human Disease
Dimitris A. Papanicolaou;Ronald L. Wilder;Stavros C. Manolagas;George P. Chrousos.
Annals of Internal Medicine (1998)
Glucocorticoid Therapy for Immune-Mediated Diseases: Basic and Clinical Correlates
Dimitrios T. Boumpas;George P. Chrousos;Ronald L. Wilder;Thomas R. Cupps.
Annals of Internal Medicine (1993)
Cyclooxygenase-1 and -2 expression in rheumatoid synovial tissues. Effects of interleukin-1 beta, phorbol ester, and corticosteroids.
Leslie J. Crofford;Ronald L. Wilder;Ari P. Ristimaki;Hajime Sano.
Journal of Clinical Investigation (1994)
Inflammatory mediator-induced hypothalamic-pituitary-adrenal axis activation is defective in streptococcal cell wall arthritis-susceptible Lewis rats
Esther M. Sternberg;Joanna M. Hill;George P. Chrousos;Themis Kamilaris.
Proceedings of the National Academy of Sciences of the United States of America (1989)
Induction of vascular endothelial growth factor expression in synovial fibroblasts by prostaglandin E and interleukin‐1: a potential mechanism for inflammatory angiogenesis
Pazit Ben-Av;Leslie J. Crofford;Ronald L. Wilder;Timothy Hla.
FEBS Letters (1995)
Neuroendocrine-immune system interactions and autoimmunity.
R L Wilder.
Annual Review of Immunology (1995)
Hypothalamic-pituitary-adrenal axis perturbations in patients with fibromyalgia.
Leslie J. Crofford;Stanley R. Pillemer;Konstantine T. Kalogeras;Joseph M. Cash.
Arthritis & Rheumatism (1994)
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