What is she best known for?
The fields of study she is best known for:
Manuela Neumann focuses on Pathology, Frontotemporal lobar degeneration, Amyotrophic lateral sclerosis, Frontotemporal dementia and Ubiquitin.
The various areas that she examines in her Pathology study include Neuroscience and Phosphorylation.
Her studies in Frontotemporal lobar degeneration integrate themes in fields like Neuropathology, Pathological, RNA-Binding Protein FUS and Genetics.
She works mostly in the field of Amyotrophic lateral sclerosis, limiting it down to concerns involving Neurodegeneration and, occasionally, C9orf72 Protein.
Her study in Frontotemporal dementia is interdisciplinary in nature, drawing from both Progressive supranuclear palsy, Anatomical pathology and Gene mutation.
She has included themes like Immunohistochemistry, TAR DNA-Binding Protein 43, Monoclonal antibody and Pathogenesis in her Ubiquitin study.
Her most cited work include:
- Ubiquitinated TDP-43 in frontotemporal lobar degeneration and amyotrophic lateral sclerosis (4135 citations)
- Neuropathologic diagnostic and nosologic criteria for frontotemporal lobar degeneration: consensus of the Consortium for Frontotemporal Lobar Degeneration (829 citations)
- Nomenclature and nosology for neuropathologic subtypes of frontotemporal lobar degeneration: an update (699 citations)
What are the main themes of her work throughout her whole career to date?
Her primary areas of study are Pathology, Frontotemporal lobar degeneration, Frontotemporal dementia, Amyotrophic lateral sclerosis and Disease.
Much of her study explores Pathology relationship to Ubiquitin.
The concepts of her Frontotemporal lobar degeneration study are interwoven with issues in Genetics, Pathological, Molecular biology, Cytoplasmic inclusion and Neuroscience.
Her Frontotemporal dementia research is multidisciplinary, incorporating perspectives in Mutation, Gene mutation, Progressive supranuclear palsy and Tau protein.
Her Amyotrophic lateral sclerosis research is multidisciplinary, incorporating elements of C9orf72, Neurodegeneration and Spinal cord.
Manuela Neumann focuses mostly in the field of Disease, narrowing it down to topics relating to Immunology and, in certain cases, Disease progression and Biomarker.
She most often published in these fields:
- Pathology (62.78%)
- Frontotemporal lobar degeneration (43.89%)
- Frontotemporal dementia (33.89%)
What were the highlights of her more recent work (between 2014-2021)?
- Pathology (62.78%)
- Disease (25.00%)
- C9orf72 (21.67%)
In recent papers she was focusing on the following fields of study:
Pathology, Disease, C9orf72, Cell biology and Neurofilament light are her primary areas of study.
Her work carried out in the field of Pathology brings together such families of science as Phenotype and Neuroscience.
Her Disease study combines topics from a wide range of disciplines, such as Genetics, Neurofilament and Genotyping.
Her research integrates issues of C9orf72 Protein and Mutation in her study of C9orf72.
In her research, Whole genome sequencing is intimately related to Frontotemporal lobar degeneration, which falls under the overarching field of Gene.
Her research in Congenic intersects with topics in Internal medicine, Amyotrophic lateral sclerosis and Neurodegeneration.
Between 2014 and 2021, her most popular works were:
- Neurofilament Light Chain in Blood and CSF as Marker of Disease Progression in Mouse Models and in Neurodegenerative Diseases. (191 citations)
- Neurofilament Light Chain in Blood and CSF as Marker of Disease Progression in Mouse Models and in Neurodegenerative Diseases. (191 citations)
- PART is part of Alzheimer disease. (180 citations)
In her most recent research, the most cited papers focused on:
Her scientific interests lie mostly in Cell biology, Pathology, Disease, Tauopathy and C9orf72.
Her biological study spans a wide range of topics, including Arginine, Neurodegeneration and RNA-Binding Protein FUS.
Her Alzheimer's disease study, which is part of a larger body of work in Pathology, is frequently linked to Invariant feature, bridging the gap between disciplines.
Her work on Disease progression, Alpha-synuclein and Neurofilament light as part of general Disease research is frequently linked to Intermediate filament, thereby connecting diverse disciplines of science.
Her C9orf72 study combines topics in areas such as C9orf72 Protein, Haploinsufficiency, Mutation and Gene isoform.
Manuela Neumann combines subjects such as Frontotemporal dementia and Pathogenesis with her study of Trinucleotide repeat expansion.
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